Trichostatin A, a histone deacetylase inhibitor, attenuates invasiveness and reactivates E-cadherin expression in immortalized endometriotic cells.
about
Molecular aspects of development and regulation of endometriosisSelective inhibition of prostaglandin E2 receptors EP2 and EP4 inhibits adhesion of human endometriotic epithelial and stromal cells through suppression of integrin-mediated mechanismsPPARγ activation inhibits growth and survival of human endometriotic cells by suppressing estrogen biosynthesis and PGE2 signaling.Novel three-dimensional in vitro models of ovarian endometriosis.Selective inhibition of prostaglandin E2 receptors EP2 and EP4 induces apoptosis of human endometriotic cells through suppression of ERK1/2, AKT, NFkappaB, and beta-catenin pathways and activation of intrinsic apoptotic mechanisms.The dynamics of nuclear receptors and nuclear receptor coregulators in the pathogenesis of endometriosis.Involvement of members of the cadherin superfamily in cancerTSA suppresses miR-106b-93-25 cluster expression through downregulation of MYC and inhibits proliferation and induces apoptosis in human EMC.HDAC1 and HDAC2 are differentially expressed in endometriosis.DNA methylation in endometriosis (Review).Tranylcypromine, a lysine-specific demethylase 1 (LSD1) inhibitor, suppresses lesion growth and improves generalized hyperalgesia in mouse with induced endometriosis.Hereditary diffuse gastric cancer and lost cell polarity: a short path to cancer.The role of iron in the pathogenesis of endometriosis.Genome-wide DNA methylation profiling in cultured eutopic and ectopic endometrial stromal cells.Epigenetics of endometriosis.Endometriosis is characterized by a distinct pattern of histone 3 and histone 4 lysine modifications.Aberrant DNA methylation status of endometriosis: epigenetics as the pathogenesis, biomarker and therapeutic target.Trichostatin A, a histone deacetylase inhibitor, reduces lesion growth and hyperalgesia in experimentally induced endometriosis in mice.A current view of the role of epigenetic changes in the aetiopathogenesis of endometriosis.Reduced Expression of Eukaryotic Translation Initiation Factor 3 Subunit e and Its Possible Involvement in the Epithelial-Mesenchymal Transition in Endometriosis.Recent insights on the genetics and epigenetics of endometriosis.Progesterone resistance in endometriosis: origins, consequences and interventions.Role of Oxidative Stress in Epigenetic Modification in Endometriosis.HDAC inhibitors regulate claudin-1 expression in colon cancer cells through modulation of mRNA stability.Trichostatin A Induces NAG-1 Expression and Apoptosis in Human Endometriotic Stromal Cells.
P2860
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P2860
Trichostatin A, a histone deacetylase inhibitor, attenuates invasiveness and reactivates E-cadherin expression in immortalized endometriotic cells.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年学术文章
@wuu
2007年学术文章
@zh-cn
2007年学术文章
@zh-hans
2007年学术文章
@zh-my
2007年学术文章
@zh-sg
2007年學術文章
@yue
2007年學術文章
@zh
2007年學術文章
@zh-hant
name
Trichostatin A, a histone deac ...... ortalized endometriotic cells.
@en
type
label
Trichostatin A, a histone deac ...... ortalized endometriotic cells.
@en
prefLabel
Trichostatin A, a histone deac ...... ortalized endometriotic cells.
@en
P2860
P356
P1476
Trichostatin A, a histone deac ...... ortalized endometriotic cells.
@en
P2093
Anna Starzinski-Powitz
P2860
P304
P356
10.1177/1933719107302913
P50
P577
2007-05-01T00:00:00Z