ATP released by electrical stimuli elicits calcium transients and gene expression in skeletal muscle. - Wikidata - awgldk - TriplyDB

ATP released by electrical stimuli elicits calcium transients and gene expression in skeletal muscle.

ATP released by electrical stimuli elicits calcium transients and gene expression in skeletal muscle.

http://www.wikidata.org/entity/Q39788355

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Extrinsic and intrinsic control of macrophage inflammatory responses Contribution of non-endothelium-dependent substances to exercise hyperaemia: are they O(2) dependent?The pannexins: past and present Alternating bipolar field stimulation identifies muscle fibers with defective excitability but maintained local Ca(2+) signals and contraction.Pannexin1 stabilizes synaptic plasticity and is needed for learning.Anti-inflammatory drugs for Duchenne muscular dystrophy: focus on skeletal muscle-releasing factors Abundant expression and functional participation of TRPV1 at Zusanli acupoint (ST36) in mice: mechanosensitive TRPV1 as an "acupuncture-responding channel"Physiological mechanisms for the modulation of pannexin 1 channel activity.Skeletal muscle regeneration in mice is stimulated by local overexpression of V1a-vasopressin receptor.Connexins, Pannexins, and Their Channels in Fibroproliferative Diseases IP(3)-dependent, post-tetanic calcium transients induced by electrostimulation of adult skeletal muscle fibers Pannexin 1 constitutes the large conductance cation channel of cardiac myocytes ATP release due to Thy-1-integrin binding induces P2X7-mediated calcium entry required for focal adhesion formation.Electrical stimuli are anti-apoptotic in skeletal muscle via extracellular ATP. Alteration of this signal in Mdx mice is a likely cause of dystrophy.Deficits in endogenous adenosine formation by ecto-5'-nucleotidase/CD73 impair neuromuscular transmission and immune competence in experimental autoimmune myasthenia gravis.Nifedipine treatment reduces resting calcium concentration, oxidative and apoptotic gene expression, and improves muscle function in dystrophic mdx mice.Purinergic effects on Na,K-ATPase activity differ in rat and human skeletal muscle.Anabolic androgenic steroids and intracellular calcium signaling: a mini review on mechanisms and physiological implications.ROS Production via P2Y1-PKC-NOX2 Is Triggered by Extracellular ATP after Electrical Stimulation of Skeletal Muscle Cells.Effect of development on [Ca2+]i transients to ATP in petrosal ganglion neurons: a pharmacological approach using optical recording Vesicular and conductive mechanisms of nucleotide release.Role of connexins and pannexins during ontogeny, regeneration, and pathologies of bone Increased resting intracellular calcium modulates NF-κB-dependent inducible nitric-oxide synthase gene expression in dystrophic mdx skeletal myotubes Hyperthermia increases interleukin-6 in mouse skeletal muscle.Sources of intravascular ATP during exercise in humans: critical role for skeletal muscle perfusion Electrical stimuli release ATP to increase GLUT4 translocation and glucose uptake via PI3Kγ-Akt-AS160 in skeletal muscle cells.Effect of extraluminal ATP application on vascular tone and blood flow in skeletal muscle: implications for exercise hyperemia.Purinergic signalling in the musculoskeletal system.Contribution of intravascular versus interstitial purines and nitric oxide in the regulation of exercise hyperaemia in humans.Connexin- and pannexin-based channels in normal skeletal muscles and their possible role in muscle atrophy.The regulation of interleukin-6 implicates skeletal muscle as an integrative stress sensor and endocrine organ Exercise-induced capillary growth in human skeletal muscle and the dynamics of VEGF.Neuronal involvement in muscular atrophy.Regulation of pannexin and connexin channels and their functional role in skeletal muscles.Vascular function in health, hypertension, and diabetes: effect of physical activity on skeletal muscle microcirculation.Cardiovascular Adaptations to Exercise Training.Characterization of a multiprotein complex involved in excitation-transcription coupling of skeletal muscle.Electrical stimulation induces IL-6 in skeletal muscle through extracellular ATP by activating Ca(2+) signals and an IL-6 autocrine loop.Pannexin 1 channels in skeletal muscles Contracting C2C12 myotubes release CCL2 in an NF-κB-dependent manner to induce monocyte chemoattraction.

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P2860

ATP released by electrical stimuli elicits calcium transients and gene expression in skeletal muscle.

http://www.wikidata.org/entity/Q39788355

description

2009 nî lūn-bûn

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2009年の論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年论文

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2009年论文

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2009年论文

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name

ATP released by electrical sti ...... expression in skeletal muscle.

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ATP released by electrical sti ...... expression in skeletal muscle.

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ATP released by electrical sti ...... expression in skeletal muscle.

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ATP released by electrical sti ...... expression in skeletal muscle.

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ATP released by electrical sti ...... expression in skeletal muscle.

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ATP released by electrical sti ...... expression in skeletal muscle.

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Journal of Biological Chemistry

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ATP released by electrical sti ...... expression in skeletal muscle

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Enrique Jaimovich

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Gonzalo Almarza

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Javiera López

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Juan Carlos Sáez

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Juan Pablo Huidobro-Toro

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Manuel Riquelme

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Mariana Casas

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Sonja Buvinic

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P2860

Skeletal muscle hypertrophy is mediated by a Ca2+-dependent calcineurin signalling pathway

Cloning and expression of a human P2U nucleotide receptor, a target for cystic fibrosis pharmacotherapy

Cloning of P2XM, a novel human P2X receptor gene regulated by p53

Multiple regions of RyR1 mediate functional and structural interactions with alpha(1S)-dihydropyridine receptors in skeletal muscle

International Union of Pharmacology LVIII: update on the P2Y G protein-coupled nucleotide receptors: from molecular mechanisms and pathophysiology to therapy

Single-Step Method of RNA Isolation by Acid Guanidinium Thiocyanate–Phenol–Chloroform Extraction

Molecular physiology of P2X receptors

Identification of a novel human ADP receptor coupled to G(i)

Pannexins and gap junction protein diversity

Pannexin membrane channels are mechanosensitive conduits for ATP

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Mario Bustamante

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