Redox mechanisms switch on hypoxia-dependent epithelial-mesenchymal transition in cancer cells.
about
Epithelial-mesenchymal transition in cancer: parallels between normal development and tumor progressionROS, Cell Senescence, and Novel Molecular Mechanisms in Aging and Age-Related DiseasesReactive Oxygen Species and Targeted Therapy for Pancreatic CancerTargeting CSCs in tumor microenvironment: the potential role of ROS-associated miRNAs in tumor aggressivenessAntioxidant Mechanisms and ROS-Related MicroRNAs in Cancer Stem CellsEmerging therapeutic biomarkers in endometrial cancerEndothelial to mesenchymal transition is common in atherosclerotic lesions and is associated with plaque instabilityNoncoding RNAs in Tumor Epithelial-to-Mesenchymal TransitionNuclear factor-κB-dependent epithelial to mesenchymal transition induced by HIF-1α activation in pancreatic cancer cells under hypoxic conditionsChemotherapy-induced monoamine oxidase expression in prostate carcinoma functions as a cytoprotective resistance enzyme and associates with clinical outcomesHypoxia triggers a Nur77-β-catenin feed-forward loop to promote the invasive growth of colon cancer cells.Structure-redox-relaxivity relationships for redox responsive manganese-based magnetic resonance imaging probes.Hyperoxic treatment induces mesenchymal-to-epithelial transition in a rat adenocarcinoma model.Tumorigenic WAP-T mouse mammary carcinoma cells: a model for a self-reproducing homeostatic cancer cell system.Glucose deprivation elicits phenotypic plasticity via ZEB1-mediated expression of NNMT.A three-dimensional in vitro model of tumor cell intravasation.Discovery of Mieap-regulated mitochondrial quality control as a new function of tumor suppressor p53The emerging role of QSOX1 in cancerPancreatic cancer stem cells and EMT in drug resistance and metastasis.Growth inhibition of pancreatic cancer cells by histone deacetylase inhibitor belinostat through suppression of multiple pathways including HIF, NFkB, and mTOR signaling in vitro and in vivoRapamycin enhances cetuximab cytotoxicity by inhibiting mTOR-mediated drug resistance in mesenchymal hepatoma cells.The hypoxic microenvironment upgrades stem-like properties of ovarian cancer cells.Hypoxia-inducible factors modulate the stemness and malignancy of colon cancer cells by playing opposite roles in canonical Wnt signaling.Tackling the cancer stem cells - what challenges do they pose?Transcriptional shift identifies a set of genes driving breast cancer chemoresistanceCurcumin limits the fibrogenic evolution of experimental steatohepatitis.Implication of snail in metabolic stress-induced necrosis.The role of Notch signaling pathway in epithelial-mesenchymal transition (EMT) during development and tumor aggressivenessA microfluidic device to study cancer metastasis under chronic and intermittent hypoxia.Novel regulatory program for norepinephrine-induced epithelial-mesenchymal transition in gastric adenocarcinoma cell lines.Aldose reductase inhibition prevents hypoxia-induced increase in hypoxia-inducible factor-1alpha (HIF-1alpha) and vascular endothelial growth factor (VEGF) by regulating 26 S proteasome-mediated protein degradation in human colon cancer cellsHypoxia and free radicals: role in tumor progression and the use of engineering-based platforms to address these relationships.Exploiting base excision repair to improve therapeutic approaches for pancreatic cancerThymosin β4 promotes hepatoblastoma metastasis via the induction of epithelial-mesenchymal transition.Hispidulin prevents hypoxia-induced epithelial-mesenchymal transition in human colon carcinoma cells.Hypoxia alters the recruitment of tropomyosins into the actin stress fibres of neuroblastoma cells.Copper depletion inhibits CoCl2-induced aggressive phenotype of MCF-7 cells via downregulation of HIF-1 and inhibition of Snail/Twist-mediated epithelial-mesenchymal transition.APE1/Ref-1 role in redox signaling: translational applications of targeting the redox function of the DNA repair/redox protein APE1/Ref-1.Strong Expression of Hypoxia-Inducible Factor-1α (HIF-1α) Is Associated with Axl Expression and Features of Aggressive Tumors in African Breast Cancer.Regulation of the protein stability of EMT transcription factors
P2860
Q24633875-5EE4A5FC-7BB2-4D7E-84A8-867432B69FC5Q26744325-B2DC537D-DE37-4901-A3D1-584C00E3AED1Q26765827-68FA8B27-89FE-433B-953F-EFFDC28C243BQ26822776-8BA07790-14DB-458E-8F1A-16ECD45D2AA2Q26853528-F46DC106-05E9-44C2-953E-2ED9444657BDQ26865904-F1A51F8D-6407-4C5D-96EC-7036D882580AQ27313607-9BB5E784-8088-40D1-A9AE-FC0769B98668Q28080346-B8A8E3FB-3CFA-4C8A-9F71-A7D118710A35Q28476694-07D81785-0514-4D9B-B84D-3CC5330D9420Q28542841-750A131F-FF05-4B8D-80A9-C31D8DDE4D00Q30408026-2FD41534-4A8B-41BE-A417-0B879434ECE9Q30851880-E5CC4C3E-4562-4402-868D-FAC453D074C6Q33487733-098143E5-75CF-4769-847B-82F42A4773F5Q33668830-CFD67162-B0E0-47B2-B1A8-3D6F2D0E7939Q33688593-15E7E509-D97F-4113-8310-99D371A9853BQ33716526-F9DD72AB-1E6A-4D31-A7F1-4C7350295CC4Q33738750-CCDA90A6-0842-4520-8D34-ED5FB5B94CD4Q33828308-0C7DB1E8-B2AA-4BF5-8A16-73E7E6CAA0DDQ33886120-5DD36D2F-9311-4BCD-9214-6BE07850D771Q33927370-B3186444-6C2B-43CB-BB64-2D11BFF039D5Q33990018-E655B129-D0CA-40AA-A535-67166F58324DQ34285178-07B3052A-F37E-4611-AB5D-DEE1826CDD2BQ34513657-633B1791-B666-4879-B37D-537199F73AF5Q34523263-30627780-7925-4FE0-B547-FA374418DAD2Q34552025-A35010ED-316F-4277-AB18-BC23465AD1D2Q34613675-7A49389A-2FFF-42C7-B959-3A6395F24B36Q34713056-17A63DD9-95F1-4AB0-B890-62E85A1AD2F3Q34885299-36CCD850-7AD9-4029-9BA3-9CA14EC8A110Q34912705-0E595F52-2E48-4BAE-A27D-0BC975C0427EQ35051882-860C83D6-C0DF-44D1-A1E6-FEFE76370C1CQ35085094-B44B5EDC-21CF-4C0B-B2D7-70818745C102Q35117865-248D4B16-182A-416E-A115-EDCF5ECFEF1AQ35596477-D7129DDE-3281-47DB-850C-D94A3687BCB0Q35628431-EBC7FBBC-14A3-471E-AA27-0892AF28C05FQ35666423-D88A9F3E-BA14-42DE-9ED5-6BA62AB6655EQ35811243-73296B94-069A-46B0-8BE0-DC4D8E9E42F3Q35852067-46709285-22BE-401C-B92A-4945354EF0B1Q35870045-1C12FB35-FB8D-46EA-9BC9-50E7DCBDFF8BQ35892646-7C2DB09F-6EBC-4AD2-B01B-693B37E6E2B7Q36124788-82973656-ABDC-48E9-99F7-2CF66C7E1E93
P2860
Redox mechanisms switch on hypoxia-dependent epithelial-mesenchymal transition in cancer cells.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
2008年论文
@zh
2008年论文
@zh-cn
name
Redox mechanisms switch on hyp ...... al transition in cancer cells.
@en
Redox mechanisms switch on hyp ...... al transition in cancer cells.
@nl
type
label
Redox mechanisms switch on hyp ...... al transition in cancer cells.
@en
Redox mechanisms switch on hyp ...... al transition in cancer cells.
@nl
prefLabel
Redox mechanisms switch on hyp ...... al transition in cancer cells.
@en
Redox mechanisms switch on hyp ...... al transition in cancer cells.
@nl
P2093
P2860
P356
P1433
P1476
Redox mechanisms switch on hyp ...... al transition in cancer cells.
@en
P2093
Alessandra Compagnone
Andrea Bandino
Carlo Cravanzola
Chiara Busletta
Claudia Paternostro
Davide Povero
Elena Zamara
Erica Novo
Francesca Bozzo
Lorenzo Valfrè di Bonzo
P2860
P304
P356
10.1093/CARCIN/BGN216
P407
P577
2008-09-12T00:00:00Z