Soluble N-cadherin overexpression reduces features of atherosclerotic plaque instability.
about
Genetic inactivation of IL-1 signaling enhances atherosclerotic plaque instability and reduces outward vessel remodeling in advanced atherosclerosis in miceMMP-7 mediates cleavage of N-cadherin and promotes smooth muscle cell apoptosis.A selective matrix metalloproteinase-12 inhibitor retards atherosclerotic plaque development in apolipoprotein E-knockout mice.Cell-cell interactions mediate the response of vascular smooth muscle cells to substrate stiffness.EC4, a truncation of soluble N-cadherin, reduces vascular smooth muscle cell apoptosis and markers of atherosclerotic plaque instability.Differential effects of tissue inhibitor of metalloproteinase (TIMP)-1 and TIMP-2 on atherosclerosis and monocyte/macrophage invasionC-reactive protein/oxidised low-density lipoprotein/β2-glycoprotein I complex promotes atherosclerosis in diabetic BALB/c mice via p38mitogen-activated protein kinase signal pathwayCadherin-6B is proteolytically processed during epithelial-to-mesenchymal transitions of the cranial neural crest.The degradation of chondrogenic pellets using cocultures of synovial fibroblasts and U937 cells.Life after proteolysis: Exploring the signaling capabilities of classical cadherin cleavage fragments.Cyclin-dependent kinase inhibitor 2B regulates efferocytosis and atherosclerosisInflammation in atherosclerosis: a cause or a result of vascular disorders?Emerging regulators of vascular smooth muscle cell function in the development and progression of atherosclerosis.Soluble N-cadherin: A novel inhibitor of VSMC proliferation and intimal thickening.Should I stay or should I go? Cadherin function and regulation in the neural crest.Cadherins function during the collective cell migration of Xenopus Cranial Neural Crest cells: revisiting the role of E-cadherin.TGFβ1 suppresses vascular smooth muscle cell motility by expression of N-cadherin.Targeting Wnt/β-Catenin Activated Cells with Dominant-Negative N-cadherin to Reduce Neointima Formation.Inhibition of N-cadherin retards smooth muscle cell migration and intimal thickening via induction of apoptosis.Aneurysm Severity is Increased by Combined Mmp-7 Deletion and N-cadherin Mimetic (EC4-Fc) Over-Expression.Role of smooth muscle cells in coronary artery bypass grafting failure.Oxysterol-induced apoptosis of smooth muscle cells is under the control of a soluble adenylyl cyclase.Impact of lysophosphatidylcholine on survival and function of UEA-1(+)acLDL (+) endothelial progenitor cells in patients with coronary artery disease.Suppression of soluble adenylyl cyclase protects smooth muscle cells against oxidative stress-induced apoptosis.
P2860
Q30429015-F10E0CB6-FB9A-4490-B4B4-381A1D23B367Q33906860-FB3DBB93-7221-4DAD-92D0-C85E9A0D08D6Q34585560-CEBDC585-121E-4778-BEE4-792E3441D118Q35132810-8771436F-37A9-4A14-AE86-1516588592E3Q35184213-785ACE68-F185-4F81-B5F9-221D2124E170Q36496342-DE27AEFF-82F7-47CD-A09E-256191C9029BQ36814928-B8E23128-BC0A-4263-885D-3EE6B31A9370Q37416517-BB7E9DD0-50F2-4473-BDE9-63284F78D2F7Q37424417-9429421C-8594-4C5B-A8C4-A4DB5E45212EQ37513627-4BCA5038-8C15-4E13-843E-94DAC6F8BFFCQ37609624-FFA68423-9748-4726-B3B7-F504C1984892Q37986175-75D543F2-7449-4B1A-A407-71E89C5D559BQ38232998-A802A7ED-1F69-42D4-8E8F-85C265374E61Q38358592-FF5D97C1-9E9A-4CFF-82F4-039F66B0E79FQ38933085-F4DA6C25-2A9F-45A9-92CA-8EBF851656B8Q39280745-73548B50-6F43-4EFD-A295-7081BD948C04Q39581315-4BB8A6C3-E941-4B1F-9628-4C16F6370705Q40187241-C8973F67-E5C3-4F39-A370-CDC50EDF0528Q42177448-E4945697-9001-47CA-A991-DB8D16D90ACEQ47151616-61141D45-C2DC-49B8-8185-11046FA622A0Q47709776-CE778618-7FA8-4E78-86F0-C28E5AE5EE1AQ50942948-D13DB998-3814-454D-86A1-CBBCC89F105BQ51753960-947AA780-5AD3-40AE-95DE-340EDF8E1334Q54356199-B868FA7A-29AB-4B62-B742-F659553AAFF2
P2860
Soluble N-cadherin overexpression reduces features of atherosclerotic plaque instability.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年学术文章
@wuu
2008年学术文章
@zh-cn
2008年学术文章
@zh-hans
2008年学术文章
@zh-my
2008年学术文章
@zh-sg
2008年學術文章
@yue
2008年學術文章
@zh
2008年學術文章
@zh-hant
name
Soluble N-cadherin overexpression reduces features of atherosclerotic plaque instability.
@en
Soluble N-cadherin overexpression reduces features of atherosclerotic plaque instability.
@nl
type
label
Soluble N-cadherin overexpression reduces features of atherosclerotic plaque instability.
@en
Soluble N-cadherin overexpression reduces features of atherosclerotic plaque instability.
@nl
prefLabel
Soluble N-cadherin overexpression reduces features of atherosclerotic plaque instability.
@en
Soluble N-cadherin overexpression reduces features of atherosclerotic plaque instability.
@nl
P2093
P2860
P1476
Soluble N-cadherin overexpression reduces features of atherosclerotic plaque instability.
@en
P2093
Cressida A Lyon
Graciela B Sala-Newby
Sarah J George
P2860
P304
P356
10.1161/ATVBAHA.108.178087
P407
P577
2008-11-13T00:00:00Z