Downregulation of class I major histocompatibility complex surface expression by varicella-zoster virus involves open reading frame 66 protein kinase-dependent and -independent mechanisms.
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Herpes simplex virus and varicella zoster virus, the house guests who never leaveImmune regulation of human herpesviruses and its implications for human transplantationA comparison of herpes simplex virus type 1 and varicella-zoster virus latency and reactivationViral inhibition of the transporter associated with antigen processing (TAP): a striking example of functional convergent evolutionVaricellovirus UL 49.5 proteins differentially affect the function of the transporter associated with antigen processing, TAP.Viral load, clinical disease severity and cellular immune responses in primary varicella zoster virus infection in Sri Lanka.Sex differences underlying orofacial varicella zoster associated pain in rats.Molecular mechanisms of varicella zoster virus pathogenesis.Varicella-zosterCharacterization of the host immune response in human Ganglia after herpes zoster.Pathogenesis of varicelloviruses in primatesThe capacity of UL49.5 proteins to inhibit TAP is widely distributed among members of the genus Varicellovirus.Simian varicella virus pathogenesis.Induction and function of virus-specific CD4+ T cell responses.Us3 kinase encoded by herpes simplex virus 1 mediates downregulation of cell surface major histocompatibility complex class I and evasion of CD8+ T cellsVaricella zoster virus latency.Varicella-zoster virus (VZV) infection of neurons derived from human embryonic stem cells: direct demonstration of axonal infection, transport of VZV, and productive neuronal infection.Review: The neurobiology of varicella zoster virus infection.Varicella Viruses Inhibit Interferon-Stimulated JAK-STAT Signaling through Multiple MechanismsAn in vitro model of latency and reactivation of varicella zoster virus in human stem cell-derived neuronsVaricella-zoster virus inhibition of the NF-κB pathway during infection of human dendritic cells: role for open reading frame 61 as a modulator of NF-κB activityNeuronal changes induced by Varicella Zoster Virus in a rat model of postherpetic neuralgia.Simian varicella virus gene expression during acute and latent infection of rhesus macaques.Varicella-zoster virus infects human embryonic stem cell-derived neurons and neurospheres but not pluripotent embryonic stem cells or early progenitors.Varicella-Zoster Virus and Herpes Simplex Virus 1 Differentially Modulate NKG2D Ligand Expression during Productive Infection.The ORF61 Protein Encoded by Simian Varicella Virus and Varicella-Zoster Virus Inhibits NF-κB Signaling by Interfering with IκBα Degradation.Human herpesviridae methods of natural killer cell evasionVaccination Targeting Native Receptors to Enhance the Function and Proliferation of Chimeric Antigen Receptor (CAR)-Modified T Cells.Immune regulation and evasion of Mammalian host cell immunity during viral infection.Age and immune status of rhesus macaques impact simian varicella virus gene expression in sensory gangliaProductive vs non-productive infection by cell-free varicella zoster virus of human neurons derived from embryonic stem cells is dependent upon infectious viral doseT-cell immunity to human alphaherpesviruses.Regulation of the catalytic activity of herpes simplex virus 1 protein kinase Us3 by autophosphorylation and its role in pathogenesis.Histone deacetylases 1 and 2 are phosphorylated at novel sites during varicella-zoster virus infection.Clinical and molecular aspects of varicella zoster virus infection.Cytomegalovirus and varicella-zoster virus vaccines in hematopoietic stem cell transplantation.Varicella zoster virus immune evasion strategies.Varicella-zoster virus open reading frame 66 protein kinase and its relationship to alphaherpesvirus US3 kinases.Varicella zoster virus-induced pain and post-herpetic neuralgia in the human host and in rodent animal models.Varicella-zoster virus and herpes simplex virus 1 can infect and replicate in the same neurons whether co- or superinfected.
P2860
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P2860
Downregulation of class I major histocompatibility complex surface expression by varicella-zoster virus involves open reading frame 66 protein kinase-dependent and -independent mechanisms.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年学术文章
@wuu
2007年学术文章
@zh-cn
2007年学术文章
@zh-hans
2007年学术文章
@zh-my
2007年学术文章
@zh-sg
2007年學術文章
@yue
2007年學術文章
@zh
2007年學術文章
@zh-hant
name
Downregulation of class I majo ...... t and -independent mechanisms.
@en
type
label
Downregulation of class I majo ...... t and -independent mechanisms.
@en
prefLabel
Downregulation of class I majo ...... t and -independent mechanisms.
@en
P2093
P2860
P356
P1433
P1476
Downregulation of class I majo ...... nt and -independent mechanisms
@en
P2093
Allison Abendroth
Amie J Eisfeld
Paul R Kinchington
P2860
P304
P356
10.1128/JVI.00711-07
P407
P577
2007-06-13T00:00:00Z