Cutting Edge: Influenza A virus activates TLR3-dependent inflammatory and RIG-I-dependent antiviral responses in human lung epithelial cells.
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Novel signaling interactions between proteinase-activated receptor 2 and Toll-like receptors in vitro and in vivoActivation of innate immune antiviral responses by Nod2Induction of innate immunity and its perturbation by influenza virusesRespiratory Syncytial Virus (RSV) Modulation at the Virus-Host Interface Affects Immune Outcome and Disease PathogenesisdsRNA-activation of TLR3 and RLR signaling: gene induction-dependent and independent effectsToll-like receptors: key players in antiviral immunityIntranasal Immunization with Pressure Inactivated Avian Influenza Elicits Cellular and Humoral Responses in MicePlatelet-activating factor receptor plays a role in lung injury and death caused by Influenza A in miceAn Ultrasensitive Mechanism Regulates Influenza Virus-Induced InflammationProcessing of Genome 5′ Termini as a Strategy of Negative-Strand RNA Viruses to Avoid RIG-I-Dependent Interferon InductionInnate immunity to influenza virus: implications for future therapyTLR3-dependent upregulation of RIG-I leads to enhanced cytokine production from cells infected with the parainfluenza virus SV5Respiratory epithelial cells orchestrate pulmonary innate immunity.Influenza virus activation of the interferon systemTLR3 activation evokes IL-6 secretion, autocrine regulation of Stat3 signaling and TLR2 expression in human bronchial epithelial cellsEvaluation of the human adaptation of influenza A/H7N9 virus in PB2 protein using human and swine respiratory tract explant culturesInsights into the increasing virulence of the swine-origin pandemic H1N1/2009 influenza virusStudy of human RIG-I polymorphisms identifies two variants with an opposite impact on the antiviral immune responseActivation of interleukin-32 pro-inflammatory pathway in response to influenza A virus infectionGreen tea catechin, epigallocatechin gallate, suppresses signaling by the dsRNA innate immune receptor RIG-ICharacterization of a novel mutation in NS1 protein of influenza A virus induced by a chemical substance for the attenuation of pathogenicityNLRX1 protein attenuates inflammatory responses to infection by interfering with the RIG-I-MAVS and TRAF6-NF-κB signaling pathwaysInduction of type I interferon by RNA viruses: cellular receptors and their substratesInfluenza A induced cellular signal transduction pathways.Disease severity is associated with differential gene expression at the early and late phases of infection in nonhuman primates infected with different H5N1 highly pathogenic avian influenza virusesInnate immune sensing and response to influenza.Mast cells and influenza a virus: association with allergic responses and beyond.Prospects for an influenza vaccine that induces cross-protective cytotoxic T lymphocytes.Infection with a Mouse-Adapted Strain of the 2009 Pandemic Virus Causes a Highly Severe Disease Associated with an Impaired T Cell Response.Differential Induction of Type I and Type III Interferons by Swine and Human Origin H1N1 Influenza A Viruses in Porcine Airway Epithelial Cells.Postinfluenza bacterial pneumonia: host defenses gone awry.p53 serves as a host antiviral factor that enhances innate and adaptive immune responses to influenza A virus.Identification and characterization of poly(I:C)-induced molecular responses attenuated by nicotine in mouse macrophages.Major histocompatibility complex class II expression and hemagglutinin subtype influence the infectivity of type A influenza virus for respiratory dendritic cells.Interferon induction and function at the mucosal surface.Limited anti-inflammatory role for interleukin-1 receptor like 1 (ST2) in the host response to murine postinfluenza pneumococcal pneumonia.Interleukin-1 receptor-associated kinase-2 genetic variant rs708035 increases NF-κB activity through promoting TRAF6 ubiquitinationRespiratory viral infections in infants: causes, clinical symptoms, virology, and immunology.Attenuation of the influenza virus sickness behavior in mice deficient in Toll-like receptor 3Prophylactic administration of bacterially derived immunomodulators improves the outcome of influenza virus infection in a murine model.
P2860
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P2860
Cutting Edge: Influenza A virus activates TLR3-dependent inflammatory and RIG-I-dependent antiviral responses in human lung epithelial cells.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年学术文章
@wuu
2007年学术文章
@zh-cn
2007年学术文章
@zh-hans
2007年学术文章
@zh-my
2007年学术文章
@zh-sg
2007年學術文章
@yue
2007年學術文章
@zh
2007年學術文章
@zh-hant
name
Cutting Edge: Influenza A viru ...... n human lung epithelial cells.
@en
type
label
Cutting Edge: Influenza A viru ...... n human lung epithelial cells.
@en
prefLabel
Cutting Edge: Influenza A viru ...... n human lung epithelial cells.
@en
P2093
P921
P1476
Cutting Edge: Influenza A viru ...... n human lung epithelial cells.
@en
P2093
Damien Vitour
Eliane Meurs
Julien Pothlichet
Michel Chignard
Ronan Le Goffic
Takashi Fujita
P304
P356
10.4049/JIMMUNOL.178.6.3368
P407
P577
2007-03-01T00:00:00Z