The TviA auxiliary protein renders the Salmonella enterica serotype Typhi RcsB regulon responsive to changes in osmolarity
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Typhoid fever: "you can't hit what you can't see"Helicobacter and salmonella persistent infection strategiesThe Vi capsular polysaccharide enables Salmonella enterica serovar typhi to evade microbe-guided neutrophil chemotaxisIntraspecies variation in the emergence of hyperinfectious bacterial strains in natureTyphoidal and non-typhoidal Salmonella infections in Africa.Salmonella enterica Serovar Typhi conceals the invasion-associated type three secretion system from the innate immune system by gene regulation.Salmonella enterica serovar Typhi impairs CD4 T cell responses by reducing antigen availabilityContrasting persistence strategies in Salmonella and MycobacteriumA rapid change in virulence gene expression during the transition from the intestinal lumen into tissue promotes systemic dissemination of Salmonella.Taming the elephant: Salmonella biology, pathogenesis, and prevention.Live recombinant Salmonella Typhi vaccines constructed to investigate the role of rpoS in eliciting immunity to a heterologous antigenDynamic modularity of host protein interaction networks in Salmonella Typhi infection.The flagellar regulator fliT represses Salmonella pathogenicity island 1 through flhDC and fliZ.In vivo regulation of the Vi antigen in Salmonella and induction of immune responses with an in vivo-inducible promoterThe flagellar regulator TviA reduces pyroptosis by Salmonella enterica serovar TyphiIntegrated stress responses in Salmonella.Non-typhoidal Salmonella Typhimurium ST313 isolates that cause bacteremia in humans stimulate less inflammasome activation than ST19 isolates associated with gastroenteritisRegulation of virulence: the rise and fall of gastrointestinal pathogens.Flagellin Is Required for Host Cell Invasion and Normal Salmonella Pathogenicity Island 1 Expression by Salmonella enterica Serovar Paratyphi AThe response regulator RcsB activates expression of Mat fimbriae in meningitic Escherichia coliDifferences in Host Cell Invasion and Salmonella Pathogenicity Island 1 Expression between Salmonella enterica Serovar Paratyphi A and Nontyphoidal S. TyphimuriumLoss of very-long O-antigen chains optimizes capsule-mediated immune evasion by Salmonella enterica serovar Typhi.Host specificity of bacterial pathogens.Live Attenuated Human Salmonella Vaccine Candidates: Tracking the Pathogen in Natural Infection and Stimulation of Host ImmunityComparative analysis of Salmonella genomes identifies a metabolic network for escalating growth in the inflamed gut.The Rcs phosphorelay: more than just a two-component pathway.RcsB is required for inducible acid resistance in Escherichia coli and acts at gadE-dependent and -independent promoters.Chronic Bacterial Pathogens: Mechanisms of Persistence.RcsB positively regulates the Yersinia Ysc-Yop type III secretion system by activating expression of the master transcriptional regulator LcrF.Potassium transport of Salmonella is important for type III secretion and pathogenesis.BglJ-RcsB heterodimers relieve repression of the Escherichia coli bgl operon by H-NS.Vi capsular polysaccharide: Synthesis, virulence, and application.The transcriptional regulator SsrB is involved in a molecular switch controlling virulence lifestyles of Salmonella.A genomewide mutagenesis screen identifies multiple genes contributing to Vi capsular expression in Salmonella enterica serovar Typhi.The Periplasmic Nitrate Reductase NapABC Supports Luminal Growth of Salmonella enterica Serovar Typhimurium during Colitis.New Insights into the Non-orthodox Two Component Rcs Phosphorelay System.Bi-valent polysaccharides of Vi capsular and O9 O-antigen in attenuated Salmonella Typhimurium induce strong immune responses against these two antigens.Mechanisms to Evade the Phagocyte Respiratory Burst Arose by Convergent Evolution in Typhoidal Salmonella Serovars.OmpR may regulate the putative YehU/YehT two-component system in Salmonella enterica serovar Typhi under hypotonic growth condition.Why Is Eradicating Typhoid Fever So Challenging: Implications for Vaccine and Therapeutic Design
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P2860
The TviA auxiliary protein renders the Salmonella enterica serotype Typhi RcsB regulon responsive to changes in osmolarity
description
2009 nî lūn-bûn
@nan
2009年の論文
@ja
2009年学术文章
@wuu
2009年学术文章
@zh-cn
2009年学术文章
@zh-hans
2009年学术文章
@zh-my
2009年学术文章
@zh-sg
2009年學術文章
@yue
2009年學術文章
@zh
2009年學術文章
@zh-hant
name
The TviA auxiliary protein ren ...... nsive to changes in osmolarity
@en
The TviA auxiliary protein ren ...... sive to changes in osmolarity.
@nl
type
label
The TviA auxiliary protein ren ...... nsive to changes in osmolarity
@en
The TviA auxiliary protein ren ...... sive to changes in osmolarity.
@nl
prefLabel
The TviA auxiliary protein ren ...... nsive to changes in osmolarity
@en
The TviA auxiliary protein ren ...... sive to changes in osmolarity.
@nl
P2093
P2860
P50
P1476
The TviA auxiliary protein ren ...... nsive to changes in osmolarity
@en
P2093
Holger Rüssmann
Maria G Winter
Sebastian E Winter
P2860
P304
P356
10.1111/J.1365-2958.2009.06859.X
P407
P577
2009-08-24T00:00:00Z