Tristetraprolin regulates the stability of HIF-1alpha mRNA during prolonged hypoxia.
about
Hallmarks of cancer and AU-rich elementsRegulation of the mRNA half-life in breast cancerPost-transcriptional control during chronic inflammation and cancer: a focus on AU-rich elements.RNase L attenuates mitogen-stimulated gene expression via transcriptional and post-transcriptional mechanisms to limit the proliferative responsePost-Transcriptional Control of the Hypoxic Response by RNA-Binding Proteins and MicroRNAs.Computational Model of MicroRNA Control of HIF-VEGF Pathway: Insights into the Pathophysiology of Ischemic Vascular Disease and CancerHistone Deacetylase Inhibitors Activate Tristetraprolin Expression through Induction of Early Growth Response Protein 1 (EGR1) in Colorectal Cancer Cells.Transcriptional and Post-Transcriptional Regulation of Thrombospondin-1 Expression: A Computational Model.Glycolysis determines dichotomous regulation of T cell subsets in hypoxiamiR-29a inhibition normalizes HuR over-expression and aberrant AU-rich mRNA stability in invasive cancer.Tristetraprolin (TTP): interactions with mRNA and proteins, and current thoughts on mechanisms of action.The cAMP pathway regulates mRNA decay through phosphorylation of the RNA-binding protein TIS11b/BRF1.Modulation of hypoxia-inducible factors (HIF) from an integrative pharmacological perspective.Tristetraprolin: roles in cancer and senescence.Tristetraprolin inhibits gastric cancer progression through suppression of IL-33.Inactivation of tristetraprolin in chronic hypoxia provokes the expression of cathepsin B.Dysregulation of TTP and HuR plays an important role in cancers.The P-body component USP52/PAN2 is a novel regulator of HIF1A mRNA stability.ZFP36 expression impairs glioblastoma cell lines viability and invasiveness by targeting multiple signal transduction pathways.A synonymous polymorphism of the Tristetraprolin (TTP) gene, an AU-rich mRNA-binding protein, affects translation efficiency and response to Herceptin treatment in breast cancer patients.Multilevel regulation of HIF-1 signaling by TTP.Effects of mitochondrial dysfunction on the immunological properties of microgliaHIF repressors under chronic hypoxia.Protein kinase D2 and heat shock protein 90 beta are required for BCL6-associated zinc finger protein mRNA stabilization induced by vascular endothelial growth factor-A.
P2860
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P2860
Tristetraprolin regulates the stability of HIF-1alpha mRNA during prolonged hypoxia.
description
2009 nî lūn-bûn
@nan
2009年の論文
@ja
2009年学术文章
@wuu
2009年学术文章
@zh-cn
2009年学术文章
@zh-hans
2009年学术文章
@zh-my
2009年学术文章
@zh-sg
2009年學術文章
@yue
2009年學術文章
@zh
2009年學術文章
@zh-hant
name
Tristetraprolin regulates the stability of HIF-1alpha mRNA during prolonged hypoxia.
@en
Tristetraprolin regulates the stability of HIF-1alpha mRNA during prolonged hypoxia.
@nl
type
label
Tristetraprolin regulates the stability of HIF-1alpha mRNA during prolonged hypoxia.
@en
Tristetraprolin regulates the stability of HIF-1alpha mRNA during prolonged hypoxia.
@nl
prefLabel
Tristetraprolin regulates the stability of HIF-1alpha mRNA during prolonged hypoxia.
@en
Tristetraprolin regulates the stability of HIF-1alpha mRNA during prolonged hypoxia.
@nl
P2093
P1476
Tristetraprolin regulates the stability of HIF-1alpha mRNA during prolonged hypoxia.
@en
P2093
Bo Hwa Sohn
Byung Joong Choi
Hyang-Sook Yoo
Jung Ju Lee
Kyung Chan Park
Tae Woo Kim
Yejin Jang
Young Il Yeom
P304
P356
10.1016/J.BBRC.2009.11.174
P407
P577
2009-12-04T00:00:00Z