DDX3 DEAD-Box RNA helicase inhibits hepatitis B virus reverse transcription by incorporation into nucleocapsids.
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Hepatitis B virus polymerase blocks pattern recognition receptor signaling via interaction with DDX3: implications for immune evasionA motif unique to the human DEAD-box protein DDX3 is important for nucleic acid binding, ATP hydrolysis, RNA/DNA unwinding and HIV-1 replicationHepatitis B virus reverse transcriptase: diverse functions as classical and emerging targets for antiviral interventionRecent advances in the identification of the host factors involved in dengue virus replicationIncorporation of eukaryotic translation initiation factor eIF4E into viral nucleocapsids via interaction with hepatitis B virus polymerase.Hepatitis C virus core protein abrogates the DDX3 function that enhances IPS-1-mediated IFN-beta induction.Translational regulation of HIV-1 replication by HIV-1 Rev cellular cofactors Sam68, eIF5A, hRIP, and DDX3.Requirement of cellular DDX3 for hepatitis C virus replication is unrelated to its interaction with the viral core protein.Encapsidated hepatitis B virus reverse transcriptase is poised on an ordered RNA latticeRNA helicases: emerging roles in viral replication and the host innate response.A Co-Opted DEAD-Box RNA helicase enhances tombusvirus plus-strand synthesis.DDX3 DEAD-box RNA helicase is a host factor that restricts hepatitis B virus replication at the transcriptional levelThe synaptoneurosome transcriptome: a model for profiling the emolecular effects of alcoholCryptic protein priming sites in two different domains of duck hepatitis B virus reverse transcriptase for initiating DNA synthesis in vitro.The expanding functions of cellular helicases: the tombusvirus RNA replication enhancer co-opts the plant eIF4AIII-like AtRH2 and the DDX5-like AtRH5 DEAD-box RNA helicases to promote viral asymmetric RNA replication.HBV life cycle: entry and morphogenesis.Unveiling the roles of HBV polymerase for new antiviral strategiesThe Cellular Proteins Grb2 and DDX3 Are Increased upon Human Cytomegalovirus Infection and Act in a Proviral Fashion.Inhibitory effect of Phyllanthus urinaria L. extract on the replication of lamivudine-resistant hepatitis B virus in vitro.In vitro epsilon RNA-dependent protein priming activity of human hepatitis B virus polymerase.DDX3 as a strongest prognosis marker and its downregulation promotes metastasis in colorectal cancer.HCV Induces Telomerase Reverse Transcriptase, Increases Its Catalytic Activity, and Promotes Caspase Degradation in Infected Human HepatocytesHuman DEAD box helicase 3 couples IκB kinase ε to interferon regulatory factor 3 activation.DDB1 Stimulates Viral Transcription of Hepatitis B Virus via HBx-Independent Mechanisms.Viruses and the human DEAD-box helicase DDX3: inhibition or exploitation?Multiple functions of DDX3 RNA helicase in gene regulation, tumorigenesis, and viral infection.hCLE/C14orf166, a cellular protein required for viral replication, is incorporated into influenza virus particles.RNA helicase DDX3: at the crossroad of viral replication and antiviral immunity.Cellular RNA Helicase DDX1 Is Involved in Transmissible Gastroenteritis Virus nsp14-Induced Interferon-Beta ProductionAssembly and Release of Hepatitis B Virus.The ATP-Dependent RNA Helicase DDX3X Modulates Herpes Simplex Virus 1 Gene Expression.Molecular analysis of Hepatitis B virus sub-genotypes and incidence of preS1/preS2 region mutations in HBV-infected Egyptian patients from Mansoura.DEAD-Box Helicase DDX25 Is a Negative Regulator of Type I Interferon Pathway and Facilitates RNA Virus Infection.Proteome analysis of Duck Tembusu virus (DTMUV)-infected BHK-21 cells.RNA Exosome Complex Regulates Stability of the Hepatitis B Virus X-mRNA Transcript in a Non-stop-mediated (NSD) RNA Quality Control Mechanism.Hepatitis B virus X protein enhances Myc stability by inhibiting SCF(Skp2) ubiquitin E3 ligase-mediated Myc ubiquitination and contributes to oncogenesis.Determination of host RNA helicases activity in viral replication.The cellular RNA helicase DDX1 interacts with coronavirus nonstructural protein 14 and enhances viral replication.Hydrophobic residues of terminal protein domain of hepatitis B virus polymerase contribute to distinct steps in viral genome replication.DDX3 suppresses type I interferons and favors viral replication during Arenavirus infection
P2860
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P2860
DDX3 DEAD-Box RNA helicase inhibits hepatitis B virus reverse transcription by incorporation into nucleocapsids.
description
2009 nî lūn-bûn
@nan
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
2009年论文
@zh
2009年论文
@zh-cn
name
DDX3 DEAD-Box RNA helicase inh ...... orporation into nucleocapsids.
@en
DDX3 DEAD-Box RNA helicase inh ...... orporation into nucleocapsids.
@nl
type
label
DDX3 DEAD-Box RNA helicase inh ...... orporation into nucleocapsids.
@en
DDX3 DEAD-Box RNA helicase inh ...... orporation into nucleocapsids.
@nl
prefLabel
DDX3 DEAD-Box RNA helicase inh ...... orporation into nucleocapsids.
@en
DDX3 DEAD-Box RNA helicase inh ...... orporation into nucleocapsids.
@nl
P2860
P356
P1433
P1476
DDX3 DEAD-Box RNA helicase inh ...... corporation into nucleocapsids
@en
P2093
Haifeng Wang
Seahee Kim
P2860
P304
P356
10.1128/JVI.00011-09
P407
P577
2009-03-18T00:00:00Z