Enterovirus 71 induces dsRNA/PKR-dependent cytoplasmic redistribution of GRP78/BiP to promote viral replication.
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dsRNA Binding Domain of PKR Is Proteolytically Released by Enterovirus A71 to Facilitate Viral Replication.iTRAQ-based Proteomic Analysis of Porcine Kidney Epithelial PK15 cells Infected with Pseudorabies virus.Recombinant heat shock protein 78 enhances enterovirus 71 propagation in Vero cells and is induced in SK-N-SH cells during the infection.HERP Binds TBK1 To Activate Innate Immunity and Repress Virus Replication in Response to Endoplasmic Reticulum Stress.The endoplasmic reticulum-residing chaperone BiP is short-lived and metabolized through N-terminal arginylation.A novel role of ER stress signal transducer ATF6 in regulating enterovirus A71 viral protein stability.
P2860
Enterovirus 71 induces dsRNA/PKR-dependent cytoplasmic redistribution of GRP78/BiP to promote viral replication.
description
2016 nî lūn-bûn
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2016年の論文
@ja
2016年論文
@yue
2016年論文
@zh-hant
2016年論文
@zh-hk
2016年論文
@zh-mo
2016年論文
@zh-tw
2016年论文
@wuu
2016年论文
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2016年论文
@zh-cn
name
Enterovirus 71 induces dsRNA/P ...... to promote viral replication.
@en
Enterovirus 71 induces dsRNA/P ...... to promote viral replication.
@nl
type
label
Enterovirus 71 induces dsRNA/P ...... to promote viral replication.
@en
Enterovirus 71 induces dsRNA/P ...... to promote viral replication.
@nl
prefLabel
Enterovirus 71 induces dsRNA/P ...... to promote viral replication.
@en
Enterovirus 71 induces dsRNA/P ...... to promote viral replication.
@nl
P2093
P2860
P356
P1476
Enterovirus 71 induces dsRNA/P ...... to promote viral replication.
@en
P2093
Chao-Rih Jheng
Jia-Rong Jheng
Jim-Tong Horng
Shin-Chyang Wang
P2860
P356
10.1038/EMI.2016.20
P577
2016-03-23T00:00:00Z