Mitochondria transmit apoptosis signalling in cardiomyocyte-like cells and isolated hearts exposed to experimental ischemia-reperfusion injury. - Wikidata - awgldk - TriplyDB

Mitochondria transmit apoptosis signalling in cardiomyocyte-like cells and isolated hearts exposed to experimental ischemia-reperfusion injury.

Mitochondria transmit apoptosis signalling in cardiomyocyte-like cells and isolated hearts exposed to experimental ischemia-reperfusion injury.

http://www.wikidata.org/entity/Q40108880

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Mitochondria as a therapeutic target in heart failure Anti-Oxidants in Parkinson's Disease Therapy: A Critical Point of View Prevention of diabetic nephropathy in Ins2(+/)⁻(AkitaJ) mice by the mitochondria-targeted therapy MitoQ.Mitochondrial targeting of vitamin E succinate enhances its pro-apoptotic and anti-cancer activity via mitochondrial complex II Lipocalin-2 as mediator of chemokine expression and granulocyte infiltration during ischemia and reperfusion.H9c2 and HL-1 cells demonstrate distinct features of energy metabolism, mitochondrial function and sensitivity to hypoxia-reoxygenation.Metabolic stress, reactive oxygen species, and arrhythmia.Mitochondrial Targeted Antioxidant in Cerebral Ischemia Reduction in mitochondrial iron alleviates cardiac damage during injury Mitochondrial pathophysiology, reactive oxygen species, and cardiovascular diseases P-glycoprotein (Mdr1a/1b) and breast cancer resistance protein (Bcrp) decrease the uptake of hydrophobic alkyl triphenylphosphonium cations by the brain.Mitochondria-targeted antioxidants for treatment of Parkinson's disease: preclinical and clinical outcomes.Pathogenesis of chronic cardiorenal syndrome: is there a role for oxidative stress?Nanoparticle delivery of mitoprotective agents to target ischemic heart disease.Neuroprotective Efficacy of Mitochondrial Antioxidant MitoQ in Suppressing Peroxynitrite-Mediated Mitochondrial Dysfunction Inflicted by Lead Toxicity in the Rat Brain.Resolution of mitochondrial oxidative stress rescues coronary collateral growth in Zucker obese fatty rats.Daxx inhibits stress-induced apoptosis in cardiac myocytes.Protection against renal ischemia-reperfusion injury in vivo by the mitochondria targeted antioxidant MitoQ Synthesis of triphenylphosphonium vitamin E derivatives as mitochondria-targeted antioxidants.MitoQ improves mitochondrial dysfunction in heart failure induced by pressure overload.Inhibition of Intracellular Type 10 Adenylyl Cyclase Protects Cortical Neurons Against Reperfusion-Induced Mitochondrial Injury and Apoptosis.Triphenylphosphonium and D-α-tocopheryl polyethylene glycol 1000 succinate-modified, tanshinone IIA-loaded lipid-polymeric nanocarriers for the targeted therapy of myocardial infarction.

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P2860

Mitochondria transmit apoptosis signalling in cardiomyocyte-like cells and isolated hearts exposed to experimental ischemia-reperfusion injury.

http://www.wikidata.org/entity/Q40108880

description

2007 nî lūn-bûn

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2007年の論文

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2007年学术文章

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2007年学术文章

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2007年学术文章

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2007年学术文章

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2007年学术文章

@zh-sg

2007年學術文章

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2007年學術文章

@zh

2007年學術文章

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name

Mitochondria transmit apoptosi ...... l ischemia-reperfusion injury.

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Mitochondria transmit apoptosi ...... l ischemia-reperfusion injury.

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Mitochondria transmit apoptosi ...... l ischemia-reperfusion injury.

@en

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Caroline Lidebjer

http://www.w3.org/2001/XMLSchema#string

Cecilia Widén

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Emma Swettenham

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Helge Dalen

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John P Headrick

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Lan-Feng Dong

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Nina Gellert

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Paul K Witting

http://www.w3.org/2001/XMLSchema#string

Renata Zobalova

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Xiu-Fang Wang

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P2860

Apoptosis repressor with caspase recruitment domain protects against cell death by interfering with Bax activation

Movement of Bax from the cytosol to mitochondria during apoptosis

Interactions of mitochondria-targeted and untargeted ubiquinones with the mitochondrial respiratory chain and reactive oxygen species. Implications for the use of exogenous ubiquinones as therapies and experimental tools

Sodium nitroprusside induces apoptosis of H9C2 cardiac muscle cells in a c-Jun N-terminal kinase-dependent manner.

Selective targeting of a redox-active ubiquinone to mitochondria within cells: antioxidant and antiapoptotic properties.

Signal transduction of nitric oxide donor-induced protection in hydrogen peroxide-mediated apoptosis in H9C2 cardiomyoblasts.

Nuclear factor-kappaB represses hypoxia-induced mitochondrial defects and cell death of ventricular myocytes.

Apoptosis in cardiac diseases: stress- and mitogen-activated signaling pathways.

Apoptosis in relevant clinical situations: contribution of apoptosis in myocardial infarction.

Mitochondria, oxygen free radicals, disease and ageing.

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148-162

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10.1179/135100007X200227

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3

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17623522

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reperfusion injury

apoptotic process