Emodin enhances cytotoxicity of chemotherapeutic drugs in prostate cancer cells: the mechanisms involve ROS-mediated suppression of multidrug resistance and hypoxia inducible factor-1.
about
SENP3 is responsible for HIF-1 transactivation under mild oxidative stress via p300 de-SUMOylationSouth Asian Medicinal Compounds as Modulators of Resistance to Chemotherapy and RadiotherapyHigh Mobility Group B Proteins, Their Partners, and Other Redox Sensors in Ovarian and Prostate CancerA network pharmacology approach to understanding the mechanisms of action of traditional medicine: Bushenhuoxue formula for treatment of chronic kidney diseaseThe biphasic redox sensing of SENP3 accounts for the HIF-1 transcriptional activity shift by oxidative stressA novel Ca2+/calmodulin antagonist HBC inhibits angiogenesis and down-regulates hypoxia-inducible factor.SENP3-mediated de-conjugation of SUMO2/3 from promyelocytic leukemia is correlated with accelerated cell proliferation under mild oxidative stressSpecies and gender differences affect the metabolism of emodin via glucuronidationProteomic analysis identified DJ-1 as a cisplatin resistant marker in non-small cell lung cancer.Emodin as an effective agent in targeting cancer stem-like side population cells of gallbladder carcinoma.Emodin augments cisplatin cytotoxicity in platinum-resistant ovarian cancer cells via ROS-dependent MRP1 downregulation.Regulation of hypoxia inducible factor-1α expression by the alteration of redox status in HepG2 cells.Anti-cancer natural products isolated from chinese medicinal herbsRadiation, inflammation, and immune responses in cancerEmodin enhances cisplatin-induced cytotoxicity in human bladder cancer cells through ROS elevation and MRP1 downregulation.Cellular Redox Status Regulates Emodin-Induced Radiosensitization of Nasopharyngeal Carcinoma Cells In Vitro and In Vivo.Emodin regulating excision repair cross-complementation group 1 through fibroblast growth factor receptor 2 signaling.Compounds from Chinese herbal medicines as reversal agents for P-glycoprotein-mediated multidrug resistance in tumours.MicroRNA-218 Increases the Sensitivity of Bladder Cancer to Cisplatin by Targeting Glut1.The exposure of cancer cells to hyperthermia, iron oxide nanoparticles, and mitomycin C influences membrane multidrug resistance protein expression levels.Exploration of effects of emodin in selected cancer cell lines: enhanced growth inhibition by ascorbic acid and regulation of LRP1 and AR under hypoxia-like conditions.Emodin regulates apoptotic pathway in human liver cancer cells.In vitro anti-fibrotic activities of herbal compounds and herbs.Emodin modulates epigenetic modifications and suppresses bladder carcinoma cell growth.Emodin Exerts an Antiapoptotic Effect on Human Chronic Myelocytic Leukemia K562 Cell Lines by Targeting the PTEN/PI3K-AKT Signaling Pathway and Deleting BCR-ABL.Redox Paradox: A novel approach to therapeutics-resistant cancer.
P2860
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P2860
Emodin enhances cytotoxicity of chemotherapeutic drugs in prostate cancer cells: the mechanisms involve ROS-mediated suppression of multidrug resistance and hypoxia inducible factor-1.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
Emodin enhances cytotoxicity o ...... nd hypoxia inducible factor-1.
@en
type
label
Emodin enhances cytotoxicity o ...... nd hypoxia inducible factor-1.
@en
prefLabel
Emodin enhances cytotoxicity o ...... nd hypoxia inducible factor-1.
@en
P2093
P921
P356
P1476
Emodin enhances cytotoxicity o ...... nd hypoxia inducible factor-1.
@en
P2093
Chao Huang
Gui-ying Shi
Qing-shen Hu
Xin-zhi Huang
Yu-ying Chen
P304
P356
10.4161/CBT.7.3.5457
P577
2007-12-21T00:00:00Z