about
Neisseria meningitidis differentially controls host cell motility through PilC1 and PilC2 components of type IV PiliRapid cytoskeletal response of epithelial cells to force generation by type IV piliInfluence of type IV pilus retraction on the architecture of the Neisseria gonorrhoeae-infected cell cortexHigh-force generation is a conserved property of type IV pilus systems.PilT2 enhances the speed of gonococcal type IV pilus retraction and of twitching motility.Speed switching of gonococcal surface motility correlates with proton motive force.Impact of calcium signaling during infection of Neisseria meningitidis to human brain microvascular endothelial cells.Activation of NF-κB by Neisseria gonorrhoeae is associated with microcolony formation and type IV pilus retraction.Nanoscale Pulling of Type IV Pili Reveals Their Flexibility and Adhesion to Surfaces over Extended Lengths of the Pili
P2860
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P2860
description
2009 nî lūn-bûn
@nan
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
2009年论文
@zh
2009年论文
@zh-cn
name
Dynamics of gonococcal type IV pili during infection.
@en
Dynamics of gonococcal type IV pili during infection.
@nl
type
label
Dynamics of gonococcal type IV pili during infection.
@en
Dynamics of gonococcal type IV pili during infection.
@nl
prefLabel
Dynamics of gonococcal type IV pili during infection.
@en
Dynamics of gonococcal type IV pili during infection.
@nl
P2093
P356
P1433
P1476
Dynamics of gonococcal type IV pili during infection.
@en
P2093
Berenike Maier
Dirk Opitz
Martin Clausen
P304
P356
10.1002/CPHC.200800654
P577
2009-07-01T00:00:00Z