Oncogene-Expressing Senescent Melanocytes Up-Regulate MHC Class II, a Candidate Melanoma Suppressor Function.

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Oncogene-Expressing Senescent Melanocytes Up-Regulate MHC Class II, a Candidate Melanoma Suppressor Function.

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2017 nî lūn-bûn

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2017年の論文

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2017年論文

@yue

2017年論文

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2017年論文

@zh-hk

2017年論文

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2017年論文

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2017年论文

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2017年论文

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2017年论文

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Oncogene-Expressing Senescent ...... Melanoma Suppressor Function.

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Oncogene-Expressing Senescent ...... Melanoma Suppressor Function.

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Oncogene-Expressing Senescent ...... Melanoma Suppressor Function.

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P1476

Oncogene-Expressing Senescent ...... Melanoma Suppressor Function.

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P2093

Andre Ivanov

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David M Nelson

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Douglas MacKenzie

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Elizabeth Mann

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Farah Jaber-Hijazi

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Hong Wu

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Jeff S Pawlikowski

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John J Cole

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John van Tuyn

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Peter D Adams

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P2860

Senescence of activated stellate cells limits liver fibrosis

MEK1 mutations confer resistance to MEK and B-RAF inhibition.

Oncogene-induced senescence relayed by an interleukin-dependent inflammatory network.

Chemokine signaling via the CXCR2 receptor reinforces senescence

Senescence and tumour clearance is triggered by p53 restoration in murine liver carcinomas

BRAFE600-associated senescence-like cell cycle arrest of human naevi

Immunostaining for the tumour suppressor gene p16 product is a useful marker to differentiate melanoma metastasis from lymph-node nevus.

Expression of MHC class II molecules in different cellular and functional compartments is controlled by differential usage of multiple promoters of the transactivator CIITA.

Senescent fibroblasts promote epithelial cell growth and tumorigenesis: a link between cancer and aging.

Cell biology of antigen processing in vitro and in vivo.

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2197-2207

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scholarly article

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10.1016/J.JID.2017.05.030

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English

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137

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Taranjit Singh Rai

Karen Blyth

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2017-06-21T00:00:00Z

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28647344

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5613751

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