Cellular mechanisms of IL-17-induced blood-brain barrier disruption. - Wikidata - awgldk - TriplyDB

Cellular mechanisms of IL-17-induced blood-brain barrier disruption.

Cellular mechanisms of IL-17-induced blood-brain barrier disruption.

http://www.wikidata.org/entity/Q39770425

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CXCR7 influences leukocyte entry into the CNS parenchyma by controlling abluminal CXCL12 abundance during autoimmunity MCAM-expressing CD4(+) T cells in peripheral blood secrete IL-17A and are significantly elevated in inflammatory autoimmune diseases Pediatric Autoimmune Disorders Associated with Streptococcal Infections and Tourette's Syndrome in Preclinical Studies.Impact of Exercise on Innate Immunity in Multiple Sclerosis Progression and Symptomatology Th17 Cells Pathways in Multiple Sclerosis and Neuromyelitis Optica Spectrum Disorders: Pathophysiological and Therapeutic Implications Non-muscle myosin II in disease: mechanisms and therapeutic opportunities Role of Tumor Necrosis Factor Superfamily in Neuroinflammation and Autoimmunity Exploiting the Pleiotropic Antioxidant Effects of Established Drugs in Cardiovascular Disease Vitiligo: interplay between oxidative stress and immune system New and emerging immune-targeted drugs for the treatment of multiple sclerosis Redox control of microglial function: molecular mechanisms and functional significance Multiple sclerosis: molecular mechanisms and therapeutic opportunities Blood-Brain Barrier Disruption Induced by Chronic Sleep Loss: Low-Grade Inflammation May Be the Link The IL-17 pathway as a major therapeutic target in autoimmune diseases A cannabigerol derivative suppresses immune responses and protects mice from experimental autoimmune encephalomyelitis Emerging Applications of Therapeutic Ultrasound in Neuro-oncology: Moving Beyond Tumor Ablation.Regulatory lymphocytes are key factors in MHC-independent resistance to EAE Interleukin 17A Promotes Lymphocytes Adhesion and Induces CCL2 and CXCL1 Release from Brain Endothelial Cells.Myelin recovery in multiple sclerosis: the challenge of remyelination Kv1.3 channel blocker (ImKTx88) maintains blood-brain barrier in experimental autoimmune encephalomyelitis.Anti-N-Methyl-D-aspartate Receptor Encephalitis: A Severe, Potentially Reversible Autoimmune Encephalitis.Poststroke neuropsychiatric symptoms: relationships with IL-17 and oxidative stress Increased interleukin-17 in peripheral blood and cerebrospinal fluid of neurosyphilis patients Th17 pathway-mediated immunopathogenesis of schizophrenia: mechanisms and implications Morphine induces bacterial translocation in mice by compromising intestinal barrier function in a TLR-dependent manner CNS-targeted production of IL-17A induces glial activation, microvascular pathology and enhances the neuroinflammatory response to systemic endotoxemia.Expression of neuronal CXCL10 induced by rabies virus infection initiates infiltration of inflammatory cells, production of chemokines and cytokines, and enhancement of blood-brain barrier permeability.Drug induced increases in CNS dopamine alter monocyte, macrophage and T cell functions: implications for HAND.Novel neuroinflammatory targets in the chronically injured spinal cord Regulation and function of IL-17A- and IL-22-producing γδ T cells.Developmental endothelial locus-1 is a homeostatic factor in the central nervous system limiting neuroinflammation and demyelination.Low dose dextromethorphan attenuates moderate experimental autoimmune encephalomyelitis by inhibiting NOX2 and reducing peripheral immune cells infiltration in the spinal cord Poststroke depression as a factor adversely affecting the level of oxidative damage to plasma proteins during a brain stroke Targeting epidermal fatty acid binding protein for treatment of experimental autoimmune encephalomyelitis.Viral Infection of the Central Nervous System and Neuroinflammation Precede Blood-Brain Barrier Disruption during Japanese Encephalitis Virus Infection.Opioid Exacerbation of Gram-positive sepsis, induced by Gut Microbial Modulation, is Rescued by IL-17A Neutralization.Mechanisms of dendritic cell trafficking across the blood-brain barrier.Interleukin-1β-induced barrier dysfunction is signaled through PKC-θ in human brain microvascular endothelium.Increased production of IL-17 in children with autism spectrum disorders and co-morbid asthma.Aquaporin 4-specific T cells in neuromyelitis optica exhibit a Th17 bias and recognize Clostridium ABC transporter

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Cellular mechanisms of IL-17-induced blood-brain barrier disruption.

http://www.wikidata.org/entity/Q39770425

description

2009 nî lūn-bûn

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2009年の論文

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2009年学术文章

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2009年学术文章

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2009年学术文章

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2009年学术文章

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2009年学术文章

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2009年學術文章

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2009年學術文章

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2009年學術文章

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name

Cellular mechanisms of IL-17-induced blood-brain barrier disruption.

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Cellular mechanisms of IL-17-induced blood-brain barrier disruption.

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type

label

Cellular mechanisms of IL-17-induced blood-brain barrier disruption.

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Cellular mechanisms of IL-17-induced blood-brain barrier disruption.

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prefLabel

Cellular mechanisms of IL-17-induced blood-brain barrier disruption.

@en

Cellular mechanisms of IL-17-induced blood-brain barrier disruption.

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Cellular mechanisms of IL-17-induced blood-brain barrier disruption.

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Andrew Croxford

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Ari Waisman

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Burkhard Becher

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Christoph R W Kuhlmann

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Dorothea Closhen

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Eweline Pietrowski

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Heiko J Luhmann

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Ingo Bechmann

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Jula Huppert

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Paulina Kulig

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1023-1034

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10.1096/FJ.09-141978

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4

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24

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2009-11-25T00:00:00Z

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40033480

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19940258

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blood–brain barrier