Requirement for vacuolar proton-ATPase activity during entry of influenza virus into cells.
about
Mechanisms of viral entry: sneaking in the front doorRole of a Transbilayer pH Gradient in the Membrane Fusion Activity of the Influenza Virus Hemagglutinin: Use of the R18 Assay to Monitor Membrane Merging.Discovery of mammalian genes that participate in virus infectionToll-like receptor-mediated activation of neutrophils by influenza A virusMulti-Omics Studies towards Novel Modulators of Influenza A Virus-Host InteractionCompounds with anti-influenza activity: present and future of strategies for the optimal treatment and management of influenza. Part II: Future compounds against influenza virusInhibition of influenza virus replication by targeting broad host cell pathwaysThe proton translocation domain of cellular vacuolar ATPase provides a target for the treatment of influenza A virus infections.The host interactome of influenza virus presents new potential targets for antiviral drugs.Matriptase proteolytically activates influenza virus and promotes multicycle replication in the human airway epithelium.Inhibitory and combinatorial effect of diphyllin, a v-ATPase blocker, on influenza viruses.Role of protein kinase C betaII in influenza virus entry via late endosomes.Intracellular route of canine parvovirus entry.Initial events in infectious salmon anemia virus infection: evidence for the requirement of a low-pH step.Cellular uptake and infection by canine parvovirus involves rapid dynamin-regulated clathrin-mediated endocytosis, followed by slower intracellular traffickingInfectious entry by amphotropic as well as ecotropic murine leukemia viruses occurs through an endocytic pathwayReceptor-mediated entry by equine infectious anemia virus utilizes a pH-dependent endocytic pathway.Prolidase is required for early trafficking events during influenza A virus entryStructural differences observed in arboviruses of the alphavirus and flavivirus genera.Human cytomegalovirus entry into epithelial and endothelial cells depends on genes UL128 to UL150 and occurs by endocytosis and low-pH fusion.Influenza virus can enter and infect cells in the absence of clathrin-mediated endocytosis.Requirement for vacuolar H+ -ATPase activity and Ca2+ gradient during entry of rotavirus into MA104 cellsActivation of fusion by the SER virus F protein: a low-pH-dependent paramyxovirus entry process.Variations in genome-wide RNAi screens: lessons from influenza research.A host of factors regulating influenza virus replicationA Drosophila model for genetic analysis of influenza viral/host interactions.Effect of M1 protein and low pH on nuclear transport of influenza virus ribonucleoproteinsViral Inhibition of Bacterial Phagocytosis by Human Macrophages: Redundant Role of CD36.Rotaviruses induce an early membrane permeabilization of MA104 cells and do not require a low intracellular Ca2+ concentration to initiate their replication cycle.Influenza A virus-induced early activation of ERK and PI3K mediates V-ATPase-dependent intracellular pH change required for fusion.Inside job: ligand-receptor pharmacology beneath the plasma membrane.Inhibition of endosomal fusion activity of influenza virus by Rheum tanguticum (da-huang)Uncovering the global host cell requirements for influenza virus replication via RNAi screening.Cytosolic Gag p24 as an index of productive entry of human immunodeficiency virus type 1.Role of the cytoplasmic domain of the beta-subunit of integrin alpha(v)beta6 in infection by foot-and-mouth disease virusHemagglutinin 1-specific immunoglobulin G and Fab molecules mediate postattachment neutralization of influenza A virus by inhibition of an early fusion event.Transcriptional analysis of the innate immune response of ducks to different species-of-origin low pathogenic H7 avian influenza virusesActivation of p38 and ERK signaling during adenovirus vector cell entry lead to expression of the C-X-C chemokine IP-10.Subcellular localization and calcium and pH requirements for proteolytic processing of the Hendra virus fusion protein.The entry of reovirus into L cells is dependent on vacuolar proton-ATPase activity.
P2860
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P2860
Requirement for vacuolar proton-ATPase activity during entry of influenza virus into cells.
description
1995 nî lūn-bûn
@nan
1995年の論文
@ja
1995年論文
@yue
1995年論文
@zh-hant
1995年論文
@zh-hk
1995年論文
@zh-mo
1995年論文
@zh-tw
1995年论文
@wuu
1995年论文
@zh
1995年论文
@zh-cn
name
Requirement for vacuolar proto ...... of influenza virus into cells.
@en
Requirement for vacuolar proto ...... of influenza virus into cells.
@nl
type
label
Requirement for vacuolar proto ...... of influenza virus into cells.
@en
Requirement for vacuolar proto ...... of influenza virus into cells.
@nl
prefLabel
Requirement for vacuolar proto ...... of influenza virus into cells.
@en
Requirement for vacuolar proto ...... of influenza virus into cells.
@nl
P2860
P1433
P1476
Requirement for vacuolar proto ...... of influenza virus into cells.
@en
P2093
P2860
P304
P407
P577
1995-04-01T00:00:00Z