Cytoskeletal remodeling of desmin is a more accurate measure of cardiac dysfunction than fibrosis or myocyte hypertrophy.
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Adverse perinatal environment contributes to altered cardiac development and function.Influence of heart failure on nucleocytoplasmic transport in human cardiomyocytes.Transmural heterogeneity of cellular level power output is reduced in human heart failure.Early life exposure to air pollution induces adult cardiac dysfunction.Comparison of the ventricle muscle proteome between patients with rheumatic heart disease and controls with mitral valve prolapse: HSP 60 may be a specific protein in RHD.Cardiomyocyte mitochondrial oxidative stress and cytoskeletal breakdown in the heart with a primary volume overload.Cardiac protein changes in rats after soybean oil treatment: a proteomic study.In vitro particulate matter exposure causes direct and lung-mediated indirect effects on cardiomyocyte function.Losartan treatment attenuates tumor-induced myocardial dysfunctionCardiovascular remodeling in response to long-term exposure to fine particulate matter air pollutionCardiac involvement in hereditary myopathy with early respiratory failure: A cohort studyRNA-sequencing analysis reveals new alterations in cardiomyocyte cytoskeletal genes in patients with heart failure.Desmin A213V substitution represents a rare polymorphism but not a mutation and is more prevalent in patients with heart dilation of various origins.Right ventricular remodeling in restrictive ventricular septal defect.Modulation of stretch-induced myocyte remodeling and gene expression by nitric oxide: a novel role for lipoma preferred partner in myofibrillogenesis.Effect of ouabain on myocardial ultrastructure and cytoskeleton during the development of ventricular hypertrophy.Early dystrophin loss is coincident with the transition of compensated cardiac hypertrophy to heart failure.Perinatal Inflammation Induces Sex-related Differences in Cardiovascular Morbidities in Mice.Pregestational type 2 diabetes mellitus induces cardiac hypertrophy in the murine embryo through cardiac remodeling and fibrosis.Type of desmin expression in cardiomyocytes - a good marker of heart failure development in idiopathic dilated cardiomyopathy.Cardiac protein expression patterns are associated with distinct inborn exercise capacity in non-selectively bred rats.A Multidisciplinary Assessment of Remote Myocardial Fibrosis After Reperfused Myocardial Infarction in Swine and Patients.
P2860
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P2860
Cytoskeletal remodeling of desmin is a more accurate measure of cardiac dysfunction than fibrosis or myocyte hypertrophy.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
2008年论文
@zh
2008年论文
@zh-cn
name
Cytoskeletal remodeling of des ...... brosis or myocyte hypertrophy.
@en
Cytoskeletal remodeling of des ...... brosis or myocyte hypertrophy.
@nl
type
label
Cytoskeletal remodeling of des ...... brosis or myocyte hypertrophy.
@en
Cytoskeletal remodeling of des ...... brosis or myocyte hypertrophy.
@nl
prefLabel
Cytoskeletal remodeling of des ...... brosis or myocyte hypertrophy.
@en
Cytoskeletal remodeling of des ...... brosis or myocyte hypertrophy.
@nl
P2093
P1433
P1476
Cytoskeletal remodeling of des ...... brosis or myocyte hypertrophy.
@en
P2093
Alistair B Phillips
Gretel Monreal
John Anthony Bauer
Lisa M Nicholson
Loren E Wold
Mandar S Joshi
Mark A Gerhardt
P304
P356
10.1016/J.LFS.2008.09.026
P407
P577
2008-10-15T00:00:00Z