Escherichia coli F-18 and E. coli K-12 eda mutants do not colonize the streptomycin-treated mouse large intestine
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What's for dinner?: Entner-Doudoroff metabolism in Escherichia coli.Sequence analysis of the GntII (subsidiary) system for gluconate metabolism reveals a novel pathway for L-idonic acid catabolism in Escherichia coli.Structural insight into the catalytic mechanism of gluconate 5-dehydrogenase fromStreptococcus suis: Crystal structures of the substrate-free and quaternary complex enzymesIn vivo application of photocleavable protein interaction reporter technology.The outer mucus layer hosts a distinct intestinal microbial niche.Estimation of growth rates of Escherichia coli BJ4 in streptomycin-treated and previously germfree mice by in situ rRNA hybridization.The Rhomboid Protease GlpG Promotes the Persistence of Extraintestinal Pathogenic Escherichia coli within the GutMetabolic Adaptations of Uropathogenic E. coli in the Urinary Tract.Role of intracellular carbon metabolism pathways in Shigella flexneri virulence.Carbon nutrition of Escherichia coli in the mouse intestine.Induction of protease activity in Vibrio anguillarum by gastrointestinal mucusThe Entner-Doudoroff pathway is obligatory for gluconate utilization and contributes to the pathogenicity of Vibrio cholerae.Nucleoid remodeling by an altered HU protein: reorganization of the transcription program.Microbial imbalance and intestinal pathologies: connections and contributionsGluconate 5-dehydrogenase (Ga5DH) participates in Streptococcus suis cell division.Molecular genetic characterization of the Escherichia coli gntT gene of GntI, the main system for gluconate metabolism.Comparative genome analysis of Salmonella Enteritidis PT4 and Salmonella Gallinarum 287/91 provides insights into evolutionary and host adaptation pathways.Genes of the GadX-GadW regulon in Escherichia coli.There is a specific response to pH by isolates of Haemophilus influenzae and this has a direct influence on biofilm formationBacterial protein AvrA stabilizes intestinal epithelial tight junctions via blockage of the C-Jun N-terminal kinase pathway.Commensal and Pathogenic Escherichia coli Metabolism in the Gut.Adaptive immunity increases the pace and predictability of evolutionary change in commensal gut bacteria.L-fucose stimulates utilization of D-ribose by Escherichia coli MG1655 DeltafucAO and E. coli Nissle 1917 DeltafucAO mutants in the mouse intestine and in M9 minimal medium.The salmonella transcriptome in lettuce and cilantro soft rot reveals a niche overlap with the animal host intestine.Identification of genes associated with the long-gut-persistence phenotype of the probiotic Lactobacillus johnsonii strain NCC533 using a combination of genomics and transcriptome analysisGlycolytic strategy as a tradeoff between energy yield and protein cost.Streptomycin-induced inflammation enhances Escherichia coli gut colonization through nitrate respiration.Host-Derived Sialic Acids Are an Important Nutrient Source Required for Optimal Bacterial Fitness In VivoThe Pic protease of enteroaggregative Escherichia coli promotes intestinal colonization and growth in the presence of mucin.A temperature-regulated Campylobacter jejuni gluconate dehydrogenase is involved in respiration-dependent energy conservation and chicken colonization.Sucrose metabolism contributes to in vivo fitness of Streptococcus pneumoniae.Escherichia coli pathotypes occupy distinct niches in the mouse intestine.Positive and negative transcriptional regulation of the Escherichia coli gluconate regulon gene gntT by GntR and the cyclic AMP (cAMP)-cAMP receptor protein complexAn Escherichia coli MG1655 lipopolysaccharide deep-rough core mutant grows and survives in mouse cecal mucus but fails to colonize the mouse large intestine.Mouse intestine selects nonmotile flhDC mutants of Escherichia coli MG1655 with increased colonizing ability and better utilization of carbon sources.The wax moth Galleria mellonella as a novel model system to study Enteroaggregative Escherichia coli pathogenesis.Glycolytic and gluconeogenic growth of Escherichia coli O157:H7 (EDL933) and E. coli K-12 (MG1655) in the mouse intestine.Multiple regulators control expression of the Entner-Doudoroff aldolase (Eda) of Escherichia coli.The streptomycin-treated mouse intestine selects Escherichia coli envZ missense mutants that interact with dense and diverse intestinal microbiota.Role of motility and the flhDC Operon in Escherichia coli MG1655 colonization of the mouse intestine
P2860
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P2860
Escherichia coli F-18 and E. coli K-12 eda mutants do not colonize the streptomycin-treated mouse large intestine
description
1996 nî lūn-bûn
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1996年の論文
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1996年学术文章
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1996年学术文章
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1996年学术文章
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1996年学术文章
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1996年学术文章
@zh-sg
1996年學術文章
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1996年學術文章
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1996年學術文章
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name
Escherichia coli F-18 and E. c ...... -treated mouse large intestine
@en
Escherichia coli F-18 and E. c ...... treated mouse large intestine.
@nl
type
label
Escherichia coli F-18 and E. c ...... -treated mouse large intestine
@en
Escherichia coli F-18 and E. c ...... treated mouse large intestine.
@nl
prefLabel
Escherichia coli F-18 and E. c ...... -treated mouse large intestine
@en
Escherichia coli F-18 and E. c ...... treated mouse large intestine.
@nl
P2093
P2860
P1476
Escherichia coli F-18 and E. c ...... -treated mouse large intestine
@en
P2093
P2860
P304
P407
P577
1996-09-01T00:00:00Z