Acute and chronic ethanol exposure differentially regulate CB1 receptor function at glutamatergic synapses in the rat basolateral amygdala.
about
Analyses of differentially expressed genes after exposure to acute stress, acute ethanol, or a combination of both in mice.Strain-Dependent Effects of Acute Alcohol on Synaptic Vesicle Recycling and Post-Tetanic Potentiation in Medial Glutamate Inputs to the Mouse Basolateral Amygdala.Cannabinoid-1 receptor neutral antagonist reduces binge-like alcohol consumption and alcohol-induced accumbal dopaminergic signaling.Blockade of alcohol escalation and "relapse" drinking by pharmacological FAAH inhibition in male and female C57BL/6J mice.Chronic intermittent ethanol exposure selectively alters the expression of Gα subunit isoforms and RGS subtypes in rat prefrontal cortex.
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Acute and chronic ethanol exposure differentially regulate CB1 receptor function at glutamatergic synapses in the rat basolateral amygdala.
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2015 nî lūn-bûn
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2015年の論文
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2015年論文
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2015年論文
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2015年論文
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2015年論文
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name
Acute and chronic ethanol expo ...... the rat basolateral amygdala.
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type
label
Acute and chronic ethanol expo ...... the rat basolateral amygdala.
@en
prefLabel
Acute and chronic ethanol expo ...... the rat basolateral amygdala.
@en
P2860
P50
P1433
P1476
Acute and chronic ethanol expo ...... the rat basolateral amygdala.
@en
P2093
Sachin Patel
Stacey L Robinson
P2860
P304
P356
10.1016/J.NEUROPHARM.2015.12.005
P577
2015-12-18T00:00:00Z