Constitutive RelA activation mediated by Nkx3.2 controls chondrocyte viability.
about
Nuclear factor-kappaB p65 facilitates longitudinal bone growth by inducing growth plate chondrocyte proliferation and differentiation and by preventing apoptosisRole of the homeodomain transcription factor Bapx1 in mouse distal stomach developmentASB9 interacts with ubiquitous mitochondrial creatine kinase and inhibits mitochondrial functionBimolecular fluorescence complementation (BiFC) analysis as a probe of protein interactions in living cells.New insight on FGFR3-related chondrodysplasias molecular physiopathology revealed by human chondrocyte gene expression profilingDifferential gene expression of the intermediate and outer interzone layers of developing articular cartilage in murine embryos.Activation of NF-κB/p65 facilitates early chondrogenic differentiation during endochondral ossificationNkx3.2 promotes primary chondrogenic differentiation by upregulating Col2a1 transcriptionParathyroid hormone/parathyroid hormone-related protein receptor signaling is required for maintenance of the growth plate in postnatal lifeDocosahexaenoic acid induces the degradation of HPV E6/E7 oncoproteins by activating the ubiquitin-proteasome system.In vivo genome-wide analysis of multiple tissues identifies gene regulatory networks, novel functions and downstream regulatory genes for Bapx1 and its co-regulation with Sox9 in the mammalian vertebral column.Exogenous signal-independent nuclear IkappaB kinase activation triggered by Nkx3.2 enables constitutive nuclear degradation of IkappaB-alpha in chondrocytes.c-Raf promotes angiogenesis during normal growth plate maturationSuppression of Nkx3.2 by phosphatidylinositol-3-kinase signaling regulates cartilage development by modulating chondrocyte hypertrophy.Pivotal role of Bcl-2 family proteins in the regulation of chondrocyte apoptosis.CCN2/CTGF is required for matrix organization and to protect growth plate chondrocytes from cellular stress.Functions of nuclear factor kappaB in bone.Transcriptional control of chondrocyte specification and differentiation.The role of Nkx3.2 in chondrogenesis.Transcriptional induction of ADAMTS5 protein by nuclear factor-κB (NF-κB) family member RelA/p65 in chondrocytes during osteoarthritis development.WNT5A regulates chondrocyte differentiation through differential use of the CaN/NFAT and IKK/NF-kappaB pathways.Biphasic regulation of chondrocytes by Rela through induction of anti-apoptotic and catabolic target genes.Indian Hedgehog signalling triggers Nkx3.2 protein degradation during chondrocyte maturation.Cartilage-Specific and Cre-Dependent Nkx3.2 Overexpression In Vivo Causes Skeletal Dwarfism by Delaying Cartilage Hypertrophy.Aberrant activity of NKL homeobox gene NKX3-2 in a T-ALL subset.
P2860
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P2860
Constitutive RelA activation mediated by Nkx3.2 controls chondrocyte viability.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
Constitutive RelA activation mediated by Nkx3.2 controls chondrocyte viability.
@en
type
label
Constitutive RelA activation mediated by Nkx3.2 controls chondrocyte viability.
@en
prefLabel
Constitutive RelA activation mediated by Nkx3.2 controls chondrocyte viability.
@en
P2093
P2860
P356
P1433
P1476
Constitutive RelA activation mediated by Nkx3.2 controls chondrocyte viability.
@en
P2093
Dae-Won Kim
Jae Hwan Kim
Minsun Park
Seung-Won Choi
Yeryoung Yong
P2860
P2888
P304
P356
10.1038/NCB1538
P50
P577
2007-02-18T00:00:00Z