KLF4 suppresses estrogen-dependent breast cancer growth by inhibiting the transcriptional activity of ERalpha.
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Mammalian Krüppel-like factors in health and diseasesCancer Stem Cell Hierarchy in Glioblastoma MultiformeThe network of pluripotency, epithelial-mesenchymal transition, and prognosis of breast cancerSmall ubiquitin-like modifier (SUMO) modification of zinc finger protein 131 potentiates its negative effect on estrogen signalingOverexpression of NSUN2 by DNA hypomethylation is associated with metastatic progression in human breast cancer.Expression of embryonal stem cell transcription factors in breast cancer: Oct4 as an indicator for poor clinical outcome and tamoxifen resistance.Deregulated estrogen receptor alpha and p53 heterozygosity collaborate in the development of mammary hyperplasia.Kruppel-like factor 4 inhibits epithelial-to-mesenchymal transition through regulation of E-cadherin gene expressionDoxycycline inducible Krüppel-like factor 4 lentiviral vector mediates mesenchymal to epithelial transition in ovarian cancer cells.ERK5/KLF4 signaling as a common mediator of the neuroprotective effects of both nerve growth factor and hydrogen peroxide preconditioning.Brucine suppresses colon cancer cells growth via mediating KDR signalling pathway.Integrative eQTL-based analyses reveal the biology of breast cancer risk lociCritical and reciprocal regulation of KLF4 and SLUG in transforming growth factor β-initiated prostate cancer epithelial-mesenchymal transitionUncoupled embryonic and extra-embryonic tissues compromise blastocyst development after somatic cell nuclear transfer.Krüppel-like factor 4 induces apoptosis and inhibits tumorigenic progression in SK-BR-3 breast cancer cells.N-3 poly-unsaturated fatty acids shift estrogen signaling to inhibit human breast cancer cell growth.Klf4 is a transcriptional regulator of genes critical for EMT, including Jnk1 (Mapk8).MDM2 regulates estrogen receptor α and estrogen responsiveness in breast cancer cellsKrüppel-like factor 4 inhibits tumorigenic progression and metastasis in a mouse model of breast cancer.Krüppel-like factors are effectors of nuclear receptor signaling.Nuclear factor I-C regulates E-cadherin via control of KLF4 in breast cancerc-Myc plays a key role in TADs-induced apoptosis and cell cycle arrest in human hepatocellular carcinoma cells.AGPAT9 suppresses cell growth, invasion and metastasis by counteracting acidic tumor microenvironment through KLF4/LASS2/V-ATPase signaling pathway in breast cancerA novel angiogenesis inhibitor impairs lovo cell survival via targeting against human VEGFR and its signaling pathway of phosphorylationExpression of the Pluripotency Transcription Factor OCT4 in the Normal and Aberrant Mammary GlandLoss of epigenetic Kruppel-like factor 4 histone deacetylase (KLF-4-HDAC)-mediated transcriptional suppression is crucial in increasing vascular endothelial growth factor (VEGF) expression in breast cancer.Tumor repressor protein 53 and steroid hormones provide a new paradigm for ovarian cancer metastases.KLF4α stimulates breast cancer cell proliferation by acting as a KLF4 antagonist.The miR-7 identified from collagen biomaterial-based three-dimensional cultured cells regulates neural stem cell differentiation.FOXC1 is associated with estrogen receptor alpha and affects sensitivity of tamoxifen treatment in breast cancerKrüppel-like factors 4 and 5: unity in diversityKrüppel-like factors in cancer progression: three fingers on the steering wheel.Kruppel-like factor 4 inhibits non-small cell lung cancer cell growth and aggressiveness by stimulating transforming growth factor-β1-meidated ERK/JNK/NF-κB signaling pathways.KLF4 Is Essential for Induction of Cellular Identity Change and Acinar-to-Ductal Reprogramming during Early Pancreatic Carcinogenesis.A functional interplay between ZNF217 and estrogen receptor alpha exists in luminal breast cancers.Upregulation of KLF4 by methylseleninic acid in human esophageal squamous cell carcinoma cells: Modification of histone H3 acetylation through HAT/HDAC interplay.Novel breast cancer susceptibility locus at 9q31.2: results of a genome-wide association study.Krüppel-like factor 4 (KLF4): What we currently know.Mutant p53 binds to estrogen receptor negative promoter via DNMT1 and HDAC1 in MDA-MB-468 breast cancer cells.Krüppel-like factor 4 (KLF4) suppresses neuroblastoma cell growth and determines non-tumorigenic lineage differentiation.
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P2860
KLF4 suppresses estrogen-dependent breast cancer growth by inhibiting the transcriptional activity of ERalpha.
description
2009 nî lūn-bûn
@nan
2009年の論文
@ja
2009年学术文章
@wuu
2009年学术文章
@zh-cn
2009年学术文章
@zh-hans
2009年学术文章
@zh-my
2009年学术文章
@zh-sg
2009年學術文章
@yue
2009年學術文章
@zh
2009年學術文章
@zh-hant
name
KLF4 suppresses estrogen-depen ...... riptional activity of ERalpha.
@en
KLF4 suppresses estrogen-depen ...... riptional activity of ERalpha.
@nl
type
label
KLF4 suppresses estrogen-depen ...... riptional activity of ERalpha.
@en
KLF4 suppresses estrogen-depen ...... riptional activity of ERalpha.
@nl
prefLabel
KLF4 suppresses estrogen-depen ...... riptional activity of ERalpha.
@en
KLF4 suppresses estrogen-depen ...... riptional activity of ERalpha.
@nl
P2093
P2860
P356
P1433
P1476
KLF4 suppresses estrogen-depen ...... riptional activity of ERalpha.
@en
P2093
A Murayama
J Yanagisawa
S Kawasaki
Y Nakajima
P2860
P2888
P304
P356
10.1038/ONC.2009.151
P407
P577
2009-06-08T00:00:00Z