Early growth response-1 protein is induced by JC virus infection and binds and regulates the JC virus promoter. - Wikidata - awgldk - TriplyDB

Early growth response-1 protein is induced by JC virus infection and binds and regulates the JC virus promoter.

Early growth response-1 protein is induced by JC virus infection and binds and regulates the JC virus promoter.

http://www.wikidata.org/entity/Q40000670

about

Molecular biology, epidemiology, and pathogenesis of progressive multifocal leukoencephalopathy, the JC virus-induced demyelinating disease of the human brain JC virus in the pathogenesis of colorectal cancer, an etiological agent or another component in a multistep process?Animal Models for Progressive Multifocal Leukoencephalopathy Differentiation of human fetal multipotential neural progenitor cells to astrocytes reveals susceptibility factors for JC virus.Hypoxia inducible factor-1 alpha activation of the JCV promoter: role in the pathogenesis of progressive multifocal leukoencephalopathy Regulation of human neurotropic JC virus replication by alternative splicing factor SF2/ASF in glial cells.Molecular regulation of JC virus tropism: insights into potential therapeutic targets for progressive multifocal leukoencephalopathy.Resveratrol inhibits KSHV reactivation by lowering the levels of cellular EGR-1 Cell membrane-bound Kaposi's sarcoma-associated herpesvirus-encoded glycoprotein B promotes virus latency by regulating expression of cellular Egr-1 Persistence and pathogenesis of the neurotropic polyomavirus JC Activation of Egr-1 expression in astrocytes by HIV-1 Tat: new insights into astrocyte-mediated Tat neurotoxicity.Lytic HSV-1 infection induces the multifunctional transcription factor Early Growth Response-1 (EGR-1) in rabbit corneal cells Regulation of gene expression in primate polyomaviruses.Pathogenesis of progressive multifocal leukoencephalopathy--revisited Epigenetic regulation of polyomavirus JC.Resveratrol, sirtuins, and viruses.Role for tumor necrosis factor-α in JC virus reactivation and progressive multifocal leukoencephalopathy.Modulation of JC virus transcription by C/EBPbeta Immune suppression of JC virus gene expression is mediated by SRSF1.Evidence for modulation of BAG3 by polyomavirus JC early protein.Egr-1 regulates RTA transcription through a cooperative involvement of transcriptional regulators.The Host Factor Early Growth Response Gene (EGR-1) Regulates Vaccinia virus Infectivity during Infection of Starved Mouse Cells.

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P2860

Early growth response-1 protein is induced by JC virus infection and binds and regulates the JC virus promoter.

http://www.wikidata.org/entity/Q40000670

description

2008 nî lūn-bûn

@nan

2008年の論文

@ja

2008年学术文章

@wuu

2008年学术文章

@zh-cn

2008年学术文章

@zh-hans

2008年学术文章

@zh-my

2008年学术文章

@zh-sg

2008年學術文章

@yue

2008年學術文章

@zh

2008年學術文章

@zh-hant

name

Early growth response-1 protei ...... gulates the JC virus promoter.

@en

type

label

Early growth response-1 protei ...... gulates the JC virus promoter.

@en

prefLabel

Early growth response-1 protei ...... gulates the JC virus promoter.

@en

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J.VIROL.2008.02.021

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Early growth response-1 protei ...... egulates the JC virus promoter

@en

P2093

Bassel E Sawaya

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Ilker K Sariyer

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Jacqueline Tung

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Kamel Khalili

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Luca Romagnoli

http://www.w3.org/2001/XMLSchema#string

Mahmut Safak

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Mariha Feliciano

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Martyn K White

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P2860

Activation of the JC virus Tat-responsive transcriptional control element by association of the Tat protein of human immunodeficiency virus 1 with cellular protein Pur alpha

A new technique for the assay of infectivity of human adenovirus 5 DNA

Differential effects of rabies and borna disease viruses on immediate-early- and late-response gene expression in brain tissues

MatInspector and beyond: promoter analysis based on transcription factor binding sites

Progressive multifocal leukoencephalopathy: lessons from AIDS and natalizumab.

p73 Interacts with human immunodeficiency virus type 1 Tat in astrocytic cells and prevents its acetylation on lysine 28.

Polyoma small and middle T antigens and SV40 small t antigen form stable complexes with protein phosphatase 2A.

Common host genes are activated in mouse brain by Japanese encephalitis and rabies viruses.

The interaction of SV40 small tumor antigen with protein phosphatase 2A stimulates the map kinase pathway and induces cell proliferation.

Cellular gene expression upon human immunodeficiency virus type 1 infection of CD4(+)-T-cell lines

P304

331-341

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scholarly article

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10.1016/J.VIROL.2008.02.021

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2

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375

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Pasquale Ferrante

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2008-03-18T00:00:00Z

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5499838

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18353421

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2632587

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