Expression and regulation of the NALP3 inflammasome complex in periodontal diseases. - Wikidata - awgldk - TriplyDB

Expression and regulation of the NALP3 inflammasome complex in periodontal diseases.

Expression and regulation of the NALP3 inflammasome complex in periodontal diseases.

http://www.wikidata.org/entity/Q39815306

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Modulation of inflammasome activity by Porphyromonas gingivalis in periodontitis and associated systemic diseases Periodontal disease immunology: 'double indemnity' in protecting the host Porphyromonas gingivalis mediates inflammasome repression in polymicrobial cultures through a novel mechanism involving reduced endocytosis Chrysophanol inhibits NALP3 inflammasome activation and ameliorates cerebral ischemia/reperfusion in mice Pathogen-mediated proteolysis of the cell death regulator RIPK1 and the host defense modulator RIPK2 in human aortic endothelial cells.Porphyromonas gingivalis fimbriae dampen P2X7-dependent interleukin-1β secretion.An inflammasome-independent role for epithelial-expressed Nlrp3 in renal ischemia-reperfusion injury The activation of pyrin domain-containing-3 inflammasome depends on lipopolysaccharide from Porphyromonas gingivalis and extracellular adenosine triphosphate in cultured oral epithelial cells A Dual Role for P2X7 Receptor during Porphyromonas gingivalis Infection Dental Calculus Stimulates Interleukin-1β Secretion by Activating NLRP3 Inflammasome in Human and Mouse Phagocytes.Effects of aging in the expression of NOD-like receptors and inflammasome-related genes in oral mucosa.Inhibition of pre-existing natural periodontitis in non-human primates by a locally administered peptide inhibitor of complement C3 Complement inhibition in pre-clinical models of periodontitis and prospects for clinical application Sterol Regulatory Element-Binding Protein-1c Regulates Inflammasome Activation in Gingival Fibroblasts Infected with High-Glucose-Treated Porphyromonas gingivalis.Activation of NLRP1 and NLRP3 inflammasomes contributed to cyclic stretch-induced pyroptosis and release of IL-1β in human periodontal ligament cells.How has research into cytokine interactions and their role in driving immune responses impacted our understanding of periodontitis?The inflammasome in host defense.The role of the inflammasome in cardiovascular diseases.The inflammasome and danger molecule signaling: at the crossroads of inflammation and pathogen persistence in the oral cavity.Metabolic Remodeling, Inflammasome Activation, and Pyroptosis in Macrophages Stimulated by Porphyromonas gingivalis and Its Outer Membrane Vesicles.Involvement of NLRP10 in IL-1α induction of oral epithelial cells by periodontal pathogens.ASC-dependent RIP2 kinase regulates reduced PGE2 production in chronic periodontitis.The Immune Response in Periodontal Tissues.The Role of Reactive Oxygen Species and Autophagy in Periodontitis and Their Potential Linkage.Necroptosis in the periodontal homeostasis: Signals emanating from dying cells.ATP-dependent activation of an inflammasome in primary gingival epithelial cells infected by Porphyromonas gingivalis The actin-bundling protein L-plastin: a novel local inflammatory marker associated with periodontitis.Titanium ions form particles that activate and execute interleukin-1β release from lipopolysaccharide-primed macrophages.Immunohistochemical localization of NALP3 inflammasome in experimental periapical lesions.Porphyromonas gingivalis-impaired innate immune response via NLRP3 proteolysis in endothelial cells.New evidence of connections between increased O-GlcNAcylation and inflammasome in the oral mucosa of patients with oral lichen planus.Salivary Levels of NLRP3 Inflammasome-Related Proteins as Potential Biomarkers of Periodontal Clinical Status.Aggregatibacter actinomycetemcomitans induces Th17 cells in atherosclerotic lesions.Gene expression of transcription factor NFATc1 in periodontal diseases.The inflammasomes, immune guardians at defence barriers

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P2860

Expression and regulation of the NALP3 inflammasome complex in periodontal diseases.

http://www.wikidata.org/entity/Q39815306

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2009年の論文

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2009年学术文章

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2009年學術文章

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2009年學術文章

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name

Expression and regulation of the NALP3 inflammasome complex in periodontal diseases.

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Expression and regulation of the NALP3 inflammasome complex in periodontal diseases.

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type

label

Expression and regulation of the NALP3 inflammasome complex in periodontal diseases.

@en

Expression and regulation of the NALP3 inflammasome complex in periodontal diseases.

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prefLabel

Expression and regulation of the NALP3 inflammasome complex in periodontal diseases.

@en

Expression and regulation of the NALP3 inflammasome complex in periodontal diseases.

@nl

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Clinical and Experimental Immunology

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Expression and regulation of the NALP3 inflammasome complex in periodontal diseases

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P2860

Mutation of a new gene encoding a putative pyrin-like protein causes familial cold autoinflammatory syndrome and Muckle-Wells syndrome

CATERPILLER: a novel gene family important in immunity, cell death, and diseases

Cutting edge: ASC mediates the induction of multiple cytokines by Porphyromonas gingivalis via caspase-1-dependent and -independent pathways

The inflammasome: a molecular platform triggering activation of inflammatory caspases and processing of proIL-beta

Cryopyrin-induced interleukin 1beta secretion in monocytic cells: enhanced activity of disease-associated mutants and requirement for ASC

NALP3 forms an IL-1beta-processing inflammasome with increased activity in Muckle-Wells autoinflammatory disorder

Inflammasome components NALP 1 and 3 show distinct but separate expression profiles in human tissues suggesting a site-specific role in the inflammatory response

Function of Nod-like receptors in microbial recognition and host defense

Regulation of cryopyrin/Pypaf1 signaling by pyrin, the familial Mediterranean fever gene product

The PYRIN-CARD protein ASC is an activating adaptor for caspase-1

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