Induction of Epstein-Barr virus lytic cycle by tumor-promoting and non-tumor-promoting phorbol esters requires active protein kinase C.
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Identification of Novel Small Organic Compounds with Diverse Structures for the Induction of Epstein-Barr Virus (EBV) Lytic Cycle in EBV-Positive Epithelial MalignanciesFunctional analysis of the CD4(+) T-cell response to Epstein-Barr virus: T-cell-mediated activation of resting B cells and induction of viral BZLF1 expression.Activation and repression of Epstein-Barr Virus and Kaposi's sarcoma-associated herpesvirus lytic cycles by short- and medium-chain fatty acidsProtein kinase C-independent activation of the Epstein-Barr virus lytic cycle.Molecular interactions of Epstein-Barr virus capsid proteins.Role of TAF4 in transcriptional activation by Rta of Epstein-Barr Virus.Interplay between PKCδ and Sp1 on histone deacetylase inhibitor-mediated Epstein-Barr virus reactivation.MCAF1 and Rta-activated BZLF1 transcription in Epstein-Barr virus.Synthesis of infectious human papillomavirus type 18 in differentiating epithelium transfected with viral DNAProtein kinase inhibitors that inhibit induction of lytic program and replication of Epstein-Barr virus.Role of RNF4 in the ubiquitination of Rta of Epstein-Barr virus.TRIM5α Promotes Ubiquitination of Rta from Epstein-Barr Virus to Attenuate Lytic Progression.Cellular differentiation regulator BLIMP1 induces Epstein-Barr virus lytic reactivation in epithelial and B cells by activating transcription from both the R and Z promoters.Transforming growth factor beta 1 stimulates expression of the Epstein-Barr virus BZLF1 immediate-early gene product ZEBRA by an indirect mechanism which requires the MAPK kinase pathway.Regulation of the Epstein-Barr virus C promoter by AUF1 and the cyclic AMP/protein kinase A signaling pathway.Divergent requirements for the MAPK(ERK) signal transduction pathway during initial virus infection of quiescent primary B cells and disruption of Epstein-Barr virus latency by phorbol esters.Activators of the Epstein-Barr virus lytic program concomitantly induce apoptosis, but lytic gene expression protects from cell death.Cellular immediate-early gene expression occurs kinetically upstream of Epstein-Barr virus bzlf1 and brlf1 following cross-linking of the B cell antigen receptor in the Akata Burkitt lymphoma cell line.Three pathways of Epstein-Barr virus gene activation from EBNA1-positive latency in B lymphocytes.De novo protein synthesis is required for lytic cycle reactivation of Epstein-Barr virus, but not Kaposi's sarcoma-associated herpesvirus, in response to histone deacetylase inhibitors and protein kinase C agonists.Activation of Epstein-Barr virus by saliva from Sjogren's syndrome patients.EBV Reactivation and Chromosomal Polysomies: Euphorbia tirucalli as a Possible Cofactor in Endemic Burkitt Lymphoma.Prevalence of Epstein-Barr virus DNA and Porphyromonas gingivalis in Japanese peri-implantitis patients.
P2860
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P2860
Induction of Epstein-Barr virus lytic cycle by tumor-promoting and non-tumor-promoting phorbol esters requires active protein kinase C.
description
1991 nî lūn-bûn
@nan
1991年の論文
@ja
1991年学术文章
@wuu
1991年学术文章
@zh-cn
1991年学术文章
@zh-hans
1991年学术文章
@zh-my
1991年学术文章
@zh-sg
1991年學術文章
@yue
1991年學術文章
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1991年學術文章
@zh-hant
name
Induction of Epstein-Barr viru ...... uires active protein kinase C.
@en
type
label
Induction of Epstein-Barr viru ...... uires active protein kinase C.
@en
prefLabel
Induction of Epstein-Barr viru ...... uires active protein kinase C.
@en
P2093
P2860
P1433
P1476
Induction of Epstein-Barr viru ...... uires active protein kinase C.
@en
P2093
P2860
P304
P407
P577
1991-12-01T00:00:00Z