Corticotropin (ACTH) acts directly on amygdala neurons to down-regulate corticotropin-releasing hormone gene expression.
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Attention deficit associated with early life interictal spikes in a rat model is improved with ACTHHippocampal dysfunction and cognitive impairments provoked by chronic early-life stress involve excessive activation of CRH receptorsHippocampal neuroplasticity induced by early-life stress: functional and molecular aspects.Neonatal seizures: an update on mechanisms and managementDevelopmental factors in the pathogenesis of neonatal seizuresWONOEP appraisal: Molecular and cellular biomarkers for epilepsy.Hippocampal neurogenesis is not enhanced by lifelong reduction of glucocorticoid levelsStressed-out, or in (utero)?The mystery of the Doctor's son, or the riddle of West syndrome.Neonatal melanocortin receptor agonist treatment reduces play fighting and promotes adult attachment in prairie voles in a sex-dependent mannerNeuropeptides as targets for the development of anticonvulsant drugs.Neurobiology of the stress response early in life: evolution of a concept and the role of corticotropin releasing hormoneHow do the many etiologies of West syndrome lead to excitability and seizures? The corticotropin releasing hormone excess hypothesis.What are the reasons for the strikingly different approaches to the use of ACTH in infants with West syndrome?ACTH treatment of infantile spasms: mechanisms of its effects in modulation of neuronal excitability.Region-specific onset of handling-induced changes in corticotropin-releasing factor and glucocorticoid receptor expressionImmune-mediated epilepsies.Hyper-excitability and epilepsy generated by chronic early-life stress.Basic science behind the catastrophic epilepsies.Leptin inhibits 4-aminopyridine- and pentylenetetrazole-induced seizures and AMPAR-mediated synaptic transmission in rodentsLong-term, progressive hippocampal cell loss and dysfunction induced by early-life administration of corticotropin-releasing hormone reproduce the effects of early-life stress.Infantile spasms: a critical review of emerging animal models.Differential contribution of CBP:CREB binding to corticotropin-releasing hormone expression in the infant and adult hypothalamusThe role of inflammation in epilepsyNovel animal models of pediatric epilepsy.Inflammation in Epileptic Encephalopathies.Treatment of Epileptic Encephalopathies: Current State of the Art.Go "West," young man...The quest for animal models of infantile spasms (West syndrome).Treatment of infantile spasms: the ideal and the mundane.Models for infantile spasms: an arduous journey to the Holy Grail..Seizures in the developing brain cause adverse long-term effects on spatial learning and anxiety.Insulin-Like Growth Factors in the Pathogenesis of Neurological Diseases in Children.Adrenocorticotropic hormone protects learning and memory function in epileptic Kcna1-null mice.A new animal model of infantile spasms with unprovoked persistent seizures.
P2860
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P2860
Corticotropin (ACTH) acts directly on amygdala neurons to down-regulate corticotropin-releasing hormone gene expression.
description
2001 nî lūn-bûn
@nan
2001年の論文
@ja
2001年学术文章
@wuu
2001年学术文章
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2001年学术文章
@zh-hans
2001年学术文章
@zh-my
2001年学术文章
@zh-sg
2001年學術文章
@yue
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@zh-hant
name
Corticotropin (ACTH) acts dire ...... asing hormone gene expression.
@en
Corticotropin
@nl
type
label
Corticotropin (ACTH) acts dire ...... asing hormone gene expression.
@en
Corticotropin
@nl
prefLabel
Corticotropin (ACTH) acts dire ...... asing hormone gene expression.
@en
Corticotropin
@nl
P2093
P2860
P356
P1433
P1476
Corticotropin (ACTH) acts dire ...... asing hormone gene expression.
@en
P2093
K L Brunson
M Eghbal-Ahmadi
P2860
P304
P356
10.1002/ANA.66.ABS
P577
2001-03-01T00:00:00Z