STAT1 contributes to the maintenance of the latency III viral programme observed in Epstein-Barr virus-transformed B cells and their recognition by CD8+ T cells.
about
Gene expression analysis of whole blood, peripheral blood mononuclear cells, and lymphoblastoid cell lines from the Framingham Heart Study.Human Cytomegalovirus Immediate-Early 1 Protein Rewires Upstream STAT3 to Downstream STAT1 Signaling Switching an IL6-Type to an IFNγ-Like ResponseAllele-Independent Turnover of Human Leukocyte Antigen (HLA) Class Ia MoleculesAttack, parry and riposte: molecular fencing between the innate immune system and human herpesviruses.Deletion of Kaposi's sarcoma-associated herpesvirus FLICE inhibitory protein, vFLIP, from the viral genome compromises the activation of STAT1-responsive cellular genes and spindle cell formation in endothelial cells
P2860
STAT1 contributes to the maintenance of the latency III viral programme observed in Epstein-Barr virus-transformed B cells and their recognition by CD8+ T cells.
description
2009 nî lūn-bûn
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2009年の論文
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name
STAT1 contributes to the maint ...... r recognition by CD8+ T cells.
@en
STAT1 contributes to the maint ...... r recognition by CD8+ T cells.
@nl
type
label
STAT1 contributes to the maint ...... r recognition by CD8+ T cells.
@en
STAT1 contributes to the maint ...... r recognition by CD8+ T cells.
@nl
prefLabel
STAT1 contributes to the maint ...... r recognition by CD8+ T cells.
@en
STAT1 contributes to the maint ...... r recognition by CD8+ T cells.
@nl
P2093
P50
P356
P1476
STAT1 contributes to the maint ...... r recognition by CD8+ T cells.
@en
P2093
James E McLaren
Julia Grimstead
Zara Poghosyan
P304
P356
10.1099/VIR.0.011627-0
P407
P577
2009-05-13T00:00:00Z