Bacteroides fragilis toxin exhibits polar activity on monolayers of human intestinal epithelial cells (T84 cells) in vitro
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A key role for E-cadherin in intestinal homeostasis and Paneth cell maturationEnterotoxigenic Bacteroides fragilis: a rogue among symbiotes.The Bacteroides fragilis toxin binds to a specific intestinal epithelial cell receptorThe Bacteroides fragilis pathogenicity island is contained in a putative novel conjugative transposonNSP4 enterotoxin of rotavirus induces paracellular leakage in polarized epithelial cellsMutation of the zinc-binding metalloprotease motif affects Bacteroides fragilis toxin activity but does not affect propeptide processing.The alleles of the bft gene are distributed differently among enterotoxigenic Bacteroides fragilis strains from human sources and can be present in double copies.Molecular evolution of the pathogenicity island of enterotoxigenic Bacteroides fragilis strains.Identification of a third metalloprotease toxin gene in extraintestinal isolates of Bacteroides fragilis.Stat3 activation in murine colitis induced by enterotoxigenic Bacteroides fragilis.Bacteroides fragilis subverts mucosal biology: from symbiont to colon carcinogenesis.Bacteroides fragilis enterotoxin cleaves the zonula adherens protein, E-cadherin.Intestinal epithelial responses to enteric pathogens: effects on the tight junction barrier, ion transport, and inflammation.The C-terminal region of Bacteroides fragilis toxin is essential to its biological activityInduction of persistent colitis by a human commensal, enterotoxigenic Bacteroides fragilis, in wild-type C57BL/6 miceBacteroides fragilis enterotoxin induces intestinal epithelial cell secretion of interleukin-8 through mitogen-activated protein kinases and a tyrosine kinase-regulated nuclear factor-kappaB pathway.Nuclear factor-kappa B activation pathway in intestinal epithelial cells is a major regulator of chemokine gene expression and neutrophil migration induced by Bacteroides fragilis enterotoxin.Diversity of the metalloprotease toxin produced by enterotoxigenic Bacteroides fragilis.Polarized secretion of CXC chemokines by human intestinal epithelial cells in response to Bacteroides fragilis enterotoxin: NF-kappa B plays a major role in the regulation of IL-8 expression.Bacteroides fragilis enterotoxin induces the expression of IL-8 and transforming growth factor-beta (TGF-beta) by human colonic epithelial cells.Bacteroides fragilis toxin stimulates intestinal epithelial cell shedding and gamma-secretase-dependent E-cadherin cleavage.Bacteroides fragilis toxin 2 damages human colonic mucosa in vitro
P2860
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P2860
Bacteroides fragilis toxin exhibits polar activity on monolayers of human intestinal epithelial cells (T84 cells) in vitro
description
1997 nî lūn-bûn
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1997年の論文
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1997年学术文章
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1997年学术文章
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name
Bacteroides fragilis toxin exh ...... ial cells (T84 cells) in vitro
@en
Bacteroides fragilis toxin exh ...... an intestinal epithelial cells
@nl
type
label
Bacteroides fragilis toxin exh ...... ial cells (T84 cells) in vitro
@en
Bacteroides fragilis toxin exh ...... an intestinal epithelial cells
@nl
prefLabel
Bacteroides fragilis toxin exh ...... ial cells (T84 cells) in vitro
@en
Bacteroides fragilis toxin exh ...... an intestinal epithelial cells
@nl
P2093
P2860
P1476
Bacteroides fragilis toxin exh ...... ial cells (T84 cells) in vitro
@en
P2093
P2860
P304
P407
P577
1997-09-01T00:00:00Z