Highly permissive infection of microglial cells by Japanese encephalitis virus: a possible role as a viral reservoir.
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Fenofibrate reduces mortality and precludes neurological deficits in survivors in murine model of Japanese encephalitis viral infectionInteractions of human microglia cells with Japanese encephalitis virusMiR-155 induction in microglial cells suppresses Japanese encephalitis virus replication and negatively modulates innate immune responses.p21-Activated Kinase 4 Signaling Promotes Japanese Encephalitis Virus-Mediated Inflammation in Astrocytes.Zika virus tropism and interactions in myelinating neural cell cultures: CNS cells and myelin are preferentially affected.Neuropathogenesis of Japanese encephalitis in a primate model.NLRP3 inflammasome: key mediator of neuroinflammation in murine Japanese encephalitis.Proteomic analysis of chikungunya virus infected microgial cells.RNase L interacts with Filamin A to regulate actin dynamics and barrier function for viral entrymiR-146a suppresses cellular immune response during Japanese encephalitis virus JaOArS982 strain infection in human microglial cells.The involvement of microglial cells in Japanese encephalitis infections.Japanese Encephalitis Virus exploits the microRNA-432 to regulate the expression of Suppressor of Cytokine Signaling (SOCS) 5Virus-induced transcriptional changes in the brain include the differential expression of genes associated with interferon, apoptosis, interleukin 17 receptor A, and glutamate signaling as well as flavivirus-specific upregulation of tRNA synthetasesNeuroinvasive flavivirus infections.MicroRNA-29b modulates Japanese encephalitis virus-induced microglia activation by targeting tumor necrosis factor alpha-induced protein 3.Regulation of inflammation in Japanese encephalitis.Microarray Analysis Identifies the Potential Role of Long Non-Coding RNA in Regulating Neuroinflammation during Japanese Encephalitis Virus Infection.Characterization of putative Japanese encephalitis virus receptor molecules on microglial cells.Glutamate released by Japanese encephalitis virus-infected microglia involves TNF-α signaling and contributes to neuronal death.Voltage dependent anion channel is redistributed during Japanese encephalitis virus infection of insect cells.Flavivirus Receptors: Diversity, Identity, and Cell Entry
P2860
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P2860
Highly permissive infection of microglial cells by Japanese encephalitis virus: a possible role as a viral reservoir.
description
2009 nî lūn-bûn
@nan
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
2009年论文
@zh
2009年论文
@zh-cn
name
Highly permissive infection of ...... ble role as a viral reservoir.
@en
Highly permissive infection of ...... ble role as a viral reservoir.
@nl
type
label
Highly permissive infection of ...... ble role as a viral reservoir.
@en
Highly permissive infection of ...... ble role as a viral reservoir.
@nl
prefLabel
Highly permissive infection of ...... ble role as a viral reservoir.
@en
Highly permissive infection of ...... ble role as a viral reservoir.
@nl
P2093
P1476
Highly permissive infection of ...... ble role as a viral reservoir.
@en
P2093
Chutima Rattanarungsan
Poonlarp Cheepsunthorn
Thananya Thongtan
Voravasa Chaiworakul
P356
10.1016/J.MICINF.2009.09.013
P577
2009-09-26T00:00:00Z