Multiple pathways for Epstein-Barr virus episome loss from nasopharyngeal carcinoma. - Wikidata - awgldk - TriplyDB

Multiple pathways for Epstein-Barr virus episome loss from nasopharyngeal carcinoma.

Multiple pathways for Epstein-Barr virus episome loss from nasopharyngeal carcinoma.

http://www.wikidata.org/entity/Q39952661

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MicroRNA let-7 suppresses nasopharyngeal carcinoma cells proliferation through downregulating c-Myc expression Comprehensive high-throughput RNA sequencing analysis reveals contamination of multiple nasopharyngeal carcinoma cell lines with HeLa cell genomes.Vaccination against a hit-and-run viral cancer.Clinical, genomic, and metagenomic characterization of oral tongue squamous cell carcinoma in patients who do not smoke.Epithelial cell retention of transcriptionally active, P3HR-1-derived heterogeneous Epstein-Barr virus DNA with concurrent loss of parental virus.Comparison of humoral immune responses to Epstein-Barr virus and Kaposi's sarcoma-associated herpesvirus using a viral proteome microarray.The tumor virus landscape of AIDS-related lymphomas Molecular pathological study of the human nasopharyngeal carcinoma CNE3 cell line.Epstein-Barr virus nuclear antigen 1 replication and segregation functions in nasopharyngeal carcinoma cell lines Features distinguishing Epstein-Barr virus infections of epithelial cells and B cells: viral genome expression, genome maintenance, and genome amplification.A study of Epstein-Barr virus infection in the Chinese tree shrew(Tupaia belangeri chinensis).Latent Membrane Protein 1 Is a Novel Determinant of Epstein-Barr Virus Genome Persistence and Reactivation.Interferon-γ-inducible protein 16 (IFI16) is required for the maintenance of Epstein-Barr virus latency.Non-malignant epithelial cells preferentially proliferate from nasopharyngeal carcinoma biopsy cultured under conditionally reprogrammed conditions.Modulation of the Tumor Microenvironment by Epstein-Barr virus Latent Membrane Protein-1 in Nasopharyngeal Carcinoma.New Insights from Elucidating the Role of LMP1 in Nasopharyngeal Carcinoma.Interferon beta induces apoptosis in nasopharyngeal carcinoma cells via the TRAIL-signaling pathway.Air-Liquid Interface System To Understand Epstein-Barr Virus-Associated Nasopharyngeal Carcinoma Air-Liquid Interface Method To Study Epstein-Barr Virus Pathogenesis in Nasopharyngeal Epithelial Cells Establishment and characterization of new tumor xenografts and cancer cell lines from EBV-positive nasopharyngeal carcinoma Circular DNA tumor viruses make circular RNAs

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Multiple pathways for Epstein-Barr virus episome loss from nasopharyngeal carcinoma.

http://www.wikidata.org/entity/Q39952661

description

2008 nî lūn-bûn

@nan

2008年の論文

@ja

2008年学术文章

@wuu

2008年学术文章

@zh-cn

2008年学术文章

@zh-hans

2008年学术文章

@zh-my

2008年学术文章

@zh-sg

2008年學術文章

@yue

2008年學術文章

@zh

2008年學術文章

@zh-hant

name

Multiple pathways for Epstein-Barr virus episome loss from nasopharyngeal carcinoma.

@en

Multiple pathways for Epstein-Barr virus episome loss from nasopharyngeal carcinoma.

@nl

type

label

Multiple pathways for Epstein-Barr virus episome loss from nasopharyngeal carcinoma.

@en

Multiple pathways for Epstein-Barr virus episome loss from nasopharyngeal carcinoma.

@nl

prefLabel

Multiple pathways for Epstein-Barr virus episome loss from nasopharyngeal carcinoma.

@en

Multiple pathways for Epstein-Barr virus episome loss from nasopharyngeal carcinoma.

@nl

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International Journal of Cancer

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Multiple pathways for Epstein-Barr virus episome loss from nasopharyngeal carcinoma.

@en

P2093

Chelsey J Hilscher

http://www.w3.org/2001/XMLSchema#string

Dirk P Dittmer

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Eric V Yang

http://www.w3.org/2001/XMLSchema#string

Margaret L Gulley

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Min Chen

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Ronald Glaser

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P2860

Faster quantitative real-time PCR protocols may lose sensitivity and show increased variability.

Relation of Burkitt's tumor-associated herpes-ytpe virus to infectious mononucleosis

Cluster analysis and display of genome-wide expression patterns

EMBOSS: the European Molecular Biology Open Software Suite

Epstein-Barr virus nuclear antigen 2 (EBNA2) gene deletion is consistently linked with EBNA3A, -3B, and -3C expression in Burkitt's lymphoma cells and with increased resistance to apoptosis.

Expression of the Epstein-Barr virus genome in a nasopharyngeal carcinoma epithelial tumor cell line.

VIRUS PARTICLES IN CULTURED LYMPHOBLASTS FROM BURKITT'S LYMPHOMA.

Two epithelial tumor cell lines (HNE-1 and HONE-1) latently infected with Epstein-Barr virus that were derived from nasopharyngeal carcinomas.

Stable replication of plasmids derived from Epstein-Barr virus in various mammalian cells.

Three restricted forms of Epstein-Barr virus latency counteracting apoptosis in c-myc-expressing Burkitt lymphoma cells.

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2105-2112

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scholarly article

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10.1002/IJC.23685

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9

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123

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2008-11-01T00:00:00Z

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23157612

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18688856

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Epstein–Barr virus

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2866115

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