Translocation of enteropathogenic Escherichia coli across an in vitro M cell model is regulated by its type III secretion system.
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Comparative analysis of EspF variants in inhibition of Escherichia coli phagocytosis by macrophages and inhibition of E. coli translocation through human- and bovine-derived M cells.Divergent roles of Salmonella pathogenicity island 2 and metabolic traits during interaction of S. enterica serovar typhimurium with host cellsAnalysis of the virulence of an atypical enteropathogenic Escherichia coli strain in vitro and in vivo and the influence of type three secretion system.Interactions with M cells and macrophages as key steps in the pathogenesis of enterohemorrhagic Escherichia coli infectionsTypical and atypical enteropathogenic Escherichia coli bacterial translocation associated with tissue hypoperfusion in rats.Bacterial-epithelial contact is a key determinant of host innate immune responses to enteropathogenic and enteroaggregative Escherichia coli.Crohn disease--associated adherent-invasive E. coli bacteria target mouse and human Peyer's patches via long polar fimbriaeBioavailability and biodistribution of differently charged polystyrene nanoparticles upon oral exposure in rats.Progress and future of in vitro models to study translocation of nanoparticles.Escherichia albertii, a novel human enteropathogen, colonizes rat enterocytes and translocates to extra-intestinal sitesEscherichia coli O157:H7 survives within human macrophages: global gene expression profile and involvement of the Shiga toxinsM-cells: origin, morphology and role in mucosal immunity and microbial pathogenesis.Protective and destructive innate immune responses to enteropathogenic Escherichia coli and related A/E pathogens.Pathogenesis of human enterovirulent bacteria: lessons from cultured, fully differentiated human colon cancer cell linesAutophagy at the gut interface: mucosal responses to stress and the consequences for inflammatory bowel diseasesThe EspF effector, a bacterial pathogen's Swiss army knifeAlways one step ahead: How pathogenic bacteria use the type III secretion system to manipulate the intestinal mucosal immune system.In vitro and in vivo model systems for studying enteropathogenic Escherichia coli infections.Cultured enterocytes internalise bacteria across their basolateral surface for, pathogen-inhibitable, trafficking to the apical compartment.Candida albicans is able to use M cells as a portal of entry across the intestinal barrier in vitro.In vitro gastrointestinal digestion increases the translocation of polystyrene nanoparticles in an in vitro intestinal co-culture model.Interspecies comparisons on the uptake and toxicity of silver and cerium dioxide nanoparticles.Identification of adhesin-receptor interactions driving bacterial translocation through M cells.Peyer's Patches: The Immune Sensors of the Intestine.EspJ of enteropathogenic and enterohaemorrhagic Escherichia coli inhibits opsono-phagocytosis.
P2860
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P2860
Translocation of enteropathogenic Escherichia coli across an in vitro M cell model is regulated by its type III secretion system.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
Translocation of enteropathoge ...... its type III secretion system.
@en
type
label
Translocation of enteropathoge ...... its type III secretion system.
@en
prefLabel
Translocation of enteropathoge ...... its type III secretion system.
@en
P2860
P1476
Translocation of enteropathoge ...... its type III secretion system
@en
P2093
Caroline Sands
Mark A Jepson
P2860
P304
P356
10.1111/J.1462-5822.2007.00891.X
P577
2007-02-09T00:00:00Z