Analysis of human papillomavirus type 16 late mRNA 3' processing signals in vitro and in vivo.
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Papillomavirus genome structure, expression, and post-transcriptional regulationA 57-nucleotide upstream early polyadenylation element in human papillomavirus type 16 interacts with hFip1, CstF-64, hnRNP C1/C2, and polypyrimidine tract binding protein.The reciprocal regulation between splicing and 3'-end processingA downstream polyadenylation element in human papillomavirus type 16 L2 encodes multiple GGG motifs and interacts with hnRNP H.Regulation of human papillomavirus type 31 polyadenylation during the differentiation-dependent life cyclePapillomavirus capsid protein expression level depends on the match between codon usage and tRNA availability.Early polyadenylation signals of human papillomavirus type 31 negatively regulate capsid gene expression.The human papillomavirus type 16 negative regulatory RNA element interacts with three proteins that act at different posttranscriptional levels.The Rev protein of human immunodeficiency virus type 1 counteracts the effect of an AU-rich negative element in the human papillomavirus type 1 late 3' untranslated region.Efficient expression of the human papillomavirus type 16 L1 protein in epithelial cells by using Rev and the Rev-responsive element of human immunodeficiency virus or the cis-acting transactivation element of simian retrovirus type 1Identification of nuclear and cytoplasmic proteins that interact specifically with an AU-rich, cis-acting inhibitory sequence in the 3' untranslated region of human papillomavirus type 1 late mRNAs.A cellular 65-kDa protein recognizes the negative regulatory element of human papillomavirus late mRNAAnalysis of novel human papillomavirus type 16 late mRNAs in differentiated W12 cervical epithelial cellsCharacterization of chimeric full-length molecular clones of Aleutian mink disease parvovirus (ADV): identification of a determinant governing replication of ADV in cell culture.Sequences homologous to 5' splice sites are required for the inhibitory activity of papillomavirus late 3' untranslated regions.Expression of human papillomavirus type 11 L1 protein in insect cells: in vivo and in vitro assembly of viruslike particlesAn element in the bovine papillomavirus late 3' untranslated region reduces polyadenylated cytoplasmic RNA levels.Human papillomavirus type 16 late gene expression is regulated by cellular RNA processing factors in response to epithelial differentiationtRNASer(CGA) differentially regulates expression of wild-type and codon-modified papillomavirus L1 genes.SF2/ASF binds the human papillomavirus type 16 late RNA control element and is regulated during differentiation of virus-infected epithelial cells.The inhibitory activity of the AU-rich RNA element in the human papillomavirus type 1 late 3' untranslated region correlates with its affinity for the elav-like HuR protein.Enhancement of capsid gene expression: preparing the human papillomavirus type 16 major structural gene L1 for DNA vaccination purposes.mRNA instability elements in the human papillomavirus type 16 L2 coding region.Specific inactivation of inhibitory sequences in the 5' end of the human papillomavirus type 16 L1 open reading frame results in production of high levels of L1 protein in human epithelial cells.The human papillomavirus type 31 late 3' untranslated region contains a complex bipartite negative regulatory elementActivity of the human papillomavirus type 16 late negative regulatory element is partly due to four weak consensus 5' splice sites that bind a U1 snRNP-like complexHuman papillomavirus type 31b late gene expression is regulated through protein kinase C-mediated changes in RNA processing.Mutational inactivation of two distinct negative RNA elements in the human papillomavirus type 16 L2 coding region induces production of high levels of L2 in human cells.Polypyrimidine tract binding protein induces human papillomavirus type 16 late gene expression by interfering with splicing inhibitory elements at the major late 5' splice site, SD3632Keratinocyte Differentiation-Dependent Human Papillomavirus Gene Regulation.A negative regulatory element in the human papillomavirus type 16 genome acts at the level of late mRNA stability.The alternative splicing factor hnRNP A1 is up-regulated during virus-infected epithelial cell differentiation and binds the human papillomavirus type 16 late regulatory element.Identification of an hnRNP A1-dependent splicing silencer in the human papillomavirus type 16 L1 coding region that prevents premature expression of the late L1 gene.
P2860
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P2860
Analysis of human papillomavirus type 16 late mRNA 3' processing signals in vitro and in vivo.
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1990 nî lūn-bûn
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name
Analysis of human papillomavir ...... signals in vitro and in vivo.
@en
type
label
Analysis of human papillomavir ...... signals in vitro and in vivo.
@en
prefLabel
Analysis of human papillomavir ...... signals in vitro and in vivo.
@en
P2093
P2860
P1433
P1476
Analysis of human papillomavir ...... signals in vitro and in vivo.
@en
P2093
Clements JB
Kennedy IM
P2860
P304
P407
P577
1990-04-01T00:00:00Z