Amphotericin B-induced renal tubular cell injury is mediated by Na+ Influx through ion-permeable pores and subsequent activation of mitogen-activated protein kinases and elevation of intracellular Ca2+ concentration.

Amphotericin B-induced renal tubular cell injury is mediated by Na+ Influx through ion-permeable pores and subsequent activation of mitogen-activated protein kinases and elevation of intracellular Ca2+ concentration.

Item
http://www.wikidata.org/entity/Q39896114
Toxicokinetic and mechanistic basis for the safety and tolerability of liposomal amphotericin B.A phospholipid-apolipoprotein A-I nanoparticle containing amphotericin B as a drug delivery platform with cell membrane protective propertiesIt only takes one to do many jobs: Amphotericin B as antifungal and immunomodulatory drugAn optimized nanoparticle delivery system based on chitosan and chondroitin sulfate molecules reduces the toxicity of amphotericin B and is effective in treating tegumentary leishmaniasisIschemic kidney injury and mechanisms of tissue repair.Incidence, Predictors, and Impact on Hospital Mortality of Amphotericin B Nephrotoxicity Defined Using Newer Acute Kidney Injury Diagnostic CriteriaBrief Report: Effects of Tenofovir and Amphotericin B Deoxycholate Coadministration on Kidney Function in Patients Treated for Cryptococcal MeningitisPathophysiology of acute kidney injuryNew delivery systems for amphotericin B applied to the improvement of leishmaniasis treatment.Amphotericin B potentiates the anticancer activity of doxorubicin on the MCF-7 breast cancer cells.Role of protein kinase A signaling pathway in cyclosporine nephrotoxicity.Long open amphotericin channels revealed in cholesterol-containing phospholipid membranes are blocked by thiazole derivative.Protective and antifungal properties of Nanodisk-Amphotericin B over commercially available Amphotericin B.Alteration in cellular viability, pro-inflammatory cytokines and nitric oxide production in nephrotoxicity generation by Amphotericin B: involvement of PKA pathway signaling.
P2860

Amphotericin B-induced renal tubular cell injury is mediated by Na+ Influx through ion-permeable pores and subsequent activation of mitogen-activated protein kinases and elevation of intracellular Ca2+ concentration.

Item
http://www.wikidata.org/entity/Q39896114
description
2009 nî lūn-bûn
@nan
2009年の論文
@ja
2009年学术文章
@wuu
2009年学术文章
@zh-cn
2009年学术文章
@zh-hans
2009年学术文章
@zh-my
2009年学术文章
@zh-sg
2009年學術文章
@yue
2009年學術文章
@zh
2009年學術文章
@zh-hant
name
Amphotericin B-induced renal t ...... racellular Ca2+ concentration.
@en
Amphotericin B-induced renal t ...... racellular Ca2+ concentration.
@nl
type
label
Amphotericin B-induced renal t ...... racellular Ca2+ concentration.
@en
Amphotericin B-induced renal t ...... racellular Ca2+ concentration.
@nl
prefLabel
Amphotericin B-induced renal t ...... racellular Ca2+ concentration.
@en
Amphotericin B-induced renal t ...... racellular Ca2+ concentration.
@nl
P1433
P1476
P2093
P2860
P304
P31
P356
P407
P433
P478
P50
P577
P698
P932
P356
P1433
P1476
Amphotericin B-induced renal t ...... tracellular Ca2+ concentration
@en
P2093
P2860
P304
P31
P356
P407
P433
P478
P50
P577
P698
P932