The satiety effect of cholecystokinin: a progress report.
about
Cognitive and neuronal systems underlying obesity.Roux-en-Y gastric bypass surgery increases number but not density of CCK-, GLP-1-, 5-HT-, and neurotensin-expressing enteroendocrine cells in rats.Canine obesity: an overview.The blood-brain barrier: connecting the gut and the brain.Integration of satiety signals by the central nervous system.Hypothalamus as an endocrine organ.The actions of bombesin-like peptides on food intake.Neural and hormonal mechanisms regulating food intake.Physiological consequences of the passage of peptides across the blood-brain barrier.Melanotropins as growth factors.Insulin detemir is not transported across the blood-brain barrier.The cholecystokinin receptor antagonist L364,718 increases food intake in the rat by attenuation of the action of endogenous cholecystokinin.[The regulation of feed intake and selection with special reference to poultry]Validation and characterization of a novel method for selective vagal deafferentation of the gut.Conditioned food aversions and satiety signals.
P2860
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P2860
The satiety effect of cholecystokinin: a progress report.
description
1981 nî lūn-bûn
@nan
1981年の論文
@ja
1981年論文
@yue
1981年論文
@zh-hant
1981年論文
@zh-hk
1981年論文
@zh-mo
1981年論文
@zh-tw
1981年论文
@wuu
1981年论文
@zh
1981年论文
@zh-cn
name
The satiety effect of cholecystokinin: a progress report.
@en
type
label
The satiety effect of cholecystokinin: a progress report.
@en
prefLabel
The satiety effect of cholecystokinin: a progress report.
@en
P2093
P1433
P1476
The satiety effect of cholecystokinin: a progress report.
@en
P2093
P356
10.1016/0196-9781(81)90011-5
P478
P577
1981-01-01T00:00:00Z