Dioxin-mediated up-regulation of aryl hydrocarbon receptor target genes is dependent on the calcium/calmodulin/CaMKIalpha pathway.
about
Association of dioxin and other persistent organic pollutants (POPs) with diabetes: epidemiological evidence and new mechanisms of beta cell dysfunctionDynamic zebrafish interactome reveals transcriptional mechanisms of dioxin toxicityGenome-wide RNAi high-throughput screen identifies proteins necessary for the AHR-dependent induction of CYP1A1 by 2,3,7,8-tetrachlorodibenzo-p-dioxin.RNAi-based screening identifies kinases interfering with dioxin-mediated up-regulation of CYP1A1 activityThe Aryl Hydrocarbon Receptor: A Target for Breast Cancer Therapy.Benzimidazoisoquinolines: a new class of rapidly metabolized aryl hydrocarbon receptor (AhR) ligands that induce AhR-dependent Tregs and prevent murine graft-versus-host disease.Induction of intracellular calcium concentration by environmental benzo(a)pyrene involves a β2-adrenergic receptor/adenylyl cyclase/Epac-1/inositol 1,4,5-trisphosphate pathway in endothelial cells.AHR-mediated immunomodulation: the role of altered gene transcription.Comparative analysis of AhR-mediated TCDD-elicited gene expression in human liver adult stem cells.Environmental Factors Impacting Bone-Relevant Chemokines.Cytokines as molecular targets for aryl hydrocarbon receptor ligands: implications for toxicity and xenobiotic detoxification.Identification of breast cancer mechanism based on weighted gene coexpression network analysis.Activation of aryl hydrocarbon receptor promotes invasion of clear cell renal cell carcinoma and is associated with poor prognosis and cigarette smoke.Aryl hydrocarbon receptor (AhR) agonists increase airway epithelial matrix metalloproteinase activity.Aryl hydrocarbon receptor-mediated disruption of contact inhibition is associated with connexin43 downregulation and inhibition of gap junctional intercellular communication.Aryl hydrocarbon receptor-independent up-regulation of intracellular calcium concentration by environmental polycyclic aromatic hydrocarbons in human endothelial HMEC-1 cells.2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) induces calcium influx through T-type calcium channel and enhances lysosomal exocytosis and insulin secretion in INS-1 cells.Benzo(a)pyrene triggers desensitization of β2-adrenergic pathway.AhR activation increases IL-2 production by alloreactive CD4+ T cells initiating the differentiation of mucosal-homing Tim3+ Lag3+ Tr1 cells.Lipophilic Chemicals from Diesel Exhaust Particles Trigger Calcium Response in Human Endothelial Cells via Aryl Hydrocarbon Receptor Non-Genomic Signalling.
P2860
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P2860
Dioxin-mediated up-regulation of aryl hydrocarbon receptor target genes is dependent on the calcium/calmodulin/CaMKIalpha pathway.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
Dioxin-mediated up-regulation ...... calmodulin/CaMKIalpha pathway.
@en
type
label
Dioxin-mediated up-regulation ...... calmodulin/CaMKIalpha pathway.
@en
prefLabel
Dioxin-mediated up-regulation ...... calmodulin/CaMKIalpha pathway.
@en
P2093
P356
P1476
Dioxin-mediated up-regulation ...... /calmodulin/CaMKIalpha pathway
@en
P2093
Claudine Rauch
David Gilot
Olivier Fardel
Patricia Monteiro
P304
P356
10.1124/MOL.107.043125
P577
2007-12-18T00:00:00Z