Type, prevalence, and significance of core promoter/enhancer II mutations in hepatitis B viruses from immunosuppressed patients with severe liver disease.
about
Viral quasispecies evolutionMechanism of suppression of hepatitis B virus precore RNA transcription by a frequent double mutation.Properties of monoclonal antibodies directed against hepatitis B virus polymerase protein.Distinct hepatitis B virus dynamics in the immunotolerant and early immunoclearance phases.Naturally occurring mutations define a novel function of the hepatitis B virus core promoter in core protein expression.Novel type of hepatitis B virus mutation: replacement mutation involving a hepatocyte nuclear factor 1 binding site tandem repeat in chronic hepatitis B virus genotype E.Hepatitis B virus genotype and basal core promoter/precore mutations are associated with hepatitis B-related acute-on-chronic liver failure without pre-existing liver cirrhosis.Immune suppression uncovers endogenous cytopathic effects of the hepatitis B virusEnhancement of hepatitis B virus replication by its X protein in transgenic miceGenome replication, virion secretion, and e antigen expression of naturally occurring hepatitis B virus core promoter mutants.Non-A hepatitis B virus genotypes in antenatal clinics, United Kingdom.HLA-DP polymorphisms affect the outcomes of chronic hepatitis B virus infections, possibly through interacting with viral mutationsLUC7L3/CROP inhibits replication of hepatitis B virus via suppressing enhancer II/basal core promoter activity.Strong association between genotype F and hepatitis B virus (HBV) e antigen-negative variants among HBV-infected argentinean blood donors.Core promoter: a critical region where the hepatitis B virus makes decisions.Molecular epidemiology of an outbreak of fulminant hepatitis B.Amplification of full-length hepatitis B virus genomes from samples from patients with low levels of viremia: frequency and functional consequences of PCR-introduced mutations.The hepatitis B virus core promoter is strongly activated by the liver nuclear receptor fetoprotein transcription factor or by ectopically expressed steroidogenic factor 1.Functional analysis of ground squirrel hepatitis virus enhancer II.The HNF1/HNF4-dependent We2 element of woodchuck hepatitis virus controls viral replication and can activate the N-myc2 promoter.Genotype-dependent activation or repression of HBV enhancer II by transcription factor COUP-TF1.Molecular characterization of hepatitis B virus X gene in chronic hepatitis B patients.Association between Hepatitis B Virus X Gene Mutations and Clinical Status in Patients with Chronic Hepatitis B InfectionPatients with Coexistence of Circulating Hepatitis B Surface Antigen and Its Antibody May Have a Strong Predisposition to Virus Reactivation During Immunosuppressive Therapy: A Hypothesis.Pathogenesis of hepatitis B virus infection.Applications of next-generation sequencing analysis for the detection of hepatocellular carcinoma-associated hepatitis B virus mutations.
P2860
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P2860
Type, prevalence, and significance of core promoter/enhancer II mutations in hepatitis B viruses from immunosuppressed patients with severe liver disease.
description
1996 nî lūn-bûn
@nan
1996年の論文
@ja
1996年学术文章
@wuu
1996年学术文章
@zh-cn
1996年学术文章
@zh-hans
1996年学术文章
@zh-my
1996年学术文章
@zh-sg
1996年學術文章
@yue
1996年學術文章
@zh
1996年學術文章
@zh-hant
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Type, prevalence, and signific ...... nts with severe liver disease.
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Type, prevalence, and signific ...... nts with severe liver disease.
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Type, prevalence, and signific ...... nts with severe liver disease.
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Type, prevalence, and signific ...... nts with severe liver disease.
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Type, prevalence, and signific ...... nts with severe liver disease.
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Type, prevalence, and signific ...... nts with severe liver disease.
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P2093
P2860
P1433
P1476
Type, prevalence, and signific ...... ents with severe liver disease
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P2093
P2860
P304
P407
P577
1996-12-01T00:00:00Z