Intermediate-conductance Ca2+-activated K+ channels (IKCa1) regulate human prostate cancer cell proliferation through a close control of calcium entry.
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Calcium and vitamin D supplementation during androgen deprivation therapy for prostate cancer: a critical reviewThe Potassium Channel KCa3.1 Represents a Valid Pharmacological Target for Astrogliosis-Induced Neuronal Impairment in a Mouse Model of Alzheimer’s DiseaseEpigenetic dysregulation of KCa 3.1 channels induces poor prognosis in lung cancer.Tumor-associated macrophage-derived IL-6 and IL-8 enhance invasive activity of LoVo cells induced by PRL-3 in a KCNN4 channel-dependent mannerThe intermediate conductance Ca2+-activated K+ channel inhibitor TRAM-34 stimulates proliferation of breast cancer cells via activation of oestrogen receptors.The relation of serum parathyroid hormone and serum calcium to serum levels of prostate-specific antigen: a population-based study.Novel diagnostic biomarkers for prostate cancer.Overcoming key technological challenges in using mass spectrometry for mapping cell surfaces in tissues.Dendrotoxin-κ suppresses tumor growth induced by human lung adenocarcinoma A549 cells in nude mice.Altered expression of two-pore domain potassium (K2P) channels in cancerHigh expression of KCa3.1 in patients with clear cell renal carcinoma predicts high metastatic risk and poor survival.Calcium intake and prostate cancer among African Americans: effect modification by vitamin D receptor calcium absorption genotypeThe Blockage of KCa3.1 Channel Inhibited Proliferation, Migration and Promoted Apoptosis of Human Hepatocellular Carcinoma CellsActivation of K(+) channel by 1-EBIO rescues the head and neck squamous cell carcinoma cells from Ca(2+) ionophore-induced cell deathProtective effects of low calcium intake and low calcium absorption vitamin D receptor genotype in the California Collaborative Prostate Cancer StudyFunctional cooperation between KCa3.1 and TRPC1 channels in human breast cancer: Role in cell proliferation and patient prognosis.Identification and functional characterization of the intermediate-conductance Ca(2+)-activated K(+) channel (IK-1) in biliary epithelium.KCNN4 and S100A14 act as predictors of recurrence in optimally debulked patients with serous ovarian cancer.A three ion channel genes-based signature predicts prognosis of primary glioblastoma patients and reveals a chemotherapy sensitive subtype.Intermediate-Conductance-Ca2-Activated K Channel IKCa1 Is Upregulated and Promotes Cell Proliferation in Cervical CancerK(+) channels as therapeutic targets in oncology.Roles of ion transport in control of cell motility.Potassium channels in cell cycle and cell proliferation.Identification of prohibitin 1 as a potential prognostic biomarker in human pancreatic carcinoma using modified aqueous two-phase partition system combined with 2D-MALDI-TOF-TOF-MS/MS.An inhibitor of K+ channels modulates human endometrial tumor-initiating cells.KCa3.1 channels and glioblastoma: in vitro studies.IK channel activation increases tumor growth and induces differential behavioral responses in two breast epithelial cell lines.KCa3.1 (IK) modulates pancreatic cancer cell migration, invasion and proliferation: anomalous effects on TRAM-34.SK4 channels modulate Ca2+ signalling and cell cycle progression in murine breast cancer.Effects of Intermediate-Conductance Ca(2+)-Activated K(+) Channels on Human Endometrial Carcinoma Cells.Effects of ibandronate sodium, a nitrogen-containing bisphosphonate, on intermediate-conductance calcium-activated potassium channels in osteoclast precursor cells (RAW 264.7).KCNN4 channels participate in the EMT induced by PRL-3 in colorectal cancer.Inhibitory effects of blockage of intermediate conductance Ca(2+)-activated K (+) channels on proliferation of hepatocellular carcinoma cells.Human ether à-gogo K(+) channel 1 (hEag1) regulates MDA-MB-231 breast cancer cell migration through Orai1-dependent calcium entry.Dual-color quantum dot detection of a heterotetrameric potassium channel (hKCa3.1).Sotalol, but not digoxin is associated with decreased prostate cancer risk: A population-based case-control study.Functional Cooperation between KCa3.1 and TRPV4 Channels in Bronchial Smooth Muscle Cell Proliferation Associated with Chronic Asthma.K(+) channels and cell cycle progression in tumor cells.Bisphenol A stimulates human prostate cancer cell migration via remodelling of calcium signalling.Functional coupling between large-conductance potassium channels and Cav3.2 voltage-dependent calcium channels participates in prostate cancer cell growth.
P2860
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P2860
Intermediate-conductance Ca2+-activated K+ channels (IKCa1) regulate human prostate cancer cell proliferation through a close control of calcium entry.
description
2009 nî lūn-bûn
@nan
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
2009年论文
@zh
2009年论文
@zh-cn
name
Intermediate-conductance Ca2+- ...... lose control of calcium entry.
@en
Intermediate-conductance Ca2+-activated K+ channels
@nl
type
label
Intermediate-conductance Ca2+- ...... lose control of calcium entry.
@en
Intermediate-conductance Ca2+-activated K+ channels
@nl
prefLabel
Intermediate-conductance Ca2+- ...... lose control of calcium entry.
@en
Intermediate-conductance Ca2+-activated K+ channels
@nl
P2093
P2860
P356
P1433
P1476
Intermediate-conductance Ca2+- ...... lose control of calcium entry.
@en
P2093
A Bavencoffe
C Slomianny
E Dewailly
F Gackière
H Lallet-Daher
J L Bonnal
L Fleurisse
M Roudbaraki
P2860
P2888
P304
P356
10.1038/ONC.2009.25
P407
P577
2009-03-09T00:00:00Z
P5875
P6179
1004766344