The hepatitis C virus core protein of genotypes 3a and 1b downregulates insulin receptor substrate 1 through genotype-specific mechanisms. - Wikidata - awgldk - TriplyDB

The hepatitis C virus core protein of genotypes 3a and 1b downregulates insulin receptor substrate 1 through genotype-specific mechanisms.

The hepatitis C virus core protein of genotypes 3a and 1b downregulates insulin receptor substrate 1 through genotype-specific mechanisms.

http://www.wikidata.org/entity/Q40140184

about

Hepatic steatosis: a benign disease or a silent killer Hepatitis C virus infection protein network Hepatitis C virus and type 2 diabetes NAFLD and NASH in HCV Infection: Prevalence and Significance in Hepatic and Extrahepatic Manifestations Hepatitis C virus infection and type 1 and type 2 diabetes mellitus The biology and mechanism of action of suppressor of cytokine signaling 3 Liver steatosis in hepatitis C patients Pathophysiology of insulin resistance and steatosis in patients with chronic viral hepatitis Targeting host factors: A novel rationale for the management of hepatitis C virus Molecular mechanisms of insulin resistance in chronic hepatitis C Supplementing Conventional Treatment with Pycnogenol® May Improve Hepatitis C Virus-Associated Type 2 Diabetes: A Mini Review Impact of IL28B-related single nucleotide polymorphisms on liver histopathology in chronic hepatitis C genotype 2 and 3 Metabolic syndrome in chronic hepatitis C infection: does it still matter in the era of directly acting antiviral therapy?Molecular mechanism of hepatitis C virus-induced glucose metabolic disorders Association of hepatitis C virus with insulin resistance: evidences from animal studies and clinical studies Production and pathogenicity of hepatitis C virus core gene products.Importance of hepatitis C virus-associated insulin resistance: therapeutic strategies for insulin sensitization Hepatitis C virus NS5A activates the mammalian target of rapamycin (mTOR) pathway, contributing to cell survival by disrupting the interaction between FK506-binding protein 38 (FKBP38) and mTOR Hepatitis C virus E2 protein involve in insulin resistance through an impairment of Akt/PKB and GSK3β signaling in hepatocytes.Mutual antagonism between circadian protein period 2 and hepatitis C virus replication in hepatocytes.Insulin resistance and liver steatosis in chronic hepatitis C infection genotype 3.The hepatitis C virus modulates insulin signaling pathway in vitro promoting insulin resistance Inhibition of IRS-1 by hepatitis C virus infection leads to insulin resistance in a PTEN-dependent manner.Insulin resistance and chronic liver disease.Steatosis is an independent predictor of relapse following rapid virologic response in patients with HCV genotype 3.Hepatitis C virus infection: molecular pathways to steatosis, insulin resistance and oxidative stress Hepatitis C virus infection: molecular pathways to insulin resistance.Chronic hepatitis c genotype-4 infection: role of insulin resistance in hepatocellular carcinoma.Hepatitis C virus core protein decreases lipid droplet turnover: a mechanism for core-induced steatosis.Hepatitis C Virus Driven AXL Expression Suppresses the Hepatic Type I Interferon Response New treatments for chronic hepatitis C.The impact of host factors on management of hepatitis C virus.Metabolic disorders and steatosis in patients with chronic hepatitis C: metabolic strategies for antiviral treatments.Diabetes and Hepatitis C: A Two-Way Association.Hepatitis C virus core protein upregulates serine phosphorylation of insulin receptor substrate-1 and impairs the downstream akt/protein kinase B signaling pathway for insulin resistance.Influence of insulin resistance on the development of hepatocellular carcinoma after antiviral treatment for non-cirrhotic patients with chronic hepatitis C.Dipeptidyl peptidase-4: a key player in chronic liver disease.Peroxisome proliferator-activated receptors and hepatitis C virus-induced insulin resistance Reactivation of the insulin-like growth factor-II signaling pathway in human hepatocellular carcinoma.Viral clearance is associated with improved insulin resistance in genotype 1 chronic hepatitis C but not genotype 2/3

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P2860

The hepatitis C virus core protein of genotypes 3a and 1b downregulates insulin receptor substrate 1 through genotype-specific mechanisms.

http://www.wikidata.org/entity/Q40140184

description

2007 nî lūn-bûn

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2007年の論文

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2007年論文

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2007年論文

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2007年論文

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2007年論文

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2007年論文

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2007年论文

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2007年论文

@zh

2007年论文

@zh-cn

name

The hepatitis C virus core pro ...... genotype-specific mechanisms.

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type

label

The hepatitis C virus core pro ...... genotype-specific mechanisms.

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prefLabel

The hepatitis C virus core pro ...... genotype-specific mechanisms.

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The hepatitis C virus core pro ...... genotype-specific mechanisms.

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Alessandra Mangia

http://www.w3.org/2001/XMLSchema#string

Francesco Negro

http://www.w3.org/2001/XMLSchema#string

Michelangelo Foti

http://www.w3.org/2001/XMLSchema#string

Paolo Pugnale

http://www.w3.org/2001/XMLSchema#string

Sophie Clément

http://www.w3.org/2001/XMLSchema#string

Stéphanie Conzelman

http://www.w3.org/2001/XMLSchema#string

P2860

Hepatitis C Virus Down-Regulates Insulin Receptor Substrates 1 and 2 through Up-Regulation of Suppressor of Cytokine Signaling 3

Regulation of insulin/insulin-like growth factor-1 signaling by proteasome-mediated degradation of insulin receptor substrate-2.

Impaired IRS-1/PI3-kinase signaling in patients with HCV: a mechanism for increased prevalence of type 2 diabetes.

Prevalence of type 2 diabetes mellitus among persons with hepatitis C virus infection in the United States.

P304

1164-1171

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scholarly article

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10.1002/HEP.21634

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5

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45

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Valerio Pazienza

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2007-05-01T00:00:00Z

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6364859

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17465001

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