Co-culturing human prostate carcinoma cells with hepatocytes leads to increased expression of E-cadherin.
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Cancer Stem Cells and Epithelial-to-Mesenchymal Transition (EMT)-Phenotypic Cells: Are They Cousins or Twins?MicroRNAs that affect prostate cancer: emphasis on prostate cancer in African AmericansAn update on microRNAs as colorectal cancer biomarkers: where are we and what's next?Role of cancer microenvironment in metastasis: focus on colon cancerLHRH-conjugated lytic peptides directly target prostate cancer cells.Epithelial-to-mesenchymal transition in prostate cancer: paradigm or puzzle?Hepatic nonparenchymal cells drive metastatic breast cancer outgrowth and partial epithelial to mesenchymal transitionThe mitogen-activated protein (MAP) kinases p38 and extracellular signal-regulated kinase (ERK) are involved in hepatocyte-mediated phenotypic switching in prostate cancer cellsBreast carcinoma cells re-express E-cadherin during mesenchymal to epithelial reverting transition.E-cadherin as an indicator of mesenchymal to epithelial reverting transitions during the metastatic seeding of disseminated carcinomas."A novel in vivo model for the study of human breast cancer metastasis using primary breast tumor-initiating cells from patient biopsies".E-cadherin and the cytoskeletal network in colorectal cancer development and metastasis.The role of epithelial plasticity in prostate cancer dissemination and treatment resistanceThe role of hepatocyte nuclear factor 4alpha in metastatic tumor formation of hepatocellular carcinoma and its close relationship with the mesenchymal-epithelial transition markers.Novel 3D co-culture model for epithelial-stromal cells interaction in prostate cancer.Lung epithelial cells induce both phenotype alteration and senescence in breast cancer cells.MiRNA 26a expression in a novel panel of African American prostate cancer cell linesMetastatic progression of prostate cancer and e-cadherin regulation by zeb1 and SRC family kinases.Role of microRNAs in the regulation of breast cancer stem cells.The epidermal growth factor receptor (EGFR) is proteolytically modified by the Matriptase-Prostasin serine protease cascade in cultured epithelial cells.Expression of pluripotent stem cell reprogramming factors by prostate tumor initiating cells.Regulation of stem cell plasticity: mechanisms and relevance to tissue biology and cancer.A microphysiological system model of therapy for liver micrometastasesThe use of chelated radionuclide (samarium-153-ethylenediaminetetramethylenephosphonate) to modulate phenotype of tumor cells and enhance T cell-mediated killingClaudin-2 promotes breast cancer liver metastasis by facilitating tumor cell interactions with hepatocytesNuclear Kaiso indicates aggressive prostate cancers and promotes migration and invasiveness of prostate cancer cells.Hepatocyte growth factor increases the invasive potential of PC-3 human prostate cancer cells via an ERK/MAPK and Zeb-1 signaling pathway.An evolutionary explanation for the perturbation of the dynamics of metastatic tumors induced by surgery and acute inflammation.MicroRNA-200c modulates epithelial-to-mesenchymal transition (EMT) in human colorectal cancer metastasisSciellin mediates mesenchymal-to-epithelial transition in colorectal cancer hepatic metastasis.Hepatocyte induced re-expression of E-cadherin in breast and prostate cancer cells increases chemoresistance.Neurotrophin-3 modulates breast cancer cells and the microenvironment to promote the growth of breast cancer brain metastasis.Epithelial and mesenchymal phenotypic switchings modulate cell motility in metastasisβ-catenin as a potential key target for tumor suppression.Prostate tumor cell plasticity: a consequence of the microenvironment.Mesenchymal-epithelial transition (MET) as a mechanism for metastatic colonisation in breast cancer.Plasticity of disseminating cancer cells in patients with epithelial malignancies.Isolated, disseminated and circulating tumour cells in prostate cancer.Role of splice variants in the metastatic progression of prostate cancer.Cancer stem cells, tumor dormancy, and metastasis.
P2860
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P2860
Co-culturing human prostate carcinoma cells with hepatocytes leads to increased expression of E-cadherin.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
Co-culturing human prostate ca ...... ased expression of E-cadherin.
@en
type
label
Co-culturing human prostate ca ...... ased expression of E-cadherin.
@en
prefLabel
Co-culturing human prostate ca ...... ased expression of E-cadherin.
@en
P2093
P2860
P356
P1476
Co-culturing human prostate ca ...... ased expression of E-cadherin.
@en
P2093
P2860
P2888
P304
P356
10.1038/SJ.BJC.6603700
P407
P50
P577
2007-04-03T00:00:00Z