Endothelial cells and normal breast epithelial cells enhance invasion of breast carcinoma cells by CXCR-4-dependent up-regulation of urokinase-type plasminogen activator receptor (uPAR, CD87) expression.
about
Benign mammary epithelial cells enhance the transformed phenotype of human breast cancer cells.Vitamin D binding protein-macrophage activating factor directly inhibits proliferation, migration, and uPAR expression of prostate cancer cells.Melanoma cell therapy: Endothelial progenitor cells as shuttle of the MMP12 uPAR-degrading enzymeRapid mobilization of hematopoietic progenitors by AMD3100 and catecholamines is mediated by CXCR4-dependent SDF-1 release from bone marrow stromal cells.Density enhanced phosphatase-1 down-regulates urokinase receptor surface expression in confluent endothelial cellsAltered features and increased chemosensitivity of human breast cancer cells mediated by adipose tissue-derived mesenchymal stromal cells.Tumor heterogeneity: mechanisms and bases for a reliable application of molecular marker designu-PAR expression in cancer associated fibroblast: new acquisitions in multiple myeloma progressionVascular endothelial cells facilitated HCC invasion and metastasis through the Akt and NF-κB pathways induced by paracrine cytokines.Bone metastases: molecular mechanisms and novel therapeutic interventions.Differential roles for membrane-bound and soluble syndecan-1 (CD138) in breast cancer progression.Urokinase receptor and CXCR4 are regulated by common microRNAs in leukaemia cells.Breast tumor stroma: A driving force in the development of resistance to therapies.Phenotype of Gc-globulin influences the macrophage activating factor (MAF) levels in serum.Systemic sclerosis-endothelial cell antiangiogenic pentraxin 3 and matrix metalloprotease 12 control human breast cancer tumor vascularization and development in miceTranscriptional regulation of CXCR4 in prostate cancer: significance of TMPRSS2-ERG fusionsMorphine and breast tumor metastasis: the role of matrix-degrading enzymes.Reduction of in vitro invasion and in vivo cartilage degradation in a SCID mouse model by loss of function of the fibrinolytic system of rheumatoid arthritis synovial fibroblasts.
P2860
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P2860
Endothelial cells and normal breast epithelial cells enhance invasion of breast carcinoma cells by CXCR-4-dependent up-regulation of urokinase-type plasminogen activator receptor (uPAR, CD87) expression.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
2008年论文
@zh
2008年论文
@zh-cn
name
Endothelial cells and normal b ...... eptor (uPAR, CD87) expression.
@en
type
label
Endothelial cells and normal b ...... eptor (uPAR, CD87) expression.
@en
prefLabel
Endothelial cells and normal b ...... eptor (uPAR, CD87) expression.
@en
P2093
P2860
P50
P356
P1476
Endothelial cells and normal b ...... ceptor (uPAR, CD87) expression
@en
P2093
P2860
P304
P356
10.1002/PATH.2309
P577
2008-04-01T00:00:00Z