about
Proteomic analysis of human very low-density lipoprotein by two-dimensional gel electrophoresis and MALDI-TOF/TOFEvidence for a common progenitor of epithelial and mesenchymal components of the liver.Searching for DNA-protein interactions by lambda phage display.Determination of anti-HIV drug concentration in human plasma by MALDI-TOF/TOF.The RNA-dependent RNA polymerase essential for post-transcriptional gene silencing in Neurospora crassa interacts with replication protein A.Determination of abacavir, amprenavir, didanosine, efavirenz, nevirapine, and stavudine concentration in human plasma by MALDI-TOF/TOF.Mitochondrial localization of PARP-1 requires interaction with mitofilin and is involved in the maintenance of mitochondrial DNA integrityDetermination of antituberculosis drug concentration in human plasma by MALDI-TOF/TOF.SILAC labeling coupled to shotgun proteomics analysis of membrane proteins of liver stem/hepatocyte allows to candidate the inhibition of TGF-beta pathway as causal to differentiation.Coexpression of IL-6 and soluble IL-6R causes nodular regenerative hyperplasia and adenomas of the liver.Cytokine-sensitive replication of hepatitis B virus in immortalized mouse hepatocyte culturesHuman haemato-endothelial precursors: cord blood CD34+ cells produce haemogenic endotheliumDiscovery of chemotherapy-associated ovarian cancer antigens by interrogating memory T cells.EIF6 over-expression increases the motility and invasiveness of cancer cells by modulating the expression of a critical subset of membrane-bound proteinsCD90+ liver cancer cells modulate endothelial cell phenotype through the release of exosomes containing H19 lncRNA.Identification of a bipotential precursor cell in hepatic cell lines derived from transgenic mice expressing cyto-Met in the liverElucidation of lipoprotein particles structure by proteomic analysis.Conventional protein kinase C inhibition prevents alpha interferon-mediated hepatitis C virus replicon clearance by impairing STAT activationApplying proteomic technology to clinical virology.Molecular mechanisms controlling the phenotype and the EMT/MET dynamics of hepatocyte.Spike-in SILAC proteomic approach reveals the vitronectin as an early molecular signature of liver fibrosis in hepatitis C infections with hepatic iron overload.TGFβ overrides HNF4α tumor suppressing activity through GSK3β inactivation: implication for hepatocellular carcinoma gene therapy.Ferritin heavy chain is the host factor responsible for HCV-induced inhibition of apoB-100 production and is required for efficient viral infection.Autophagy protects cells from HCV-induced defects in lipid metabolism.The stable repression of mesenchymal program is required for hepatocyte identity: a novel role for hepatocyte nuclear factor 4α.Proteomic analysis reveals a major role for contact inhibition in the terminal differentiation of hepatocytes.ERK5/MAPK is activated by TGFbeta in hepatocytes and required for the GSK-3beta-mediated Snail protein stabilization.TGFbeta-induced EMT requires focal adhesion kinase (FAK) signaling.Gene regulation by homeobox transcription factor Prox1 in murine hepatoblasts.Activation of Vgamma9Vdelta2 T cells by non-peptidic antigens induces the inhibition of subgenomic HCV replication.Murine hepatocyte cell lines promote expansion and differentiation of NK cells from stem cell precursors.MMH cells: An in vitro model for the study of retinol-binding protein secretion regulated by retinol.Oxidation affects the regulation of hepatic lipid synthesis by chylomicron remnants.Epigenetic control of EMT/MET dynamics: HNF4α impacts DNMT3s through miRs-29.Transgenic expression in the liver of truncated Met blocks apoptosis and permits immortalization of hepatocytes.An epistatic mini-circuitry between the transcription factors Snail and HNF4α controls liver stem cell and hepatocyte features exhorting opposite regulation on stemness-inhibiting microRNAs.Steatosis and intrahepatic lymphocyte recruitment in hepatitis C virus transgenic mice.Hepatitis C virus inhibits interferon signaling through up-regulation of protein phosphatase 2A.Impaired interferon type I signalling in the liver modulates the hepatic acute phase response in hepatitis C virus transgenic mice.Hepatitis C virus production requires apolipoprotein A-I and affects its association with nascent low-density lipoproteins.
P50
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P50
description
hulumtues
@sq
onderzoeker
@nl
researcher
@en
հետազոտող
@hy
name
Marco Tripodi
@ast
Marco Tripodi
@en
Marco Tripodi
@es
Marco Tripodi
@sl
type
label
Marco Tripodi
@ast
Marco Tripodi
@en
Marco Tripodi
@es
Marco Tripodi
@sl
prefLabel
Marco Tripodi
@ast
Marco Tripodi
@en
Marco Tripodi
@es
Marco Tripodi
@sl
P1053
F-1143-2011
P106
P1153
7004492791
P21
P31
P3829
P496
0000-0001-6135-971X