Drug-induced apoptosis in hepatoma cells is mediated by the CD95 (APO-1/Fas) receptor/ligand system and involves activation of wild-type p53. - Wikidata - awgldk - TriplyDB

Drug-induced apoptosis in hepatoma cells is mediated by the CD95 (APO-1/Fas) receptor/ligand system and involves activation of wild-type p53.

Drug-induced apoptosis in hepatoma cells is mediated by the CD95 (APO-1/Fas) receptor/ligand system and involves activation of wild-type p53.

http://www.wikidata.org/entity/Q39777499

about

Zerumbone induced apoptosis in liver cancer cells via modulation of Bax/Bcl-2 ratio The Fas death signaling pathway connecting reactive oxygen species generation and FLICE inhibitory protein down-regulation Nitric oxide negatively regulates Fas CD95-induced apoptosis through inhibition of ubiquitin-proteasome-mediated degradation of FLICE inhibitory protein CDIP, a novel pro-apoptotic gene, regulates TNFalpha-mediated apoptosis in a p53-dependent manner PMLRARα binds to Fas and suppresses Fas-mediated apoptosis through recruiting c-FLIP in vivo The effects of chemotherapeutics on cellular metabolism and consequent immune recognition Cellular Pathways in Response to Ionizing Radiation and Their Targetability for Tumor Radiosensitization Clofibrate treatment in pigs: effects on parameters critical with respect to peroxisome proliferator-induced hepatocarcinogenesis in rodents Colchicine protects mice from the lethal effect of an agonistic anti-Fas antibody Apoptosis of haematopoietic cells upon thymidylate synthase inhibition is independent of p53 accumulation and CD95-CD95 ligand interaction Butyric acid-induced T-cell apoptosis is mediated by caspase-8 and -9 activation in a Fas-independent manner Gallium compound GaQ(3) -induced Ca(2+) signalling triggers p53-dependent and -independent apoptosis in cancer cells DNA damage induces down-regulation of Prp19 via impairing Prp19 stability in hepatocellular carcinoma cells Ceramide Synthase 6 Is a Novel Target of Methotrexate Mediating Its Antiproliferative Effect in a p53-Dependent Manner Differential role of transcription-coupled repair in UVB-induced G2 arrest and apoptosis in mouse epidermis Chemosensitivity of TEL-AML1 fusion transcript positive acute lymphoblastic leukemia cells.p53/T-antigen complex disruption in T-antigen transformed NIH3T3 fibroblasts exposed to oxidative stress: correlation with the appearance of a Fas/APO-1/CD95 dependent, caspase independent, necrotic pathway.Effect of methotrexate on neuroepithelium in the rat fetal brain.Adoptive transfer of human papillomavirus E7-specific CTL enhances tumor chemoresponse through the perforin/granzyme-mediated pathway.Cell death induced by the Fas/Fas ligand pathway and its role in pathology.Relationship between therapeutic efficacy of arterial infusion chemotherapy and expression of P-glycoprotein and p53 protein in advanced hepatocellular carcinoma.Involvement of Fas system-mediated apoptosis in pathogenesis of viral hepatitis.TRAIL receptor signaling regulation of chemosensitivity in vivo but not in vitro The effects of fucodian on senescence are controlled by the p16INK4a-pRb and p14Arf-p53 pathways in hepatocellular carcinoma and hepatic cell lines.Up-regulation of Fas reverses cisplatin resistance of human small cell lung cancer cells Nitric oxide and redox regulation in the liver: Part I. General considerations and redox biology in hepatitis Relationship of p53 overexpression on cancers and recognition by anti-p53 T cell receptor-transduced T cells Vitamin C enhances anticancer activity in methotrexate‑treated Hep3B hepatocellular carcinoma cells Combination of hepatocyte specific delivery and transformation dependent expression of shRNA inducing transcriptional gene silencing of c-Myc promoter in hepatocellular carcinoma cells.Royal jelly modulates oxidative stress and apoptosis in liver and kidneys of rats treated with cisplatin.Apoptosis in diseases of the liver.Resveratrol--from the bottle to the bedside?The effect of drugs and toxins on the process of apoptosis.Pigment epithelial-derived factor (PEDF)-triggered lung cancer cell apoptosis relies on p53 protein-driven Fas ligand (Fas-L) up-regulation and Fas protein cell surface translocation.CD95 is part of a let-7/p53/miR-34 regulatory network Complex changes in the apoptotic and cell differentiation programs during initiation of the hair follicle response to chemotherapy Fas receptor-mediated apoptosis: a clinical application?Serum starvation induces DRAM expression in liver cancer cells via histone modifications within its promoter locus Hepatitis C virus NS2 protein inhibits DNA damage pathway by sequestering p53 to the cytoplasm.The emergence of resistance to targeted cancer therapeutics.

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P2860

Drug-induced apoptosis in hepatoma cells is mediated by the CD95 (APO-1/Fas) receptor/ligand system and involves activation of wild-type p53.

http://www.wikidata.org/entity/Q39777499

description

1997 nî lūn-bûn

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1997年の論文

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1997年学术文章

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1997年学术文章

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1997年学术文章

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1997年学术文章

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1997年学术文章

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1997年學術文章

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1997年學術文章

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1997年學術文章

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name

Drug-induced apoptosis in hepa ...... s activation of wild-type p53.

@en

Drug-induced apoptosis in hepatoma cells is mediated by the CD95

@nl

type

label

Drug-induced apoptosis in hepa ...... s activation of wild-type p53.

@en

Drug-induced apoptosis in hepatoma cells is mediated by the CD95

@nl

prefLabel

Drug-induced apoptosis in hepa ...... s activation of wild-type p53.

@en

Drug-induced apoptosis in hepatoma cells is mediated by the CD95

@nl

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Journal of Clinical Investigation

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Drug-induced apoptosis in hepa ...... es activation of wild-type p53

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Galle PR

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Heinemann EM

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Hofmann WJ

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Hug H

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Krammer PH

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Müller M

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Strand S

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Walczak H

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P2860

Interaction of the p53-regulated protein Gadd45 with proliferating cell nuclear antigen

Cytotoxicity-dependent APO-1 (Fas/CD95)-associated proteins form a death-inducing signaling complex (DISC) with the receptor

Identification of programmed cell death in situ via specific labeling of nuclear DNA fragmentation

WAF1, a potential mediator of p53 tumor suppression

Assignment of the human Fas antigen gene (Fas) to 10q24.1

Tumor suppressor p53 is a regulator of bcl-2 and bax gene expression in vitro and in vivo

FLICE, a novel FADD-homologous ICE/CED-3-like protease, is recruited to the CD95 (Fas/APO-1) death--inducing signaling complex

Molecular cloning and expression of the Fas ligand, a novel member of the tumor necrosis factor family

Tumor suppressor p53 is a direct transcriptional activator of the human bax gene

Bcl-2 and Fas/APO-1 regulate distinct pathways to lymphocyte apoptosis

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403-413

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10.1172/JCI119174

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apoptotic process

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507813

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