Simian virus 40 large T antigen stably complexes with a 185-kilodalton host protein.
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Association of insulin receptor substrate 1 with simian virus 40 large T antigenThe CUL7 E3 ubiquitin ligase targets insulin receptor substrate 1 for ubiquitin-dependent degradationInduction of cullin 7 by DNA damage attenuates p53 functionTargeted disruption of p185/Cul7 gene results in abnormal vascular morphogenesisFbxw8 is essential for Cul1-Cul7 complex formation and for placental developmentA truncated T antigen expressed from an alternatively spliced BK virus early mRNA.Interaction and co-localization of JC virus large T antigen and the F-box protein β-transducin-repeat containing protein.Cellular transformation by Simian Virus 40 and Murine Polyoma Virus T antigens.Cul7/p185/p193 binding to simian virus 40 large T antigen has a role in cellular transformation.Association of p300 and CBP with simian virus 40 large T antigenInduction versus progression of brain tumor development: differential functions for the pRB- and p53-targeting domains of simian virus 40 T antigenAssociation of p53 binding and immortalization of primary C57BL/6 mouse embryo fibroblasts by using simian virus 40 T-antigen mutants bearing internal overlapping deletion mutationsThe cullin7 E3 ubiquitin ligase: a novel player in growth control.T antigen transgenic mouse modelsInhibition of Cullin-RING E3 ubiquitin ligase 7 by simian virus 40 large T antigen.Hypothesis: "Rogue cell"-type chromosomal damage in lymphocytes is associated with infection with the JC human polyoma virus and has implications for oncopenesisAtaxia telangiectasia-mutated damage-signaling kinase- and proteasome-dependent destruction of Mre11-Rad50-Nbs1 subunits in Simian virus 40-infected primate cellsLoss of p19(ARF) eliminates the requirement for the pRB-binding motif in simian virus 40 large T antigen-mediated transformation.Inactivation of the retinoblastoma susceptibility protein is not sufficient for the transforming function of the conserved region 2-like domain of simian virus 40 large T antigen.Simian virus 40 small t antigen activates the carboxyl-terminal transforming p53-binding domain of large T antigen.Adding an Rb-binding site to an N-terminally truncated simian virus 40 T antigen restores growth to high cell density, and the T common region in trans provides anchorage-independent growth and rapid growth in low serum concentrations.The kinetics of simian virus 40-induced progression of quiescent cells into S phase depend on four independent functions of large T antigen.Simian virus 40 large T antigen's association with the CUL7 SCF complex contributes to cellular transformation.Modified glutathione S-transferase fusion proteins for simplified analysis of protein-protein interactionsA large-scale mutation search reveals genetic heterogeneity in 3M syndrome.
P2860
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P2860
Simian virus 40 large T antigen stably complexes with a 185-kilodalton host protein.
description
1992 nî lūn-bûn
@nan
1992年の論文
@ja
1992年論文
@yue
1992年論文
@zh-hant
1992年論文
@zh-hk
1992年論文
@zh-mo
1992年論文
@zh-tw
1992年论文
@wuu
1992年论文
@zh
1992年论文
@zh-cn
name
Simian virus 40 large T antigen stably complexes with a 185-kilodalton host protein.
@en
type
label
Simian virus 40 large T antigen stably complexes with a 185-kilodalton host protein.
@en
prefLabel
Simian virus 40 large T antigen stably complexes with a 185-kilodalton host protein.
@en
P2860
P1433
P1476
Simian virus 40 large T antigen stably complexes with a 185-kilodalton host protein.
@en
P2093
D C Kohrman
M J Imperiale
P2860
P304
P407
P577
1992-03-01T00:00:00Z