Extralymphatic virus sanctuaries as a consequence of potent T-cell activation.
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Impaired antibody response causes persistence of prototypic T cell-contained virusCommon threads in persistent viral infectionsT-cell STAT3 is required for the maintenance of humoral immunity to LCMV.The initial draining lymph node primes the bulk of the CD8 T cell response and influences memory T cell trafficking after a systemic viral infectionIncreased CD8+ T cell response to Epstein-Barr virus lytic antigens in the active phase of multiple sclerosisHypomorphic mutation in the site-1 protease Mbtps1 endows resistance to persistent viral infection in a cell-specific manner.Viral persistence redirects CD4 T cell differentiation toward T follicular helper cellsUsp18 driven enforced viral replication in dendritic cells contributes to break of immunological tolerance in autoimmune diabetes.Targeting of non-dominant antigens as a vaccine strategy to broaden T-cell responses during chronic viral infection.CEACAM1 induces B-cell survival and is essential for protective antiviral antibody productionOf mice, macaques and men: scaling of virus dynamics and immune responsesNatural killer cell activation enhances immune pathology and promotes chronic infection by limiting CD8+ T-cell immunityT cell vaccinology: exploring the known unknowns.Virus-specific antibodies allow viral replication in the marginal zone, thereby promoting CD8(+) T-cell priming and viral controlImmunotherapy with internally inactivated virus loaded dendritic cells boosts cellular immunity but does not affect feline immunodeficiency virus infection course.Reactive oxygen species delay control of lymphocytic choriomeningitis virusAn HIV/AIDS Prophylactic vaccine is possible.Biphasic CD8+ T-Cell Defense in Simian Immunodeficiency Virus Control by Acute-Phase Passive Neutralizing Antibody Immunization.Spatiotemporally restricted arenavirus replication induces immune surveillance and type I interferon-dependent tumour regression.Multiple sclerosis and environmental factors: the role of vitamin D, parasites, and Epstein-Barr virus infection.Persistence of viremia and production of neutralizing antibodies differentially regulated by polymorphic APOBEC3 and BAFF-R loci in friend virus-infected mice.Toso regulates differentiation and activation of inflammatory dendritic cells during persistence-prone virus infection.Oxidized ATP inhibits T-cell-mediated autoimmunity.Epstein-Barr virus nuclear antigen-1 B-cell epitopes in multiple sclerosis twins.Inhibition of the FGL2:FcγRIIB/RIII Immunosuppressive Pathway Enhances Antiviral T cell and B Cell Responses Leading to Clearance of LCMV cl-13.
P2860
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P2860
Extralymphatic virus sanctuaries as a consequence of potent T-cell activation.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
Extralymphatic virus sanctuaries as a consequence of potent T-cell activation.
@en
type
label
Extralymphatic virus sanctuaries as a consequence of potent T-cell activation.
@en
prefLabel
Extralymphatic virus sanctuaries as a consequence of potent T-cell activation.
@en
P2093
P2860
P50
P356
P1433
P1476
Extralymphatic virus sanctuaries as a consequence of potent T-cell activation.
@en
P2093
Ahmed N Hegazy
Alexandre Theocharides
Bernhard Odermatt
Bruno Eschli
Doron Merkler
Hans Hengartner
Karl S Lang
Katja Fink
Lars Hangartner
Lukas Hunziker
P2860
P2888
P304
P356
10.1038/NM1670
P407
P577
2007-11-04T00:00:00Z