Epigenetic modifications induced by RGC-32 in colon cancer. - Wikidata - awgldk - TriplyDB

Epigenetic modifications induced by RGC-32 in colon cancer.

Epigenetic modifications induced by RGC-32 in colon cancer.

http://www.wikidata.org/entity/Q39780293

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Response gene to complement 32 regulates the G2/M phase checkpoint during renal tubular epithelial cell repair.RGC32 induces epithelial-mesenchymal transition by activating the Smad/Sip1 signaling pathway in CRC.Upregulation of the cell-cycle regulator RGC-32 in Epstein-Barr virus-immortalized cells.Response gene to complement 32 (RGC-32) in endothelial cells is induced by glucose and helpful to maintain glucose homeostasis.Genetics, cytogenetics, and epigenetics of colorectal cancer.RGC32 deficiency protects against high-fat diet-induced obesity and insulin resistance in mice.Multi-faceted quantitative proteomics analysis of histone H2B isoforms and their modifications.Response gene to complement-32 enhances metastatic phenotype by mediating transforming growth factor beta-induced epithelial-mesenchymal transition in human pancreatic cancer cell line BxPC-3 RGC-32 Deficiency Protects against Hepatic Steatosis by Reducing Lipogenesis.Promoter Methylation and mRNA Expression of Response Gene to Complement 32 in Breast Carcinoma RGC-32 is a novel regulator of the T-lymphocyte cell cycle Role of C5b-9 complement complex and response gene to complement-32 (RGC-32) in cancer.Role of SIRT1 in autoimmune demyelination and neurodegeneration.The Role of Stem Cell DNA Methylation in Colorectal Carcinogenesis.The expression of response gene to complement 32 on renal ischemia reperfusion injury in rat.The complement system as a biomarker of disease activity and response to treatment in multiple sclerosis.RGC-32 Promotes Th17 Cell Differentiation and Enhances Experimental Autoimmune Encephalomyelitis.RGC-32 (Response Gene to Complement 32 Protein) Deficiency Protects Endothelial Cells From Inflammation and Attenuates Atherosclerosis.Renal proteomic analysis of RGC-32 knockout mice reveals the potential mechanism of RGC-32 in regulating cell cycle.Response Gene to Complement 32 in Vascular Diseases

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P2860

Epigenetic modifications induced by RGC-32 in colon cancer.

http://www.wikidata.org/entity/Q39780293

description

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2009年の論文

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2009年論文

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2009年论文

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2009年论文

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2009年论文

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name

Epigenetic modifications induced by RGC-32 in colon cancer.

@en

Epigenetic modifications induced by RGC-32 in colon cancer.

@nl

type

label

Epigenetic modifications induced by RGC-32 in colon cancer.

@en

Epigenetic modifications induced by RGC-32 in colon cancer.

@nl

prefLabel

Epigenetic modifications induced by RGC-32 in colon cancer.

@en

Epigenetic modifications induced by RGC-32 in colon cancer.

@nl

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J.YEXMP.2009.10.010

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Experimental and Molecular Pathology

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Epigenetic modifications induced by RGC-32 in colon cancer.

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Abulkalam Shamsuddin

http://www.w3.org/2001/XMLSchema#string

Cornelia D Cudrici

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Cosmin A Tegla

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Hegang Chen

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Horea Rus

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Matthew Fosbrink

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Petru A Mircea

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Philippe Azimzadeh

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Sonia I Vlaicu

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Vingh Nguyen

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P2860

Gene ontology: tool for the unification of biology

RGC-32 increases p34CDC2 kinase activity and entry of aortic smooth muscle cells into S-phase

RGC-32 mediates transforming growth factor-beta-induced epithelial-mesenchymal transition in human renal proximal tubular cells

Response gene to complement 32 is required for C5b-9 induced cell cycle activation in endothelial cells

RGC32, a novel p53-inducible gene, is located on centrosomes during mitosis and results in G2/M arrest

Cyclin A and cyclin D1 as significant prognostic markers in colorectal cancer patients

Molecular cloning and characterization of RGC-32, a novel gene induced by complement activation in oligodendrocytes

A multigenic program mediating breast cancer metastasis to bone

Cancer epigenomics: DNA methylomes and histone-modification maps

The SIRT1 deacetylase suppresses intestinal tumorigenesis and colon cancer growth

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