about
The BK channel accessory beta1 subunit determines alcohol-induced cerebrovascular constrictionA mechanism of rapidly reversible cerebral ventricular enlargement independent of tissue atrophy.Essential role for smooth muscle BK channels in alcohol-induced cerebrovascular constrictionFetal brain during a binge drinking episode: a dynamic susceptibility contrast MRI fetal brain perfusion study.Relative importance of systemic determinants of retinal arteriolar and venular caliber: the atherosclerosis risk in communities study.Type 2 ryanodine receptors are highly sensitive to alcoholEndothelial Nitric Oxide Mediates Caffeine Antagonism of Alcohol-Induced Cerebral Artery Constriction.Relationship of blood pressure and other factors to serial retinal arteriolar diameter measurements over time: the beaver dam eye study.Role of magnesium and calcium in alcohol-induced hypertension and strokes as probed by in vivo television microscopy, digital image microscopy, optical spectroscopy, 31P-NMR, spectroscopy and a unique magnesium ion-selective electrode.Alcohol abuse and brain infarction.Dietary cholesterol protects against alcohol-induced cerebral artery constriction.Peripheral and cerebrovascular actions of ethanol, acetaldehyde, and acetate: relationship to divalent cations.Smooth muscle cholesterol enables BK β1 subunit-mediated channel inhibition and subsequent vasoconstriction evoked by alcohol.Effects of ethanol on the tonicity of corporal tissue and the intracellular Ca2+ concentration of human corporal smooth muscle cells.Distinct mechanisms underlying cholesterol protection against alcohol-induced BK channel inhibition and resulting vasoconstriction.Chronic ethanol-induced glial fibrillary acidic protein (GFAP) immunoreactivity: an immunocytochemical observation in various regions of adult rat brain.BK β1 subunit-dependent facilitation of ethanol inhibition of BK current and cerebral artery constriction is mediated by the β1 transmembrane domain 2.Microangiopathy of ethylic polyneuropathy.Changes in nonhuman primate brain function following chronic alcohol consumption in previously naïve animals.Binge alcohol exposure in the second trimester attenuates fetal cerebral blood flow response to hypoxia.Age-Dependent Susceptibility to Alcohol-Induced Cerebral Artery Constriction.Ethanol stimulates immunoreactive endothelin-1 and -2 release from cultured human umbilical vein endothelial cells.Differential effects of alcohols on intracerebral arterioles. Ethanol alone causes vasoconstriction.
P2860
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P2860
description
1984 nî lūn-bûn
@nan
1984年の論文
@ja
1984年論文
@yue
1984年論文
@zh-hant
1984年論文
@zh-hk
1984年論文
@zh-mo
1984年論文
@zh-tw
1984年论文
@wuu
1984年论文
@zh
1984年论文
@zh-cn
name
Alcohol, the cerebral circulation and strokes.
@en
type
label
Alcohol, the cerebral circulation and strokes.
@en
prefLabel
Alcohol, the cerebral circulation and strokes.
@en
P1433
P1476
Alcohol, the cerebral circulation and strokes.
@en
P2093
P304
P356
10.1016/0741-8329(84)90056-9
P577
1984-07-01T00:00:00Z