NCAM-induced focal adhesion assembly: a functional switch upon loss of E-cadherin.
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Mechanisms underlying the anti-tumoral effects of Citrus Bergamia juiceMolecular Genetics of Intracranial Meningiomas with Emphasis on Canonical Wnt SignallingGlycosylation in Cancer: Interplay between Multidrug Resistance and Epithelial-to-Mesenchymal Transition?Homeostatic Signaling by Cell-Cell Junctions and Its Dysregulation during Cancer ProgressionContactin 1: A potential therapeutic target and biomarker in gastric cancerMolecular mechanisms of epithelial-mesenchymal transitionSchwann cells induce cancer cell dispersion and invasion.NCAM1 Polysialylation: The Prion Protein's Elusive Reason for Being?Initial steps of metastasis: cell invasion and endothelial transmigrationFyn: a novel molecular target in cancerThe Prion Protein Controls Polysialylation of Neural Cell Adhesion Molecule 1 during Cellular MorphogenesisN-cadherin regulates spatially polarized signals through distinct p120ctn and β-catenin-dependent signalling pathways.Mapping the first stages of mesoderm commitment during differentiation of human embryonic stem cells.Induction of epithelial mesenchimal transition and vasculogenesis in the lenses of Dbl oncogene transgenic mice.NCAM and FGFR1 coexpression and colocalization in renal tumorsThe binding of NCAM to FGFR1 induces a specific cellular response mediated by receptor trafficking.Mechanisms of the epithelial-mesenchymal transition by TGF-betaKruppel-like factor 4 inhibits epithelial-to-mesenchymal transition through regulation of E-cadherin gene expressionMolecular insights into reprogramming-initiation events mediated by the OSKM gene regulatory network.Regulation of EMT by KLF4 in gastrointestinal cancer.Polysialic acid is associated with better prognosis and IDH1-mutation in diffusely infiltrating astrocytomas.Klf4 is a transcriptional regulator of genes critical for EMT, including Jnk1 (Mapk8).The Transcriptional Repressor ZNF503/Zeppo2 Promotes Mammary Epithelial Cell Proliferation and Enhances Cell Invasion.Macrophage/epithelium cross-talk regulates cell cycle progression and migration in pancreatic progenitors.Cadherin cytoplasmic domains inhibit the cell surface localization of endogenous E-cadherin, blocking desmosome and tight junction formation and inducing cell dissociation.Promotion of cell migration by neural cell adhesion molecule (NCAM) is enhanced by PSA in a polysialyltransferase-specific mannerTranscription factor Dlx2 protects from TGFβ-induced cell-cycle arrest and apoptosis.Generation of tumor-initiating cells by exogenous delivery of OCT4 transcription factor.Variable Expression of Neural Cell Adhesion Molecule Isoforms in Renal Tissue: Possible Role in Incipient Renal Fibrosis.Epithelial-mesenchymal transition in oral squamous cell carcinomaUpregulation of miR-181s reverses mesenchymal transition by targeting KPNA4 in glioblastoma.The adhesion molecule NCAM promotes ovarian cancer progression via FGFR signallingSignificances of contactin-1 expression in human gastric cancer and knockdown of contactin-1 expression inhibits invasion and metastasis of MKN45 gastric cancer cells.Contactin 1 (CNTN1) expression associates with regional lymph node metastasis and is a novel predictor of prognosis in patients with oral squamous cell carcinomaAlisertib Induces Cell Cycle Arrest, Apoptosis, Autophagy and Suppresses EMT in HT29 and Caco-2 Cells.Characteristics of bladder transitional cell carcinoma with E-cadherin and N-cadherin double-negative expression.The pathophysiology of epithelial-mesenchymal transition induced by transforming growth factor-beta in normal and malignant mammary epithelial cells.Gene delivery of TGF-β1 induces arthrofibrosis and chondrometaplasia of synovium in vivo.Glycoproteomic analysis of two mouse mammary cell lines during transforming growth factor (TGF)-beta induced epithelial to mesenchymal transition.Disruption of E-cadherin by matrix metalloproteinase directly mediates epithelial-mesenchymal transition downstream of transforming growth factor-beta1 in renal tubular epithelial cells.
P2860
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P2860
NCAM-induced focal adhesion assembly: a functional switch upon loss of E-cadherin.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
2008年论文
@zh
2008年论文
@zh-cn
name
NCAM-induced focal adhesion assembly: a functional switch upon loss of E-cadherin.
@en
NCAM-induced focal adhesion assembly: a functional switch upon loss of E-cadherin.
@nl
type
label
NCAM-induced focal adhesion assembly: a functional switch upon loss of E-cadherin.
@en
NCAM-induced focal adhesion assembly: a functional switch upon loss of E-cadherin.
@nl
prefLabel
NCAM-induced focal adhesion assembly: a functional switch upon loss of E-cadherin.
@en
NCAM-induced focal adhesion assembly: a functional switch upon loss of E-cadherin.
@nl
P2093
P2860
P356
P1433
P1476
NCAM-induced focal adhesion assembly: a functional switch upon loss of E-cadherin.
@en
P2093
Angelika Kren
Anton Berns
Dominik Ziegler
Francois Lehembre
Gerhard Christofori
Jos Jonkers
Karin Strittmatter
Mahmut Yilmaz
Patrick W B Derksen
Phillip Went
P2860
P304
P356
10.1038/EMBOJ.2008.178
P407
P577
2008-09-04T00:00:00Z