Gene expression studies reveal that DNA damage, vascular perturbation, and inflammation contribute to the pathogenesis of carbonyl sulfide neurotoxicity.
about
Mass spectrometric analysis of rat cerebrospinal fluid proteins following exposure to the neurotoxicant carbonyl sulfide.Ablation of CCAAT/Enhancer-Binding Protein Delta (C/EBPD): Increased Plaque Burden in a Murine Alzheimer's Disease ModelRecommended neuroanatomical sampling practices for comprehensive brain evaluation in nonclinical safety studies.
P2860
Gene expression studies reveal that DNA damage, vascular perturbation, and inflammation contribute to the pathogenesis of carbonyl sulfide neurotoxicity.
description
2009 nî lūn-bûn
@nan
2009年の論文
@ja
2009年学术文章
@wuu
2009年学术文章
@zh-cn
2009年学术文章
@zh-hans
2009年学术文章
@zh-my
2009年学术文章
@zh-sg
2009年學術文章
@yue
2009年學術文章
@zh
2009年學術文章
@zh-hant
name
Gene expression studies reveal ...... arbonyl sulfide neurotoxicity.
@en
Gene expression studies reveal ...... arbonyl sulfide neurotoxicity.
@nl
type
label
Gene expression studies reveal ...... arbonyl sulfide neurotoxicity.
@en
Gene expression studies reveal ...... arbonyl sulfide neurotoxicity.
@nl
prefLabel
Gene expression studies reveal ...... arbonyl sulfide neurotoxicity.
@en
Gene expression studies reveal ...... arbonyl sulfide neurotoxicity.
@nl
P2093
P2860
P921
P356
P1476
Gene expression studies reveal ...... arbonyl sulfide neurotoxicity.
@en
P2093
Daniel L Morgan
James P Morrison
Jennifer B Collins
Peter B Little
Robert C Sills
Robert C Switzer
Thai-Vu Ton
P2860
P304
P356
10.1177/0192623309335631
P407
P577
2009-04-24T00:00:00Z