Persistence of RNA viruses in the central nervous system.
about
A matrix-less measles virus is infectious and elicits extensive cell fusion: consequences for propagation in the brain.Sporadic amyotrophic lateral sclerosis: a hypothesis of persistent (non-lytic) enteroviral infection.Synaptic pathology in Borna disease virus persistent infection.Altered central nervous system gene expression caused by congenitally acquired persistent infection with lymphocytic choriomeningitis virus.A single amino acid change in the glycoprotein of lymphocytic choriomeningitis virus is associated with the ability to cause growth hormone deficiency syndromeMolecular epidemiology and changing distribution of genotypes of measles virus field strains in Japan.Cell fusion by the envelope glycoproteins of persistent measles viruses which caused lethal human brain disease.Role of biased hypermutation in evolution of subacute sclerosing panencephalitis virus from progenitor acute measles virus.Irreversible modification of measles virus RNA in vitro by nuclear RNA-unwinding activity in human neuroblastoma cells.Functional and nonfunctional measles virus matrix genes from lethal human brain infections.Effects of cell differentiation on replication of A/WS/33, WSN, and A/PR/8/34 influenza viruses in mouse brain cell cultures: biological and immunological characterization of productsIdentification of point mutations in the envelope gene of Moloney murine leukemia virus TB temperature-sensitive paralytogenic mutant ts1: molecular determinants for neurovirulence.Generalized and localized biased hypermutation affecting the matrix gene of a measles virus strain that causes subacute sclerosing panencephalitis.Within host RNA virus persistence: mechanisms and consequences.Chemokine gene expression in astrocytes of Borna disease virus-infected rats and mice in the absence of inflammation.Strength of envelope protein interaction modulates cytopathicity of measles virus.Tyrosine phosphorylation of measles virus nucleocapsid protein in persistently infected neuroblastoma cells.Replication of lymphocytic choriomeningitis virus is restricted in terminally differentiated neurons.Functional disturbances during slow viral infections of the central nervous system.Generation and properties of measles virus mutations typically associated with subacute sclerosing panencephalitis.Mutation of the protein kinase C site in borna disease virus phosphoprotein abrogates viral interference with neuronal signaling and restores normal synaptic activity.NMDA-Receptor Antagonist Prevents Measles Virus-induced Neurodegeneration.Mumps virus alters aggregation of acetylcholine receptors in cultured rat skeletal muscle cells.
P2860
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P2860
Persistence of RNA viruses in the central nervous system.
description
1986 nî lūn-bûn
@nan
1986年の論文
@ja
1986年学术文章
@wuu
1986年学术文章
@zh-cn
1986年学术文章
@zh-hans
1986年学术文章
@zh-my
1986年学术文章
@zh-sg
1986年學術文章
@yue
1986年學術文章
@zh
1986年學術文章
@zh-hant
name
Persistence of RNA viruses in the central nervous system.
@en
Persistence of RNA viruses in the central nervous system.
@nl
type
label
Persistence of RNA viruses in the central nervous system.
@en
Persistence of RNA viruses in the central nervous system.
@nl
prefLabel
Persistence of RNA viruses in the central nervous system.
@en
Persistence of RNA viruses in the central nervous system.
@nl
P1476
Persistence of RNA viruses in the central nervous system.
@en
P2093
Kristensson K
P304
P356
10.1146/ANNUREV.MI.40.100186.001111
P577
1986-01-01T00:00:00Z