Anchorage-independent cell growth signature identifies tumors with metastatic potential.
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Oxygen in human health from life to death--An approach to teaching redox biology and signaling to graduate and medical studentsProtein interacting with C kinase 1 suppresses invasion and anchorage-independent growth of astrocytic tumor cellsSynergistic inhibitory effect of hyperbaric oxygen combined with sorafenib on hepatoma cellsEvidence that selenium binding protein 1 is a tumor suppressor in prostate cancerMitochondrial metabolism and ROS generation are essential for Kras-mediated tumorigenicityp140Cap suppresses the invasive properties of highly metastatic MTLn3-EGFR cells via impaired cortactin phosphorylation.siRNA knockdown of ribosomal protein gene RPL19 abrogates the aggressive phenotype of human prostate cancer.Targeting Colorectal Cancer Proliferation, Stemness and Metastatic Potential Using Brassicaceae Extracts Enriched in Isothiocyanates: A 3D Cell Model-Based Study.Deficiency of CCN5/WISP-2-Driven Program in breast cancer Promotes Cancer Epithelial cells to mesenchymal stem cells and Breast Cancer growth.Combined treatment with Metformin and 2-deoxy glucose induces detachment of viable MDA-MB-231 breast cancer cells in vitro.Ultrafine particles of Ulmus davidiana var. japonica induce apoptosis of gastric cancer cells via activation of caspase and endoplasmic reticulum stressCombinatorial treatment with polyI:C and anti-IL6 enhances apoptosis and suppresses metastasis of lung cancer cells.Genome-scale metabolic modeling elucidates the role of proliferative adaptation in causing the Warburg effect.Therapeutic potential of PRL-3 targeting and clinical significance of PRL-3 genomic amplification in gastric cancerInternal ribosome entry site of bFGF is the target of thalidomide for IMiDs development in multiple myeloma.The application of gene co-expression network reconstruction based on CNVs and gene expression microarray data in breast cancer.Myc-induced microRNAs integrate Myc-mediated cell proliferation and cell fate.Lysine-specific demethylase 2B (KDM2B)-let-7-enhancer of zester homolog 2 (EZH2) pathway regulates cell cycle progression and senescence in primary cells.Most random gene expression signatures are significantly associated with breast cancer outcome.Missense mutations in pyruvate kinase M2 promote cancer metabolism, oxidative endurance, anchorage independence, and tumor growth in a dominant negative manner.Metabolic imaging: a link between lactate dehydrogenase A, lactate, and tumor phenotype.Phenotypes and karyotypes of human malignant mesothelioma cell lines.Homeoprotein Six2 promotes breast cancer metastasis via transcriptional and epigenetic control of E-cadherin expression.N-glycosylation of ICAM-2 is required for ICAM-2-mediated complete suppression of metastatic potential of SK-N-AS neuroblastoma cellsA systematic review of non-hormonal treatments of vasomotor symptoms in climacteric and cancer patientsTransketolase is upregulated in metastatic peritoneal implants and promotes ovarian cancer cell proliferation.Protein kinase C δ is a downstream effector of oncogenic K-ras in lung tumors.The Interactions between Insulin and Androgens in Progression to Castrate-Resistant Prostate CancerPhosphatidylinositol Phosphate 5-Kinase Iγ and Phosphoinositide 3-Kinase/Akt Signaling Couple to Promote Oncogenic Growth.Curcumin inhibits anchorage-independent growth of HT29 human colon cancer cells by targeting epigenetic restoration of the tumor suppressor gene DLEC1Establishment and characterization of a receptor-negative, hormone-nonresponsive breast cancer cell line from an Iraqi patientA murine model for the development of melanocytic nevi and their progression to melanoma.Non-NAD-Like poly(ADP-Ribose) Polymerase-1 Inhibitors effectively Eliminate Cancer in vivoDiverse roles for the paxillin family of proteins in cancer.Depleting Tumor-NQO1 Potentiates Anoikis and Inhibits Growth of NSCLC.TTK activates Akt and promotes proliferation and migration of hepatocellular carcinoma cells.Role of metabolic modulator Bet-CA in altering mitochondrial hyperpolarization to suppress cancer associated angiogenesis and metastasis.The human gastric cancer-associated DNA polymerase β variant D160N is a mutator that induces cellular transformation.Functional genomics identifies five distinct molecular subtypes with clinical relevance and pathways for growth control in epithelial ovarian cancer.Na+/H+ exchanger NHE1 regulation modulates metastatic potential and epithelial-mesenchymal transition of triple-negative breast cancer cells.
P2860
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P2860
Anchorage-independent cell growth signature identifies tumors with metastatic potential.
description
2009 nî lūn-bûn
@nan
2009年の論文
@ja
2009年学术文章
@wuu
2009年学术文章
@zh-cn
2009年学术文章
@zh-hans
2009年学术文章
@zh-my
2009年学术文章
@zh-sg
2009年學術文章
@yue
2009年學術文章
@zh
2009年學術文章
@zh-hant
name
Anchorage-independent cell growth signature identifies tumors with metastatic potential.
@en
Anchorage-independent cell growth signature identifies tumors with metastatic potential.
@nl
type
label
Anchorage-independent cell growth signature identifies tumors with metastatic potential.
@en
Anchorage-independent cell growth signature identifies tumors with metastatic potential.
@nl
prefLabel
Anchorage-independent cell growth signature identifies tumors with metastatic potential.
@en
Anchorage-independent cell growth signature identifies tumors with metastatic potential.
@nl
P2093
P2860
P356
P1433
P1476
Anchorage-independent cell growth signature identifies tumors with metastatic potential.
@en
P2093
J R Nevins
N Matsumura
P2860
P2888
P304
P356
10.1038/ONC.2009.139
P407
P577
2009-06-01T00:00:00Z