Chronic ethanol exposure leads to divergent control of dopaminergic synapses in distinct target regions.
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Alcohol dependence and free-choice drinking in miceThe role of biogenic amine signaling in the bed nucleus of the stria terminals in alcohol abuse.Modulation of glutamatergic synaptic transmission in the bed nucleus of the stria terminalis.Dopamine enhances fast excitatory synaptic transmission in the extended amygdala by a CRF-R1-dependent process.Distinct forms of Gq-receptor-dependent plasticity of excitatory transmission in the BNST are differentially affected by stress.Altered anxiety-like behavior and long-term potentiation in the bed nucleus of the stria terminalis in adult mice exposed to chronic social isolation, unpredictable stress, and ethanol beginning in adolescence.Ethanol drinking reduces extracellular dopamine levels in the posterior ventral tegmental area of nondependent alcohol-preferring rats.Chronic intermittent ethanol exposure reduces presynaptic dopamine neurotransmission in the mouse nucleus accumbens.GluN2B subunit deletion reveals key role in acute and chronic ethanol sensitivity of glutamate synapses in bed nucleus of the stria terminalis.Chronic social isolation during adolescence augments catecholamine response to acute ethanol in the basolateral amygdalaTargeting dynorphin/kappa opioid receptor systems to treat alcohol abuse and dependence.Alcohol-induced plasticity in the dynorphin/kappa-opioid receptor system.A corticotropin releasing factor pathway for ethanol regulation of the ventral tegmental area in the bed nucleus of the stria terminalis.Conceptualizing withdrawal-induced escalation of alcohol self-administration as a learned, plasticity-dependent process.Chronic Intermittent Ethanol Exposure Alters Stress Effects on (3α,5α)-3-hydroxy-pregnan-20-one (3α,5α-THP) Immunolabeling of Amygdala Neurons in C57BL/6J Mice.Effect of voluntary alcohol consumption on Maoa expression in the mesocorticolimbic brain of adult male rats previously exposed to prolonged maternal separationExcessive alcohol consumption is blocked by glial cell line-derived neurotrophic factor.Alcohol exposure alters NMDAR function in the bed nucleus of the stria terminalis.Alcoholism and cellular vulnerability in different brain regions.Chronic ethanol exposure increases inhibition of optically targeted phasic dopamine release in the nucleus accumbens core and medial shell ex vivo.Dynorphin and κ-Opioid Receptor Dysregulation in the Dopaminergic Reward System of Human Alcoholics.Blockade of GABA(A) receptors in the paraventricular nucleus of the hypothalamus attenuates voluntary ethanol intake and activates the hypothalamic-pituitary-adrenocortical axis.Stress abolishes the effect of previous chronic ethanol consumption on drug place preference and on the mesocorticolimbic brain pathway.
P2860
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P2860
Chronic ethanol exposure leads to divergent control of dopaminergic synapses in distinct target regions.
description
2008 nî lūn-bûn
@nan
2008年の論文
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2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
2008年论文
@zh
2008年论文
@zh-cn
name
Chronic ethanol exposure leads ...... es in distinct target regions.
@en
type
label
Chronic ethanol exposure leads ...... es in distinct target regions.
@en
prefLabel
Chronic ethanol exposure leads ...... es in distinct target regions.
@en
P2093
P2860
P1433
P1476
Chronic ethanol exposure leads ...... es in distinct target regions.
@en
P2093
Danny G Winder
Julie C Healey
Thomas L Kash
P2860
P304
P356
10.1016/J.ALCOHOL.2008.01.003
P577
2008-03-20T00:00:00Z